Lobbying for a hearing
for referral to the USDOJ for a prosecution of the Lyme disease crimes.
5 – “Defrauding the Govt,” Allen Steere & CDC officers
Johnson and Barbour = ALDF.com
9 -- 1990 CDC case definition (No ELISA, Lyme has antigenic variation)
11 – Dearborn case definition (Steere shows he knows
NeuroLyme wont test positive)
14 – “Assault” of Czech children with fake vaccine to
see how serious would be the injuries
16 – Who was involved, including OspA vaccine trial
17 -- Appendix A: I-RICO and “Color of Law” charges,
II- Mayo’s ad, pg 20
21 -- Appendix B -- HLAs and TLRs, Autism vaccines; (pg 24 = Shapiro, PBS)
26 – Thimerosal was put in vaccines to prevent LYMErix or fungal
29 -- Appendix C - MB Pfeiffer in HuffPo 170109 “Holy
Shit”, we screwed up over Dearborn
30- What is OspA?
34 -- Hoof and Mouth Disease DNA in muscle biopsies in
Chronic Fatiguing disease … and with reactivation of Epstein-Barr. And oh,
ho hum, Plum Island.
This RICO, Fraud, Pediatric “Assault” (with LYMErix),
and “Color of Law” abuses case was already filed with the USDOJ in 2003.
Three years later, former DOJ prosecutor (now
Senator) Richard Blumenthal sued for Anti-Trust under CT civil law when
he was the Connecticut Attorney General.
Previously, the FDA ordered SmithKline to
voluntarily withdraw LYMErix or the FDA would order it off officially
(ultimatum). The withdrawal of this fungal toxin of a vaccine was announced
on February 26, 2002 by SmithKline Beecham (now Glaxo-SmithKline).
Although the charges in this case are clear: Defrauding
the Govt., RICO, and Color of Law, none of the non-profits or ILADS.org
participated in attempting to get it prosecuted (except Lyme.org, Hartford,
CT) and to date, still aren't. Were the case prosecuted and the scandal
exposed, there would be no need for these fake non-profits. The author
of this handbook was a member of ILADS when she blew the whistle at the FDA
(010131) and when she wrote the, ILADS’ “Klempner ‘re-treatment study’
rebuttal (“if Dearborn is invalid, Klempner & the IDSA ‘Guidelines’ are
invalid”), June, 2001.
Despite all these obvious facts, ILADS.org has never mentioned how OspA
alone causes "chronic Lyme," and never talks about the falsified case
definition, because people would then ask, "What
are you treating, if OspA alone causes the 'multi-system' disease of Chronic
has IDSA answered the specific question as to what OspA actually is, and
that’s the bigger complaint.)
uncertainty may arise if the vaccines are not completely protective;
vaccinated patients with multisystem
complaints characteristic of later presentations of Lyme disease may be
difficult to distinguish from patients with vaccine failure." - Crooks
Schoen (Yale) and Persing (Mayo), 1995, US Patent 6, 045,804 admitting that
LYMErix or OspA vaccination caused the same disease we know of as Chronic
As you have just seen, the Lyme Cryme perpetrators have SAID: “You
can’t tell Lyme and LYMErix disease apart because they are both post-septic
shock syndrome.” “Multi-system” disease means post-septic shock
syndrome. “Overwhelming the immune system” is the same thing as post-septic
What we want
prosecuted is the falsification of the "case definition" of Lyme, intended
to exclude neurologic Lyme cases in order to pass off a bogus fungal
vaccine, OspA, or Pam3Cys. OspA caused the same immunosuppression disease
that resulted in the variety show of diseases (also known as a "Great
Imitator") that are outcomes Lyme- or Tick Bite Sepsis and are mainly caused
by the secondary opportunistics (EBV, etc). One of the names for this
condition is post-sepsis syndrome. Post-sepsis syndrome is like AIDS but
without the T cell virus.
”The researchers looked for viruses like Epstein-Barr [EBV] and herpes
simplex that are often dormant in healthy people but can reactivate in those
with suppressed immune systems.”- NIH, 2014. This is a
well-known phenomenon and is seen across diseases outcomes, such as
transplant cases and Humira/Stelara users when they become too
immunosuppressed from their medications. You hear and see warnings of
reactivating latent EBV and EBV-induced cancer or leukemia/lymphoma. ‘Same
with the pediatric MMR. The monograph warns against vaccinating
immunosuppression children, because then those live viruses become
reactivated. (These facts are an “Occam’s Razor.”)
It is a criminal offense to request a grant from the federal government
based on fraud.
The biggest such fraud instance was when Mark Klempner asked for a 4.7
million dollar grant -knowing the case definition was false and that
spirochetes persist and were incurable with ceftriaxone due to being
intracellular -, to assess "long term treatment outcomes:"
Fibroblasts protect the
Lyme disease spirochete, Borrelia burgdorferi, from ceftriaxone in
Klempner also later
said Lyme did not cause any cognitive impairment after proving there
was a nerve and brain degrading enzyme in the cerebrospinal fluid of Lyme
victims in 79% of us:
Cognitive function in
post-treatment Lyme disease: do additional antibiotics help?
Next, the Klempner
“we found nerve and brain-degrading enzymes” report:
in the cerebrospinal fluid of patients with Lyme neuroborreliosis.
This is the falsified grant request and fake "study," was based on the
falsified Dearborn case definition:
Two controlled trials of
antibiotic treatment in patients with persistent symptoms and a history of
This ^^ Klempner "study" became the basis of the
Infectious Diseases Society of America's (IDSA's) "Guidelines" on the
diagnosis and treatment of Lyme disease. Here we will show the case
definition (diagnosis criteria) was falsified, and therefore there can be no
“guidelines” on “treatment.”
Understanding that the OspA vaccines caused the same
"multi-system" (Yale, Mayo Clinic, US patent, 6, 045, 804) disease we know
of as "Chronic Lyme (CLD)" is essential to this case. It turns out, CLD is
a condition of chronic immune deficiency and is physiologically the same as
AIDS or post-sepsis syndrome where the secondary opportunistic infections
reactivated via immunosuppression, tissue and organ damage (brain, gut), are
what's causing the ongoing illness.
If Lyme disease is incurable due to its being a
post-sepsis syndrome with immune system-, organ- and tissue damage, a leaky
blood brain barrier, leaky gut, and a multitude of fungal, bacterial and
viral infections that cant be fought off due to what happens to the immune
system in sepsis, then there is another charge is Color of Law where Government
employees slandered and libeled us to deny us any kind of care. Lyme
disease has been one great big charade to which no "MD" or group of "MDs"
has been competent.
Remember what the NIH said about sepsis and post-sepsis
addition to all the other things they said, like OspA alone causes this
outcome in Lyme victims):
NIH, Carolyn Beans:
Surviving Sepsis: Detection and Treatment Advances
- Live Science
Preventing Secondary Infections
Some people who survive sepsis can develop secondary
infections days or even months later. A research team that included Richard
Hotchkiss, Jonathan Green and Gregory Storch of Washington University School
of Medicine in St. Louis suspected that this is because sepsis might cause
lasting damage to the immune system. To test this hypothesis, the scientists
compared viral activation in people with sepsis, other critically ill people
and healthy individuals. The
researchers looked for viruses like Epstein-Barr and herpes simplex that are
often dormant in healthy people but can reactivate in those with suppressed
immune systems. [Sepsis
Has Long-Term Impact for Older Adults, Study Finds]
Of the three study groups, people with sepsis had
much higher levels of these viruses, suggesting reactivation due to
compromised immune responses. Immune suppression could make it difficult to
defend against the reactivated viruses as well as new infections like
pneumonia. The team now plans to test whether immune-boosting drugs can
prevent deaths in sepsis survivors.
Government with the falsified case definition: Allen Steere
923. 18 U.S.C. § 371—Conspiracy to Defraud the
United States ...
To conspire to defraud the
United States means primarily to cheat the Government out
of property or money, but it also means to interfere with or obstruct one of
its lawful governmental functions by deceit, craft or trickery, or at least
by means that are dishonest.
924. Defrauding the Government of Money or
Property | USAM ...https://www.justice.gov/.../criminal-resource-manual-924-defrauding-government-mo...
the Government of
Money or Property. Under 18 U.S.C. § 371 the act of defrauding
the government of
money or property may take many forms, including the inducement of payment.
for services or supplies not provided or provided at inflated prices; for
work for which the government is
The 1994 CDC, “Dearborn”/Current- case definition says you need 5 of
10 IgG bands (arthritis only), after the non-HLA-linked-, non-arthritis
cases are screened out in the first step, the ELISA.
That is, Chronic Neurologic Lyme cases, which are immunosuppression outcomes
- like AIDS, where the opportunistics do most of the damage and are what
keep you ill -, were left out at the first step, the ELISA, because this
outcome was caused by injections of the fungal toxin OspA (the Lyme
The Disease (fungal-toxic immunosuppression or
post-sepsis syndrome)…is the Cryme (saying OspA was a “vaccine” when
it caused the same toxic, post-sepsis syndrome).
The Dearborn case definition was not a consensus. The average accuracy was
15%, as is shown in this booklet covering the Dearborn conference:
Where does the Dearborn proposal come from? [Steere in Europe, 1992-1993]
J Infect Dis. 1994
Antibody responses to the three
genomic groups of Borrelia burgdorferi in European Lyme borreliosis. (Dressler
The above graphic shows:
1) Allen Steere in the IgM ELISA arbitrarily raised the "noise"
cutoff of 3 standard deviations to 5 std dev (3 is normally done) such that
most Neurologic cases would be missed (arthritis cases produce lower IgM,
for some reason).
2) Steere in the IgG-falsification step, averaged the concentration of IgGs
from the meningitis or neurologic Lyme (lower, like 1:400 dilution), with
acrodermatitis (autoimmune, very high antibody concentration of about
1:25o0) and arthritis (1:800 dilution).
This fraud deliberately excluded the sickest patients in the first step of
the Dearborn "2-tiered" testing criteria for Lyme. Note that it is strange
that Steere felt he had to develop this new Dearborn panel in Europe,
presumably where American Justice would have a hard time verifying the data.
Note this same report reveals an intended a later monopoly on testing for
Lyme once the bogus OspA vaccines were on the market (you never test for a
disease with the same antigens that are the vaccine antigens since you would
not know if the antibodies are from the actual infection or from the vaccine
The above graphic shows:
3) Steere used high passage strains which lose plasmids and therefore
potential antigens (meaning if you have those antibodies, they wont
be detected); We’re not even sure if strain G39/40 bears OspA/B.
4) Steere used strain B31 which essentially does not have the European kinds
of OspAs, and
he assured no one would have antibodies against OspA and B in this Dearborn
antibody panel for Western Blotting by leaving off the Pam3 or the tri-acyl
or the lipid groups of these triacyl lipoproteins which cause antibodies.
The protein ends by themselves are not immunogenic (cause antibodies to be
See full text here >>
WHO was involved with approving it anyway? [Johnson,
Barbour, Steere, McSweegan, etc]
^^^ This graphic is from pages 37 and 38 of the
Barbour (ALDF, CDC)
McSweegan (ALDF, NIH employee)
Allen Steere, Arthur Weinstein, Russell Johnson (ALDF.com)…
”CDC Lyme Disease Group,” ie., CDC employee Barbara Johnson, ALDF member,
multi-patent owner with SmithKline…
The people named here are essentially the same cabal
who own all the patents, is the ALDF.com, and who were on the
FDA committees to approved their own bogus recombinant triacyl lipoproteins
(fungal and immunosuppressive) as vaccines. Steere, Schoen, etc., the
The ALDF.com is the RICO “enterprise.”
“Another common issue concerns the element requiring a pattern of
racketeering activity. The RICO Act itself defines the term pattern as two
or more acts of racketeering activity within a 10-year period. However, the
Supreme Court has weighed in on this issue as well. According to the Court,
to qualify as a pattern, the criminal activities must be related and
continuous. Relatedness will be established if the crimes have similar
characteristics such as the same perpetrators, victims, and methods of
commission. Continuity will be established if the crimes occurred over a
substantial period of time. Some courts have interpreted this to mean at
least one year.”
The Previous, 1990, CDC case definition
(No, ELISA, IgM counts):
This 1990 standard just says to perform repeated or
sequential Western Blots to look for new IgM bands because that meant to
Allen Steere that the bug “remains alive throughout the illness.”
Where does this 1990 case definition came from?
[Steere, 1986, says band 41 alone is fine]:
J Clin Invest. 1986
Antigens of Borrelia burgdorferi recognized during
Lyme disease. Appearance of a new immunoglobulin M response and expansion of
the immunoglobulin G response late in the illness.
Craft JE, Fischer
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC423723/ (full text)
immunoblots, we identified proteins of Borrelia burgdorferi bound by IgM and
IgG antibodies during Lyme disease. In 12 patients with early disease alone,
both the IgM and IgG responses were restricted primarily to a 41-kD antigen.
This limited response disappeared within several months. In contrast, among
six patients with prolonged illness, the IgM response to the 41-kD protein
sometimes persisted for months to years, and late in the illness during
arthritis, a new IgM response sometimes developed to a 34-kD component of
the organism. The IgG response in these patients appeared in a
characteristic sequential pattern over months to years to as many as 11
spirochetal antigens. The appearance of a new IgM response and the
expansion of the IgG response late in the illness, and the lack of such
responses in patients with early disease alone, suggest that B. burgdorferi
remains alive throughout the illness.” !!!
The above graphic from the same 1986 report shows Allen
Steere believed band 41 or the anti-flagellar antibody would have been good
enough to detect, especially, early Lyme.
This Borrelia burgdorferi-specific antibody
testing, was later VALIDATED per the FDA rules on the Validation of a
Bioanalytical Method, in U.S. patent 5, 618,533. The testing was
made SPECIFIC by coding for a section of flagellin that was SPECIFIC
to burgdorferi (did not cross react with other flagellins. It was
made ACCURATE by detecting 17/18 known cases (known by a DNA
method). And it detects early late, neurologic, and arthritis Lyme. The
people who are the “inventors” of this patent, Yale’s Fikrig and Flavell,
also own the LYMErix patent. Because this testing could have been used to
assess the outcome of LYMErix, but was not (the Dearborn falsified criteria
were), and Fikrig and Flavell did not say anything or object to Dearborn or
how the OspA vaccines were assessed, these two are likeliest to be the ones
who “flip,” take a plea deal, and rat out the cabal.
But the current, Dearborn case definition, the one used
to defraud the government out of grant money to say “people are not sick
from Lyme” (Klempner) and to claim they had OspA “vaccines” (ImmuLyme and
LYMErix) is this (5 of 10 IgG bands, and practically no IgM bands, when IgM
bands are more associated with chronic, ongoing, neurologic spirochetal
"It was recommended that an IgM immunoblot be considered positive if two of
the following three bands are present: 24 kDa (OspC) * , 39 kDa (BmpA), and
41 kDa (Fla) (1). It was further recommended that an that IgG immunoblot be
considered positive if five of the following 10 bands are present: 18 kDa,
21 kDa (OspC) *, 28 kDa, 30 kDa, 39 kDa (BmpA), 41 kDa (Fla), 45 kDa, 58 kDa
(not GroEL), 66 kDa, and 93 kDa (2)."
1993 Steere showing positive cases are HLA-linked and neuroLyme is not.
When presenting this, you will be reading this
out-loud, so it is LOUD AND CLEAR that neurologic Lyme cases "who never
had arthritis" (and never will because they dont have the HLAs) "SHOWED WEAK
REACTIVITY WITH THE FUNGAL OSPS" - which is normal since they're fungal –
their immune response is turned off to these fungal Osps.
Yet, as you have just seen, Steere screened those cases out via research
fraud in Europe:
1993 - Association of
treatment-resistant chronic Lyme arthritis with HLA-DR4 and antibody
reactivity to OspA and OspB of Borrelia burgdorferi.:
So, Allen Steere falsified the case definition for Lyme
in 1993 in Europe to deliberately screen out the most severe cases of Lyme -
the ones who low antibody concentration, especially in IgG -, so that he and
his cabal could later falsify the vaccines using this same criteria … that
left out 85% of all cases, all the neurologic cases, and only identified the
HLA-linked, autoimmune, “too many antibodies” cases.
Yale owns a patent (5, 618, 533) for valid testing, not
used for the OspA vaccine, LYMErix, which they also own:
Band 41, made specific and accurate (see
FDA rules for validations of bioanalytical methods):
The Yale, LYMErix, OspA patent (5, 747, 294):
What the Western Blots look like, Normal (NeuroLyme) vs
In the above graphic, the ones on the left (neuro) represent normal people
with few to no bands, whereas on the right, the people with TOO MANY
ANTIBODIES (hence, darker bands and more of them, which refers to antibody
concentration) or have an allergy response to Borrelial antigens which are
the HLA-linked, hypersensitivity-linked arthritis responses. These latter
people are not sick, they just have bad knees according to Klempner and
Wormser in 2005:
A case-control study to
examine HLA haplotype associations in patients with posttreatment chronic
The above graphic on HLA-linked and non-HLA linked
outcomes by Mark Klempner in 2005 shows that there are 2 outcomes to Lyme:
Arthritis, where the “Patients feel well aside from their arthritis
symptoms,”… and the other 85% with post-septic shock multi-system
complaints who are clearly the sickest and never get better. This latter
immunosuppression illness was also caused by the OspA vaccines.
So, it is clear: Since the cabal was formed in 1990
(ALDF.com), these criminals falsified the case definition such as to EXCLUDE
everyone who is sick with the neurologic outcome, and INCLUDE everyone who
is not sick but just has a bad knee. It looks like they did not want anyone
treated for Lyme at all, ever.
The Czech Children Guinea Pig Stunt
Shapiro and UConn use European children as guinea pigs.
There is none of that kind of OspA in Europe, and they already knew by 1993
that it did not prevent Lyme and was harmful to humans:
J Infect Dis. 1994 Feb;169(2):313-8. (again, you have
seen this before)
Antibody responses to the three genomic groups of Borrelia burgdorferi
in European Lyme borreliosis. Dressler F1, Ackermann R, Steere AC.
”The antibody responses to the three genomic groups of Borrelia burgdorferi
(B. burgdorferi sensu stricto, Borrelia garinii, and Borrelia afzelii) were
determined in 97 German patients with various manifestations of Lyme
borreliosis. The geometric mean antibody titers in each patient group,
determined by ELISA, were similar with each antigen preparation. By Western
blotting, however, patients with meningopolyneuritis tended to respond to
more spirochetal polypeptides of B. garinii, the group 2 strain, whereas
those with arthritis recognized more antigens of B. afzelii, the group 3
strain (P < .03), as did those with acrodermatitis. Only
1 patient each with erythema migrans, arthritis, or acrodermatitis had weak
reactivity with outer surface protein A (OspA), and none responded to OspB.
It is concluded that differences among the three groups of B. burgdorferi
may result in variations in the antibody response in European Lyme
And here it shows that less than a 3rd of all Borrelia spirochetes in Europe
express the American B31 OspA serotype:
So, for Yale and UConn to use this OspA from strain B31
(LYMErix) on Czech children in Europe is merely “assault” (a criminal
charge), because American B31 OspA in Europe would not have done them any
good. They merely assaulted these children to see how bad would be the
And once again, these children were never followed.
They were observed for 15 minutes following injection and their serum was
never tested for antibody levels after one month.
No follow up, no placebo control: Just whack kids with
a vaccine that would do them no good (not much of the B31 kind of OspA in
Europe) to see how bad would be the adverse events probably because they
know OspA as an immune suppressing fungal antigens do not produce long term
antibodies, being a fungal immune suppression triacyl lipoprotein TOXIN.
Who was involved?
Robert Schoen, Durland Fish, Eugene Shapiro (all at Yale), Dave Persing
(Mayo Clinic and Corixa)
Henry Feder, Larry Zemel, (Uconn), Lenny Sigal, John Nowakowski, (?)
Nadelman, Arthur Weinstein, JJ Halperin,
CDC Officers and Patenteers: Alan Barbour, Barbara Johnson,
CDC officers Paul Mead, Allen Steere, Mark Klempner,
Gary Wormser (NYMC, founding member of the ALDF.com)
John J. Connolly (NYMC, founding member of the ALDF.com)
Others at the CDC
Paul Auwaerter (Johns Hopkins)
All the OspA accines trials administrators
The 1998 Vaccines Reports (ImmuLyme
LYMErix results (76% "safe and effective"):
ImmuLyme results (92% "safe and effective"):
Appendix A: RICO and “Color of Law” charges
https://www.law.cornell.edu/uscode/text/18/1961 << Go ahead and read
“Color of Law” – means falsely using one’s authority to
deny rights, such as is the slander and libel against Lyme and CFIDS victims
by CDC officers and “health department officials” particularly in the State
of CT which stood to profit (Uconn) by participating in Yale’s fake LYMErix
trials, especially of Czech children (Shapiro, Zemel, Feder).
This is a particularly serious charge, especially if there is a State health
department official who participated in any of Yale’s, SmithKline’s, or
ImmuLyme’s (other OspA) vaccine trials., or a CDC officer that
published a misogynistic, unreferenced book (eg., Alan Barbour)
18 U.S. Code § 242 - Deprivation of rights under
color of law [means using their official authority as CDC officers
changing testing criteria to sell their own products or patents, such as
Alan Barbour and Barbara Johnson, and who then slander and libel their
victims, depriving them of their rights, access to care, access to Social
Security Disability or Insurance income.]
Whoever, under color of
any law, statute, ordinance, regulation, or custom, willfully subjects any
person in any State, Territory, Commonwealth, Possession, or District to the
deprivation of any rights, privileges, or immunities secured or protected by
the Constitution or laws of the United States, or to different punishments,
pains, or penalties, on account of such person being an alien, or by reason
of his color, or race, than are prescribed for the punishment of citizens,
shall be fined under this title or imprisoned not more than one year, or
both; and if bodily injury results from the acts committed in violation of
this section or if such acts include the use, attempted use, or threatened
use of a dangerous weapon, explosives, or fire, shall be fined under this
title or imprisoned not more than ten years, or both; and if death results
from the acts committed in violation of this section or if such acts include
kidnapping or an attempt to kidnap, aggravated sexual abuse, or an attempt
to commit aggravated sexual abuse, or an attempt to kill, shall be fined
under this title, or imprisoned for any term of years or for life, or both,
or may be sentenced to death.
(June 25, 1948, ch.
Stat. 696; Pub.
L. 90–284, title I, § 103(b), Apr.
11, 1968, 82
Stat. 75; Pub.
L. 100–690, title VII, § 7019, Nov.
18, 1988, 102
Stat. 4396; Pub.
L. 103–322, title VI, § 60006(b), title XXXII, §§ 320103(b),
320201(b), title XXXIII, § 330016(1)(H), Sept.
13, 1994, 108
Stat. 1970, 2109, 2113, 2147; Pub.
L. 104–294, title VI, §§ 604(b)(14)(B), 607(a), Oct.
11, 1996, 110
Stat. 3507, 3511.)
Who approved Dearborn [CDC officers who own
patents that would be worthless if people know that spirochetal and other
tick borne diseases (TBDS) antigens were also fungal-ish and therefore could
never be used for test kits or vaccine - the goal of ALDF.com]?
They were the same people on the FDA's various panels to discuss the testing
(qualification) and approve LYMErix.
They were the same people who 1) own the patents, 2) changed the testing, 3)
approved of their own bogus vaccine(s) and 4) via Color of Law, trashed Lyme
and LYMErix victims.
What is the failed vaccine and how do we know they
OspA causes the same disease (What is it, and who says so? Latov, Persing
and Schoen, Marks, NIH's Martin and Marques, Philipp, Dattwyler, etc, see
the Occam's Razor/Appendix C?)
OspA is fungal, TLR2/1 agonist (triacyl-lipopeptide)
Barbour and Fish libel and slander ("Color of
Law," Barbour is a CDC officer):
Barbour’s Misogyny Book, totally unreferenced:
Douglas Dodge’s “LymeTruth”
More Slander and Libel:
In Parallels (has OspA-ish been tried before as a
vaccine? Occam's Razor/Appendix C?)
RICO within the RICO
(Mayo Clinic and Yale)
Mayo's RICO patent (US Patent # 6, 045, 804)
This patent reveals they know LYMErix causes a systemic disease like chronic
Lyme, and in this patent they reveal their
intended monopoly on post-LYMErix testing for the USA and Canada as they
claimed in their advertising:
"Additional uncertainty may arise if the
vaccines are not completely protective; vaccinated patients with multisystem
complaints characteristic of later presentations of Lyme disease may be
difficult to distinguish from patients with vaccine failure."
"The present invention provides a method useful to detect a B. burgdorferi
infection in a subject. The method provided by the invention is particularly
useful to discriminate B. burgdorferi infection from OspA vaccination,
although it is sufficiently sensitive and specific to use in any general
Lyme disease screening or diagnostic application. Thus,
the method of the invention is particularly appropriate for large scale
screening or diagnostic applications where only part of the subject
population has been vaccinated or where the vaccination status of the
population is unknown.”
(See the Mayo advertisement in
Who worked on it [Yale’s Robert Schoen and Mayo's Dave Persing]
KEY CONCEPT: Why was it done? [Monopoly on all future TBDs testing in USA
and Canada once an OspA vaccine was on the market, as shown above. To
capture the market for TBDs testing once LYMErix was on the market: Lyme
testing and test kits are more lucrative than vaccines, plus you get to
patent all the new pathogens you find in that monopoly blood.]
How does the Mayo patent show a RICO?
Blumenthal's staff asked the author if this cabal ever openly advertised
their intentions with this test?
Yes, Mayo Clinic and Yale actually did publish that they would have the only
testing you could use to assess for Lyme and TBDs once LYMErix was on the
market; that Imugen, Corixa and Yale’s L2 Diagnostics would be the only 3
labs in the country to be licensed to use the RICO patent (6,045,087)
method. (See the Mayo
advertisement in Appendix B)
ODDLY, Persing and Sigal later said none of the OspA trials were
valid as the Western Blotting was unreadable, but both LYMErix and ImmuLyme
trials said they used the Dearborn method to approve these vaccines. The 3
labs, Corixa, Yale's L2 Diagnostics, and Imugen were the only ones licensed
to use this patented testing for the post-LYMErix criminal ALDF RICO era:
Clin Infect Dis. 2000
Jul;31(1):42-7. Epub 2000 Jul 17.
Detection of multiple reactive protein species by
immunoblotting after recombinant outer surface protein A lyme disease
“The manufacturer of
the only currently FDA-approved (and released) recombinant OspA Lyme disease
vaccine has suggested that vaccination does not interfere with serological
evaluation of Lyme disease in vaccine recipients—a statement that is not
supported by the data presented here.
Neither of the 2 OspA vaccine teams could actually read their Western
Blots, yet they falsely claimed they had qualified the safety and
efficacy of those vaccines, ImmuLyme and LYMErix.
Appendix A, part
II – Mayo Advertising their intent; “the RICO within the RICO”
“Mayo Clinic Rochester News, Tuesday, August 4, 1998
“New Tests Set Standard for Diagnosing Lyme Disease
“ROCHESTER, MINN. — Mayo
Medical Laboratories and IMUGEN Inc. announced today the newest and most
accurate test series available for diagnosing Lyme disease. The tests also
are the only reliable means of diagnosing Lyme disease in people who have
been vaccinated against Lyme disease.
“Mayo Medical Laboratories, the laboratory for Mayo Clinic, and IMUGEN
Inc. of Norwood, Mass., are jointly offering the new proprietary tests
through local hospitals and clinics. Availability of the new tests coincides
with the anticipated release of new Lyme disease vaccines, such as the
widely-publicized LYMErix and ImuLyme.
”In research trials, all other Lyme tests have been shown to
produce false-positive results in people vaccinated against Lyme disease.
Moreover, the downstream costs of medical care delivered on the basis of
just one false-positive Lyme test can be as much as $15,000.
“According to Dr. David Persing, a Mayo Clinic molecular biologist
involved in the discovery of the new test components, physicians now have a
new and more reliable means of diagnosing patients who present with symptoms
of Lyme disease.
"These tests should help reduce the human and financial costs associated
with the number of undiagnosed, misdiagnosed, untreated or improperly
treated patients," Dr. Persing added.
”Scientists at IMUGEN, recognized nationally as the leading reference
laboratory for tick-borne diseases, are responsible for developing the
highly accurate immunologic methods to utilize Dr.Persing’s discovery.
[It was Schoen at Yale, unless Steere really is a
partner at Imugen as some Lyme activists have claimed. – SASH]
"Diagnosing Lyme disease has been highly problematic for a long time,"
said Victor Berardi, chief executive officer of IMUGEN, whose laboratories
have performed more than a half-million Lyme disease tests. "Our new tests
will greatly help physicians in distinguishing patients who are actually
infected from those who aren’t. Furthermore, the accuracy of these tests
will not be affected by Lyme vaccine. In any case, the tests will help
physicians render more appropriate and cost effective care."
“Lyme disease is a tick-borne illness that if left undiagnosed or
untreated can severely damage the human heart and nervous system. Nationally
more than 16,000 cases of Lyme disease were reported to the Centers for
Disease Control and Prevention (CDC) in 1996. The majority of cases were
reported in New England and the Northeast. The CDC reports that the overall
number of Lyme disease cases could climb to 25,710 by the year 2000.
“In a study of 10,936 people in states with a high incidence of Lyme
disease, one new vaccine proved 79 percent effective at preventing Lyme
disease infections after complete dosage. Given the potential popularity of
the vaccine, and the recent epidemic of Lyme disease in the Northeast, the
new tests offered by Mayo Medical Laboratories and IMUGEN will be of
”The new Lyme disease tests detect multiple classes of antibody isotypes,
enabling them to discriminate between the vaccine and a true Lyme infection.
Existing Lyme disease tests, however, have shown to produce false-positive
results in patients vaccinated for Lyme disease.
“IMUGEN Inc. of Norwood, Mass., is a pioneer in the research, development
and testing of tick-borne diseases, including Lyme disease, babesiosis and
ehrlichiosis. For the past decade, IMUGEN has provided clinics and hospitals
in the Northeast with high-quality serologic testing from its facilities in
Norwood, Mass., and Southhampton Hospital in Southhampton, N.Y. For more
information, call 781-255-0770.
“Mayo Medical Laboratories is the laboratory for Mayo Clinic and provides
lab services to community-based healthcare organizations throughout the
nation and world. Mayo Medical Laboratories draws from the expertise of Mayo
Clinic’s 1,600 physicians and scientists who provide specialized
consultation on test selection, utilization and interpretation.
“For information, call 800-533-1710. “Contact:
“Tom Huyck, “507-284-0003 (days)
Appendix B --
HLAs and TLRs, Autism vaccines; The kids are getting the viruses and not in
the classic “autoimmune, inflammatory” way
HLA-linked hypersensitivity (="Autoimmune,"
"inflammatory," ...) – What are the HLAs? They are the genetically encoded
clam-shaped molecules in “professional antigen presenting cells like
macrophages and B cells that “present antigen” against which antibodies to
be made. Here we see Wikipedia showing that the HLA’s SHAPE is
genetically determined. There is a variety. The people with Lyme arthritis
have a certain type of HLA clam-shaped molecule that results in over- or
chronic stimulation, such as an allergy or is called hypersensitivity. This
is the 15% who continue to make antibodies against Lyme and are not sick.
The 85% of us without Lyme arthritis allergy or autoimmunity stop making
antibodies because the fungal Osps are seen as serious toxins.
Khan Academy explainer on MHCs or HLAs
This “Major Histocompatability Complex” business has to
do with, originally, tissue rejection in transplants. When they look for a
donor “match” they want to make sure the donor and the recipient have the
same HLAs, so the tissue will not be rejected as “foreign.” (That was how
they were discovered.)
Toll Like Receptors take the antigen *IN,* then some
physiochemical changes happen, the bug is broken up, then the 2 clam shaped
HLA molecules "PRESENT" antigen *OUT* to the surface of the cell so the
other immune cells can see, "Hey, THIS is a bad guy." When the
antigen is a fungal toxin like LYMERIX??? The HLA molecules no longer
PRESENT ANTIGEN and you make no more antibodies - not even against VIRUSES,
due to CROSS TOLERANCE shown here:
"Because IRAK1 is required for TLR7/9-induced IFN-I
production, we propose that TLR2 signaling induces rapid depletion of IRAK1,
which impairs IFN-I induction by TLR7/9. This novel mechanism, whereby TLR2
inhibits IFN-I induction by TLR7/9, may shape immune responses to microbes
that express ligands for both TLR2 and TLR7/TLR9, or responses to
In Lyme and LYMErix disease, the HLA molecules are
“down-regulated” or stop “presenting antigen” because this is a way of
STOPPING DEATH (prevents apoptosis) from the “cytokine storm” in sepsis.
This co-occurs in other cases such as in Tuberculosis and in other trials of
vaccines with the same type of antigen, fungal or triacyl (TLR2/1 agonists):
Watch the Khan Academy and similar videos on
immunology, especially the one about “Professional Antigen-Presenting Cells”
and HLA/MHC molecules.
TLR2/1 agonists (lipoproteins, triacylated);
TLR agonist tolerance and cross tolerance Osps turn off the response to
viruses (TLR7/9), CV Harding:
"Great Imitator" = something that sets off the variety
show of syndromes after TLR2/1 agonist sepsis or polymicrobial sepsis; the
diagnosable ones being due to the secondary opportunistics, like Cancer,
Lupus & MS (EBV, etc). Arthritis is allegedly more due to foreign fungal
antigens and allegedly heat shock proteins.
Exosomes, Blebs: Steere recognized relative abundance of exported blebs in
knees with Duray in early 1990s. You may ask what is responsible for the
ongoing stimulation in the 15%, the arthritis, HLA-linked cases.
Infect Immun. 2010
Dec;78(12):5116-25. doi: 10.1128/IAI.01089-09. Epub 2010 Sep 13.
Mycobacterium tuberculosis synergizes
with ATP to induce release of microvesicles and exosomes containing major
histocompatibility complex class
II molecules capable of antigen presentation.
Ramachandra L1, Qu
GR, Harding CV.
with M. tuberculosis primes
macrophages for the increased release of exosomes and microvesicles bearing
M. tuberculosis peptide-MHC-II
complexes that may generate antimicrobial T-cell responses.”
Is it possible that a response to these fungal antigens is to release the
whole MHC/HLA-II Plus Antigen complex, which then again becomes a new
free antigen in itself? Is this what Steere saw when he claimed that there
was a cross reaction with HLA molecules as a cause of the ”autoimmunity” in
“Disease” without classic “Inflammation” or “Autoimmunity” – This FRAUD has
to do with the Autism pandemic:
The Lyme criminals claim you can’t have a “disease” unless you have
inflammation or an autoimmune outcome. Of course, such a claim betrays the
source of the Autism pandemic. The kids are getting the viruses instead of
the protection and this is shown in many places and is called an “adverse
Yale’s Eugene Shapiro in PBS’ “Life on Earth Series”:
”TOOMEY: But most physicians think there's good reason
to discount the possibility of chronic Lyme Disease.
”SHAPIRO: What some people would have you believe is that there are two
”TOOMEY: Yale physician Eugene Shapiro says first, take the obvious case of
Lyme Disease that usually starts with a distinctive rash and can lead to
arthritis and facial paralysis.
“SHAPIRO: Somehow, for that form of the disease, antibiotics are effective.
They do fine. But then there's some other form of the disease which is, you
can't put your hand around it. They don't have objective findings of
inflammation, which is the way bacteria cause disease.
”TOOMEY: What these patients do have are symptoms that doctors call
nonspecific. They span a broad spectrum. Emotional problems, as in the case
of Lisa's daughter; or fatigue, muscle pains, depression. Doctor Shapiro
helped write the Lyme treatment guidelines put out by the Infectious
Diseases Society of America. Guidelines that state even the most advanced
cases of infection can be eradicated with two months of oral or intravenous
antibiotics. But for the patients with these ongoing, nonspecific symptoms,
the maximum treatment didn't seem to work.
”SHAPIRO: They would have you believe that form of the disease, somehow this
is, the bacteria knows to act differently and it doesn't respond to
antibiotics in this sense. It really doesn't make any sense.
Of course it does make sense: Spirochaeta are not
bacteria, in the sense that 1) they are their own Phylum, 2) shed fungal
antigens and 3) have no LPS. But, what would Shapiro know about science?
His undergraduate degree is in English Literature. The point is, you see
here, Shapiro, et al, claim that you can’t have a disease unless you have
inflammation or an autoimmune one. Spirochetes are notorious immune
suppressors like Tuberculosis because the Osps are triacylated lipoproteins,
which means they are managed by Toll Like Receptors 1 and 2, and which means
they are FUNGAL. (Which means THEY ARE NOT REGULAR “BACTERIA.”)
Check the “Contraindications” of the MMR monograph by
”Histologic changes, similar to those seen in
gestational rubella, have been observed and rubella virus has been recovered
from decidua following vaccination of pregnant women with live attenuated
rubella vaccine. These vaccines may thus constitute a risk to the fetus.
“Active untreated tuberculosis. Patients receiving
immunosuppressive therapy with ACTH, corticosteroids, irradiation,
alkylating agents or antimetabolites. This contraindication does not apply
to patients who are receiving corticosteroids as replacement therapy, e.g.,
for Addison’s disease. Individuals with blood dyscrasias, leukemia,
lymphomas of any type, or other malignant neoplasms affecting the bone
marrow or lymphatic systems. Primary and acquired immunodeficiency states,
including patients who are immunosuppressed in association with AIDS or
other clinical manifestation of infection with human immunodeficiency
viruses; cellular immune deficiencies, hypogammaglobulinemic and
dysgammaglobulinemic states. Measles inclusion body encephalitis (MIBE),
pneumonitis and death as a direct consequence of disseminated measles
vaccine virus infection has been reported in severely immunocompromised
individuals inadvertently vaccinated with measles-containing vaccine…”
The above all means: don’t vaccinate someone who is
immunosuppressed or already has an infection especially a fungal one (like
Lyme or LYMErix disease) because the victim can become damaged from the live
virus, and which is not an “autoimmune” outcome.
Check also Wikipedia on the topic of “Attenuated
mutation can cause a reversion to virulence.
severe complications in immunocompromised patients.
“Some can be
difficult to transport due to requirement to maintain conditions (e.g. temperature)
Check the CDC when in their patents they claim vaccines
fail by giving people the actual virus the vaccines are intended to prevent:
CDC’s Patent, US # 7,632,510,
Methods of inducing flavivirus immune responses through the
administration of recombinant flaviviruses comprising an engineered japanese
encephalitis virus signal sequence
"Finally, there is the risk that the virus may not be fully or
completely inactivated or attenuated and thus, the vaccine may actually
CDC SAYS,…Measles, Mumps, and Rubella -- Vaccine Use and Strategies
for Elimination of Measles, Rubella, and Congenital Rubella Syndrome and
Control of Mumps: Recommendations of the Advisory Committee on Immunization
"Updated information on adverse events and contraindications, particularly
for persons with severe HIV infection, persons with a egg allergy or gelatin
allergy, persons with a history of thrombocytopenia, and persons receiving
steroid therapy." [are
immunosuppressed, steroids cause immunosuppression- KMD]
CDC SAYS: Human
Exposure to Brucella abortus Strain RB51 -- Kansas, 1997
[In the above, an immunosuppressed pregnant cow was given a Brucella
(LYMErix-like) "live attenuated" vaccine and the baby cow ended up with the
disease, which then was transferred to the humans handling the cow and her
dead baby. This parallels what is happening to children who are vaccinated
while immunosuppressed, or who receive mycoplasmally (LYMErix-like)
contaminated vaccines -$A$H.]
Check out Paul Auwaerter’s experiment where he harvested a live virus from a
child and said of this activity:
”… and ****a
virus isolated from an immunodeficient child with progressive
vaccine-induced disease (Hu2).**** …”
Therefore, many times the CDC and other have said the reasons vaccines fail
is because of immunosuppression or the live attenuated viruses become live,
was even put in vaccines to PREVENT the occurrence of co-injecting fungal
(contamination) antigens together with the live viruses in vaccines, because
when you inject both fungal antigens and live viruses together, the
immunosuppression from the fungal antigens (like LYMErix, or OspA) you get
the viruses reactivated:
2012, Dec, NYTimes; Vaccine Rule Is Said to Hurt Health Efforts
"But a proposal that the ban
include thimerosal, which has been used since the 1930s to prevent
bacterial and fungal contamination in multidose vials of vaccines, has
drawn strong criticism from pediatricians…. They say that the ethyl-mercury
compound is critical for vaccine use in the developing world, where
multidose vials are a mainstay…Banning it would require switching to
single-dose vials for vaccines, which would cost far more and require new
networks of cold storage facilities and additional capacity for waste
disposal, the authors of the articles said.'"
See what the effect is of injecting fungally contaminated vaccines into
newborns even without the live viruses:
Brain Behav Immun. 2015
Aug;48:301-12. doi: 10.1016/j.bbi.2015.04.020. Epub 2015 May 27.
Postnatal TLR2 activation impairs learning and
memory in adulthood.
the central nervous system is detrimental for learning and memory, as
evident form epidemiological studies linking developmental defects and
maternal exposure to harmful pathogens. Postnatal infections can also induce
neuroinflammatory responses with long-term consequences. These inflammatory
responses can lead to motor deficits and/or behavioral disabilities. Toll
like receptors (TLRs) are a family of innate immune receptors best known as
sensors of microbial-associated molecular patterns, and are the first
responders to infection. TLR2 forms
heterodimers with either TLR1 or
TLR6, is activated in response to gram-positive bacterial infections, and is
expressed in the brain during embryonic development. We hypothesized that
early postnatal TLR2-mediated
neuroinflammation would adversely affect cognitive behavior in the adult.
Our data indicate that postnatal TLR2 activation
affects learning and memory in adult mice in a heterodimer-dependent manner. TLR2/6
activation improved motor function and fear learning, while TLR2/1
activation impaired spatial learning and enhanced fear learning. Moreover,
significantly impairs spatial learning and enhances fear learning, stressing
the involvement of the TLR2 pathway
in learning and memory. Analysis of the transcriptional effects of TLR2 activation
reveals both common and unique transcriptional programs following
heterodimer-specific TLR2 activation.
These results imply that adult cognitive behavior could be influenced in
part, by activation or alterations in the TLR2pathway
And an experiment from the 1950s where fungi (mycoplasma) were
injected together with live viruses to show exactly how this dual injection
THE RELATIONSHIP OF EPERYTHROZOON COCCOIDES TO THE HEPATITIS VIRUS OF
"In Swiss mice, animals with high natural resistance to hepatitis virus, the
pathogenicity of this agent was markedly enhanced by combined infection with
eperythrozoa. Eperythrozoa were maintained throughout 18 successive passages
in normal Princeton and Swiss weanlings with intact spleens. The combined
infection of Princeton mice with eperythrozoa and the virus component of
Gledhill, Dick, and Andrewes, which is nearly inactive when injected alone,
resulted in acute hepatitis with fatal outcome."
In the case of the MMR vaccine damage, Borna virus is a
model of the “neurodevelopmental damage” known as Autism.
The Infectious Diseases Society of America said this
about the brain damaging vaccines:
Cures Act: Boosting Antimicrobial Development, Constraining Vaccinations
“Amanda Jezek, the vice president of Public Policy and Government Relations
at the Infectious Diseases Society of America (IDSA), in Arlington, Va.,
said there is concern that this push to recommend a vaccine before the ACIP
has reviewed the evidence would completely “jeopardize the integrity of
ACIP’s recommendations.”<![if !vml]><![endif]>
of the vaccinations given in this country are received by those younger than
2 years of age, so assuring the safety and efficacy of vaccines is
paramount. Every year, more than 40 million vaccines are given to children
younger than 1 year of age, usually between 2 and 6 months of age, Dr. Temte
said. At this age, infants are at greatest risk for certain serious medical
adverse events, including high fevers, seizures and sudden infant death
syndrome, according to the U.S. Vaccine Adverse Event Reporting System.
Therefore, it is important for the ACIP to consider carefully the risks
versus the benefits before making a recommendation rather than be on a
forced schedule that suits the manufacturer as opposed to the patient.
ISSUE: AUGUST 2015 | VOLUME: 42:8
”a recommendation rather than be on a
forced schedule that suits the manufacturer as opposed to the patient.”
MaryBeth Pfeiffer in the HuffPo, Jan 2017, re: CDC’s falsified Dearborn case
Flunk the Lyme test? Just wait and get
03:49 pm ET | Updated Jan
Beth Pfeiffer is an investigative journalist who is writing a book, “The
First Epidemic,” on the global spread of ticks and the diseases they carry.
Follow her on Twitter: @marybethpf
”In researching my book on the global spread of tick-borne disease, I spoke
about the testing regimen with Raymond Dattwyler, an author of the Lyme
protocols who was on the 1994 CDC panel that wrote the test
guidelines. He was frank about its failures.
“’They were a stop-gap measure,’ he told me. ‘Twenty years ago I would’ve
said they’re fine. Now I say, ‘oh shit, we were wrong.’ It
doesn’t look as good as we thought it was….’”
”False positives: ‘Red herring’…
“… Benjamin Luft, a physician who wrote the original
Lyme disease guidelines in 2000 but who now sees them as flawed on the
issue of whether Lyme disease can persist, called the false positive issue
‘a red herring.’
“’I think there’s a great deal of benefit for early
treatment than delay,’ he said in an interview. ‘It does become
progressively more difficult [to treat] as time goes on.’
“red herring” is that no treatment really helps even if caught early. Half
still go on to chronic illness (Dattwyler, Luft, UCSF’s Chiu), but Dearborn
was research fraud that was performed to ALSO (also in
addition to deny all treatment, early – as shown by excluding all the IgM
cases -, and late neurologic Lyme) put out a “vaccine” that caused the same
chronic, neurologic, immunosuppression disease.
All the victims, of tick bite and OspA “vaccine,” were slandered and libeled
in order to make those LYMErix-Post-Sepsis Adverse Events cases look like
The Dearborn case definition was created from research fraud.
The Klempner “re-treatment” “study” was research fraud and a false claim
(based on Dearborn).
The OspA vaccines were research fraud, false claims and assault (based on
Dearborn; OspA was Pam3Cys or a triacyl lipoprotein, which is a fungal TOXIN
that caused global immunosuppression or post-septic shock).
E: What is OspA?
A triacyl lipopeptide, which means handled by TLR2 and 1 and is fungal. A
recombinant fungal antigen could never have been a vaccine, and when tried
elsewhere faled in the same way LYMerix failed, by causing immunosuppression
and rendering people more susceptible to disease.
Tripalmitoyl Cysteine, or Pam3Cys means 3 palmitic acid groups or 3 groups
of basically, palm oil (the zig-zaggy lines refer to carbon-carbon-carbon,
each with 2 hydrogens on them).
This graphic comes from a Korean Chemistry journal not
listed in PubMed, talking about either HIV’s gp120 or a vaccine designed
with Pam3Cys added on, the language is confusing:
Extremely Hydrophobic Immunostimulatory Lipoidal Peptides by Matrix Assisted
Laser Desorption Ionization Mass Spectrometry
Anyone can go to the National Library of Medicine and
find that OspA is Pam3Cys or a triacylated lipopeptide and never could have
been a vaccine:
of Borrelia burgdorferi is a mucosal immunogen and adjuvant.
outer surface protein A (OspA) lipoprotein of
Borrelia burgdorferi, like cholera toxin and the heat-labile enterotoxin of
Escherichia coli, induces pro-inflammatory cytokines. This suggested that,
like those toxins, OspA might
be a mucosal immunogen and adjuvant. OspA,
administered intranasally (i.n.) or intragastrically, induced strong serum
IgG and salivary gland IgA responses. The serum IgG isotypes were indicative
of a mixed T helper 1 and T helper 2 response, the latter being more
pronounced. The N-terminal tripalmitoyl-S-glyceryl-cysteine (Pam3Cys)
lipid moiety was absolutely required. OspA strongly
enhanced the serum IgG and salivary gland IgA responses to jack bean urease
co-administered by the i.n. route. OspA also
enhanced the response to tetanus toxoid and induced limited protection
against challenge. A synthetic lipopeptide also adjuvanted the response to
urease by the i.n. route, but was ca 500-fold less potent on a molar basis
These results suggest that OspA or
other lipoproteins may be useful in mucosal vaccines.”
Infect Immun. 1999
Induction of pro- and anti-inflammatory cytokines
by Borrelia burgdorferi lipoproteins in monocytes is mediated by CD14.
previously showed that heat-killed Borrelia burgdorferi spirochetes and
lipidated outer surface protein A (L-OspA)
stimulated the in vitro production of interleukin-10 (IL-10) in peripheral
blood mononuclear cells (PBMC) from uninfected humans and rhesus monkeys (G.
Giambartolomei et al., Infect. Immun. 66:2691-2697, 1998). Here we
demonstrate that uninfected human peripheral blood monocytes, but not B or T
cells, are the cells that transcribe the IL-10 cytokine gene in response to
heat-killed B. burgdorferi. B. burgdorferi similarly induced an upregulation
of the IL-1beta and IL-6 cytokine genes in monocytes and the production of
IL-10 and IL-6 in culture supernatants of the human monocytic cell line
THP-1. Purified L-OspA (but
not unlipidated OspA [U-OspA]
or U-OspC) also stimulated the production of both cytokines in THP-1 cells
in a dose-dependent fashion, suggesting that acylation of the OspA protein
molecule is required for the production of both anti- and pro-inflammatory
cytokines in naive monocytes. A lipohexapeptide that contained the
tripalmitoyl-modified cysteine motif (Pam3Cys-Hex)
of B. burgdorferi lipoproteins but with an arbitrary peptide sequence had
the same effect. Monoclonal antibodies (MAbs) MY4 and 60bca, both of
which bind to CD14 and are known to block lipopolysaccharide (LPS)-mediated
cytokine production, were able to block L-OspA-mediated
IL-10 and IL-6 cytokine production. In contrast, MAb 26ic, which also binds
to CD14 but does not block LPS function, failed to inhibit L-OspA-mediated
cytokine production. These data suggest that activation of monocytes and
production of both anti- and pro-inflammatory cytokines induced by
lipoproteins proceeds via the CD14 receptor. LPS binding protein was not
required for OspA-induced
cytokine production. Our results demonstrate that pro- and anti-inflammatory
cytokines induced by B. burgdorferi lipoproteins in PBMC are produced by
monocytes and that lipoprotein and LPS signaling pathways share at least the
initial signaling event that involves the CD14 receptor.
those 2 reports mean are that:
1) that Pam3Cys, or OspA is 500 times more toxic when injected than inhaled
(think mold inhalation could be bad, too, but not directly as devastating),
and that you needed the Pam3Cys or the fatty acids in order for OspA to be a
the second report means that it does not matter what the protein end is,
it’s not very immunogenic by itself. It’s the Pam3Cys or the central
structure shown here (above) that is the poison.
After the initial “cytokine storm” of Lyme or LYMErix sepsis, this:
J Innate Immun. 2016;8(2):171-84.
doi: 10.1159/000440838. Epub 2015 Oct 13.
Endotoxin Tolerance Inhibits Lyn and c-Src
Phosphorylation and Association with Toll-Like Receptor 4 but Increases
Expression and Activity of Protein Phosphatases.
protects the host by limiting excessive “cytokine storm” during sepsis, but
compromises the ability to counteract infections in septic shock survivors.
It reprograms Toll-like receptor (TLR) 4 responses by attenuating expression
of pro-inflammatory cytokines without suppressing anti-inflammatory and
antimicrobial mediators, but the mechanisms of reprogramming remain unclear.
In this study, we demonstrate that induction of endotoxin tolerance in human
monocytes, THP-1 and MonoMac-6 cells inhibited lipopolysaccharide
(LPS)-mediated phosphorylation of Lyn, c-Src and their recruitment to TLR4
but increased total protein phosphatase (PP) activity and expression of
protein tyrosine phosphatase (PTP) 1B, PP2A, PTP non-receptor type (PTPN)
22, and mitogen-activated protein kinase phosphatase (MKP)-1.
“During infection and
inflammation, microbial components and endogenous “alarmins” activate
membrane-associated Toll-like receptors (TLRs) expressed on macrophages,
dendritic cells, and neutrophils to activate innate immunity and prime
adaptive immune responses .
TLRs contain an ectodomain with multiple leucine-rich regions involved in
ligand sensing and co-R interactions, a transmembrane domain, and a
cytoplasmic tail with a signaling Toll-IL-1R (TIR) domain [1,2].
TLR2 cooperates with TLR1 or TLR6 to detect tri- and di-acylated
lipoproteins expressed by Gram positive bacteria, mycobacteria and
As you can see ^^^ TLR2/1 handles triacyl
lipoproteins, which are basically fungal. In any event, this is all
well-known to the infectious diseases science world. Anyone can look up
what OspA is, find it is handles by TLR2 and 1, and therefore is
fungal-ish. Humans cannot be injected with fungal antigens. They’re
Why did the Lyme criminals think they could go ahead
and do this and no one would see what they were up to? The first
reason is that you can’t re-crystallize a lipid (butter never exactly
freezes along cleavage lines like a true solid). The normal way to get a
structure is by X-ray diffraction studies, but this can’t be done with an
amorphous blob. Therefore the bad guys SAY, and HAVE
SAID (Barbour), “We don’t know what OspA is”.
They can’t now say what it is, can they? The SECOND
they say “OspA is Pam3Cys,” the whole world will know it could never have
been a vaccine and Yale must have lied about it to the FDA (which of course
they did). Even the NIH and CDC to this day (Yale, too) when asked, say “We
don’t know what OspA is.” Why do they say that? Think. ‘Pretty
embarrassing for no one to have been watching what Edward McSweegan was up
to – in charge of Lyme grants at the NIH !!
What other vaccine attempts with the likes of Pam3Cys or where the likes of
the immune suppressing OspA has been co-injected with viruses,…
and has this model of post-septic shock
or an immune suppressed state been seen to result in reactivated latent
viruses like Epstein-Barr and Cytomegalovirus?
At least four. And certainly – and we even see them every day on the
television (Humira, Stelara ads).
We call this an Occam’s Razor (“All roads lead to…,” or “If it walks like a
duck…”). Chronic Fatigue from post sepsis syndrome is the results of the
reactivation of these latent viruses. When people have the HLAs for a
hypersensitivity response to one or more of the opportunistic (see Fauci’s
patent) infections that take hold after immune suppressing sepsis, they
might have an AUTOIMMUNE or HYPERSENSITIVITY or ALLERGY response, such as to
EBV, such as MS or Lupus. This is why MS and Lupus are well known “Great
Imitator” (syphilis) and “New Great Imitar” (Lyme) outcomes. Less well
received by the “medical” community is the notion that one might have a
disease without inflammation (See Shapiro, above).
THREE Tuberculosis (remember, MYCO means fungi) failed
Induction of an immune response to the 19-kD antigen by an alternative
approach of DNA vaccination had no effect on subsequent M. tuberculosis
challenge. These results are consistent with a model in which the presence
of the 19-kD protein has a detrimental effect on the efficacy of
vaccination with live mycobacteria.”
“… Thus, the immunosuppressive effect is dependent on glycosylated and
acylated 19-kDa lipoprotein present in the phagosome containing the
mycobacterium. These results suggest that the diminished protection against
challenge with M. tuberculosis seen in mice vaccinated with M. smegmatis
expressing the 19-kDa lipoprotein is the result of reduced TNF-alpha and
IL-12 production, possibly leading to reduced induction of T-cell activation.”
“… This study indicates that the 27-kDa antigen has an adverse effect on the
protection afforded by recognized vaccines.
We are currently studying how the 27-kDa antigen modulates the mouse immune
Clifford Harding on how fungal toxins like OspA turn off the immune response
to viruses like EBV:
”Because IRAK1 is required for
TLR7/9-induced IFN-I production, we propose that TLR2 signaling
induces rapid depletion of IRAK1, which impairs IFN-I induction by
TLR7/9. This novel mechanism, whereby TLR2 inhibits
IFN-I induction by TLR7/9, may shape immune responses to microbes that
express ligands for both TLR2 and
TLR7/TLR9, or responses to bacteria/virus coinfection.”
An experiment injecting mycoplasma and live viruses
THE RELATIONSHIP OF EPERYTHROZOON COCCOIDES TO THE HEPATITIS VIRUS OF
“In Swiss mice, animals with high natural resistance to hepatitis virus, the
pathogenicity of this agent was markedly enhanced by combined infection with
eperythrozoa. Eperythrozoa were maintained throughout 18 successive passages
in normal Princeton and Swiss weanlings with intact spleens. The combined
infection of Princeton mice with eperythrozoa and the virus component of
Gledhill, Dick, and Andrewes, which is nearly inactive when injected alone,
resulted in acute hepatitis with fatal outcome.”
’Which is why Thimerosal is put in vaccines – to prevent fungi -,
since the immunosuppression results in reactivated viruses and this is
actually a well-known thing.
Where else do we see immunosuppression resulting in
reactivate latent herpesviruses?
Stelara, Humira (“lymphoma” warnings, which means EBV) commercials,
transplant victims. hospital sepsis victims, astronauts and medical school
student due to their cortisol (stress !!) induced immunosuppression (Chronic
Fatigue from reactivated Mono) and disruption of their sleep-wake cycle…
You see it and you hear about it every day.
Only one thing predictably happens in
immunosuppression: EBV, et al, have a free for all. And guess what, there
is no vaccine against it, since such prevention would put BigPharma out of
business, wouldn’t it.
Mono: Symptoms, Diagnosis and Treatment Options Sep, 2014
"People believe that it is common to have a chronic mono infection that can
cause problems for years. This is really not the case," said Hymes. EBV is a
viral illness in the same family as herpes and varicella (chickenpox). Like
those other members of the herpes virus family, the virus can stick around
in your body, Hymes explained. However, the virus is dormant and only very
rarely reactivates, usually in times of stress. "Chronic active
Epstein-Barr infection is something we really only see in people with immune
problems — transplant patients, HIV patients, or people born missing a part
of their immune system or immune function," said Hymes. Lab testing can
detect chronic presence of the virus and can rule it out, so it is easily
“Some studies suggested that acute infectious mononucleosis and Epstein-Barr
virus may permanently alter or impair the immune response. A 2007 Danish
study looked at the possible correlation between multiple sclerosis and
infectious mononucleosis and found that the risk of multiple sclerosis was
persistently increased for more than 30 years after contracting infectious
“Mono may also be responsible for chronic fatigue syndrome. EBV
persistence in the muscles biopsy samples, when looked at in patients with
post-viral muscle fatigability, is seen far less commonly (9%) then in
patients recovering from enterovirus infections (24%) according to a
study done by the Department of Biochemistry at Charing Cross and
Westminster Medical School, London, UK.
The “Three Times More Coxsackie in the Muscles Than
Post-Sepsis EBV-ish Chronic Fatigue/ME Syndrome” Report referenced above:
Br Med Bull. 1991 Oct;47(4):852-71.
Persistent virus infection
of muscle in postviral fatigue syndrome.
Cunningham L1, Bowles
“Nucleic acid was extracted
from muscle biopsy samples from a series of highly selected patients
suffering from chronic muscle fatiguability following a viral infection
(Postviral Fatigue Syndrome: PVFS). Samples were examined for the presence
of enteroviral RNA sequences or Epstein-Barr (EBV) virus DNA
sequences by molecular hybridisation as these two agents have been
implicated by retrospective serology in the aetiology of PVFS. We found
enteroviral RNA in 24% of biopsy samples and EBV DNA
in a further 9% of biopsy samples: no biopsy was positive for both
enteroviral RNA and EBV DNA.
In addition, in the case of enteroviruses we found that the persisting virus is
defective in control of RNA replication as both strands of enteroviral RNA
are present in similar amounts: this is unlike the asymmetric synthesis of
genomic RNA seen in a productive, cytolytic enterovirus infection.
The implications of these data in relation to mechanisms of viral
persistence and muscle dysfunction are discussed.”
J Neurol Neurosurg Psychiatry. 2003
Enterovirus related metabolic myopathy: a
postviral fatigue syndrome.
Lane RJ1, Soteriou
“To detect and characterise enterovirus RNA in skeletal muscle from
patients with chronic fatigue syndrome
(CFS) and to compare efficiency of muscle energy
metabolism in enterovirus positive and negative CFS patients.
Quadriceps muscle biopsy
samples from 48 patients with CFS were processed to detect enterovirus RNA
by two stage, reverse transcription, nested polymerase chain reaction
(RT-NPCR), using enterovirus group specific primer sets. Direct nucleotide
sequencing of PCR products was used to characterise the enterovirus.
Controls were 29 subjects with normal muscles. On the day of biopsy, each
CFS patient undertook a subanaerobic threshold exercise test (SATET). Venous
plasma lactate was measured immediately before and after exercise, and 30
minutes after testing. An abnormal lactate response to exercise (SATET+) was
defined as an exercise test in which plasma lactate exceeded the upper 99%
confidence limits for normal sedentary controls at two or more time points.
samples from 20.8% of the CFS patients were positive for enterovirus
sequences by RT-NPCR, while all the 29 control samples were negative; 58.3%
of the CFS patients had a SATET+ response. Nine of the 10 enterovirus
positive cases were among the 28 SATET+ patients (32.1%), compared with only
one (5%) of the 20 SATET- patients. PCR products were most closely related
to coxsackie B
There is an association between abnormal lactate response to
exercise, reflecting impaired muscle energy
metabolism, and the presence of enterovirus sequences in muscle in
a proportion of CFS patients.
Conclude: Coxsackie/Enteroviruses plus reactivated
Epstein-Barr, Cytomegalovirus etc., may all contribute to these serious
illnesses. And although we have the analytical methods to look for more
common viruses/fungi and the biomarkers (sub-immunity, plus biomarkers of
CNS degradation), patients with “Chronic Lyme” or CFIDS/ME are not taken
seriously. They’re abused and slandered against instead. The mechanisms of
illness in these conditions betray the source of the Autism pandemic from
the childhood vaccines. These are Color of Law abuses to hide Fraud.
Additionally, spirochetes target the lymph nodes, and
damage the B cell maturations or germination centers there, rendering the
victims unable to fight off non-fungal infections, says Ucal, Davis:
PLoS Pathog. 2015
Jul 2;11(7):e1004976. doi: 10.1371/journal.ppat.1004976. eCollection 2015.
Suppression of Long-Lived Humoral Immunity
Following Borrelia burgdorferi
Elsner RA1, Hastey
KJ2, Baumgarth N3.
Lyme Disease caused by infection with Borrelia burgdorferi
is an emerging infectious disease and already by far the most common
vector-borne disease in the U.S. Similar to many other infections, infection
with B. burgdorferi results in strong antibody response induction, which can
be used clinically as a diagnostic measure of prior exposure. However,
clinical studies have shown a sometimes-precipitous decline of such
antibodies shortly following antibiotic treatment, revealing a potential
deficit in the host's ability to induce and/or maintain long-term protective
antibodies. This is further supported by reports of frequent repeat
infections with B. burgdorferi in endemic areas. The mechanisms underlying
such a lack of long-term humoral immunity, however, remain unknown. We show
here that B. burgdorferi infected mice show a similar rapid disappearance of Borrelia-specific
antibodies after infection and subsequent antibiotic treatment. This failure
was associated with development of only short-lived germinal centers,
micro-anatomical locations from which long-lived immunity originates. These
showed structural abnormalities and failed to induce memory B cells and
long-lived plasma cells for months after the infection, rendering the mice
susceptible to reinfection with the same strain of B. burgdorferi. The
inability to induce long-lived immune responses was not due to the
particular nature of the immunogenic antigens of B. burgdorferi, as
antibodies to both T-dependent and T-independent Borrelia antigens
lacked longevity and B cell memory induction. Furthermore, influenza
immunization administered at the time of Borrelia infection
also failed to induce robust antibody responses, dramatically reducing the
protective antiviral capacity of the humoral response. Collectively,
these studies show that B. burgdorferi-infection results in targeted and
temporary immunosuppression of the host and bring new insight into the
mechanisms underlying the failure to develop long-term immunity to this
emerging disease threat.
Diseases caused by
(Cocksackie, Foot and Mouth Disease)
via the fecal-oral route
Polio-like syndrome found
in children who tested positive for enterovirus
Nonspecific febrile illness
is the most common presentation of enterovirus infection. Other than fever,
symptoms include muscle pain, sore throat, gastrointestinal
distress/abdominal discomfort, and headache. In newborns the picture may be
that of sepsis however,
and can be severe and life-threatening.
Enteroviruses are by far the most common causes of aseptic
children. In the United States, enteroviruses are responsible for 30,000 to
50,000 meningitis hospitalizations per year as a result of 30 million to 50
Bornholm disease or epidemic
characterized by severe paroxysmal pain in the chest and abdomen, along with
fever, and sometimes nausea, headache, and emesis.
Pericarditis and/or myocarditis are
typically caused by enteroviruses; symptoms consist of fever with dyspnea and chest
heart failure, and myocardial infarction have also been reported.
Acute hemorrhagic conjunctivitis can
be caused by enteroviruses.
caused by Coxsackie A virus, and causes a vesicular rash in the oral cavity
and on the pharynx, along with high fever, sore
and often dysphagia,
loss of appetite, back pain, and headache. It is also self-limiting, with
symptoms typically ending in 3–4 days.
Hand, foot and mouth disease is
a childhood illness most commonly caused by infection by Coxsackie A virus
rare manifestation of enterovirus infection; when it occurs, the most
frequent enterovirus found to be causing it is echovirus
A 2007 study suggested that acute respiratory or gastrointestinal
infections associated with enterovirus may be a factor in chronic
Diabetes mellitus type 1 It
has been proposed that type 1 diabetes is a virus-triggered autoimmune
response in which the immune system attacks virus-infected cells along with
the insulin-producing beta cells in the pancreas. A team working at University of Tampere, Finland has
identified a type of enterovirus that has a possible link to type 1 diabetes
(which is an autoimmune disease).
More on enteroviruses, possibly in ticks:
THAT is your “Health and Human Services”
or HHS.gov, being 200% incompetent to all major issues of every day life and
health: Cancer, Chronic Fatiguing post-sepsis which by their own accounts,
affects 12 million of us, excluding Lyme -, and easily half of all
THAT is why our YouTube channel is
“HHSonthecouch,” They’re frickin mental.
Oh, and Allen Steere has never discovered how Lyme or
OspA could cause arthritis in 42 years. He’s still looking, though, in his
“high security” T cell freezers.
Yves Lobet at SmithKline said of Allen Steere’s “autoimmune bad knees”
proposal (hLFA-1) at the FDA meeting in Jan 2001 on LYMErix adverse events,
that if OspA created autoimmune response against something on an HLA
antigen, then this phenomenon would be seen all throughout the body and not
just in the knees. He basically made fun of Steere. Find that here:
Restate the Charges:
Racketeering (ALDF.com is a fake non-profit, just a commercial front),
Defrauding the Government with false claims for grants based on Dearborn,
Color of Law (slander and libel, denial of rights),
Assaulting children with a known fake vaccine just to see how bad would be
the adverse events.
The fake toxin of a
vaccine, Pam3Cys has always been known to be a toxin and never a vaccine.
Thimerosal was put in
vaccines to prevent LYMErix.
Demand a Senate hearing to refer to the USDOJ,
mention that a lack of action on this will risk their “future electability,”
and will “question the competency” of the Senator/Congressmen you’re
lobbying. Murder, permanent disability, denial of rights, slander,
assaulting children … for profit is not acceptable. Make sure this
is clear. Look them straight in the eye. They work for you.
Literally, their JOB is to do YOUR bidding.
LOBBYISTS - what
is required of you:
1) Lobbyists are expected to be the “best-of-the-best,”
Navy Seals type Lyme warriors. The goal is to put the criminals in
2) No lobbyist will be allowed to talk about themself, even if asked.
Personal experience is anecdote, not data. Quit/Stop right here and go no
further if you don’t understand what that means.
3) No lobbyist can object to the science we present here because none of the
publications are ours. Everyone should their objections to the authors of
the science we re-present.
4) We only want top-shelf people: no cowards, no
fairies, no effeminate types who think drama and personal experience counts
for anything. They absolutely DO NOT.
No prosecutor gets up in front of a jury and judge and
starts talking about his prostate problems while prosecuting a drugs charge.
SHOWS what the bad guys did. The only
kind of people we want are the ones with the balls to stick to the script
and not insert themselves into the equation. This is a CHARACTER issue,
only. Not a talent issue. Tell the story of THE
You can volunteer to be a lobbyist for these causes
(Lyme, Autism, CFIDS, ME/CFS, Fibro, etc.), but you must be trained as to
what exactly needs to be prosecuted and can present it in 5 or 10 minutes.
The goal is a Senate hearing for referral to the DOJ (think: this will go on
CSPAN). There are a lot of issues, but essentially, where CDC staff
members committed fraud to either advance their own personal prestige or
income agenda or BigPharma's, that's what we want prosecuted. We use
the CDC's own data to show what crimes they and their associates committed.
This Lyme Cryme story has never been properly re-told in any book or by any
journalist, but the facts of what happened at Dearborn have been available
to the public for over 20 years. How Steere falsified the testing in Europe
was available, what OspA is has been available since around 1999.
But not until 2005 did this author find its structure because it was not
shown in any OspA patent. A triacyl-lipopeptide could never have been a
vaccine if it is a fungal antigen (managed by TLRs 1 & 2). Except for the
scientists we quote (Latov, Marks, Harding, Medvedev, Persing, Schoen, etc
on the “Occam’s Razor” report), no "doctor" has ever explained why LYMErix
failed as a fungal antigen.
Tthe most remarkable aspect of this crime of all: Not
one single person in the USA or Canada or even Europe that we know of, with
"MD" after their names, has a clue what's going on and none ever ask. They
don’t ask, they don’t wonder. This tells you there is probably plenty more
about which "MDs" are clueless and prefer to remain so. [Turns out,
OHYEAH, there's plenty in plain sight they’re not seeing (Humira,
Stelara, MMR monograph warnings, there is no NIIID, ...)].
TAKE HOME POINT: The Klempner "long-term” “treatment” study" became the
basis of the Infectious Diseases Society of America's (IDSA's) "Guidelines"
on the “diagnosis and treatment of Lyme disease.” Here we will show the
case definition (diagnosis criteria. “Dearborn,” 2-tiered) was falsified,
and therefore there can be no guidelines on treatment or diagnosis. If the
“guidelines” are gone, insurance companies cannot deny care or testing