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Subject: The Science in ME/CFS parallels that in Vaccine-Damage/Autism
Date: Dec 12, 2014 8:20 AM
Regarding these new reports of yours:
CFIDS/FM is/are not “neuro-endocrine-immune diseases.” They/it are really actually chronic active herpesviruses, immunosuppression with opportunistics, much like AIDS (no T cell killer virus, just T cell anergy).
First, have a look (in the references, here) at the recent description of “post-sepsis syndrome” (PSS) and the statements/reports by Washington University St. Louis, MO. (wustl.edu) and the NIH confirming that model on what this disease is, how it comes about, (1-8), the older immunosuppression data on fungi like mycoplasma and mycobacteria- formerly called epERYTHROzoons - clue (9, 1953), past fungal vaccines failures and what those failure-outcomes looked like (10-14), and of course the Autism pandemic, which parallels all these mechanistically (15-21, 51).
I would like you to note that in this report, I put a lot of the explanations in the references section. Since CFIDS people have cognitive issues (53), particularly right brain issues or visual-spatial processing deficits, I have to make the overall picture as brief as possible in the body of this report.
CDC, short for “SMELLS LIKE BULLSHIT”
First, you have to be convinced that the CDC is lying about everything if you aren’t already. One of the most blatant examples was when a one CDC officer Susan Vernon tried to prove mycoplasma (epERYTHROzoons) were not involved in ME/CFS by first throwing out the blood cells to which mycoplasma adhere or hide within, when trying to falsely claim mycoplasma do not cause chronic fatigue (22). Of course they cause fatigue. They adhere to red blood cells and change the osmotic potential of the red blood cell membrane, rendering oxygen unable to cross the membrane, they’re within cells, they hijack sugar metabolism, they inhibit apoptosis of infected cells (23, 24), they mess up mitochondria (25).… There’re plenty of references on these pathologies available to be discovered in PubMed, the National Library of Medicine.
We’ve caught the CDC lying about so many things – vaccines, Lyme, ME/CFS, Autism (again, fungi); Thimerosal is put in vaccines, to prevent fungi because when you inject live, attenuated viruses together with fungi the viruses become reactivated (21).
The CDC still lies about the viability of the borrelial cyst or spheroplast form. In 1964 the CDC showed us how to create these viable spheroplasts (26). Yet today, the CDC and their crooked counter parts say the Borrelia DNA found in post-treated Lyme patients are “dead DNA.” That never happens. Such debris is consumed/phagocytized right away, unless it is incorporated by the host (cancer), or is present in immortalized cells – EBV or mycoplasma transformed or pre-cancerous cells – and at that you still will have ILLNESS, right? And not some weird hypothetical linguistified garbage such as from the DSM that’s code language for “witch” (somatoform, ie.).
The CDC lies about who should get a flu shot. We’ve all seen families where when the old Grandma gets a flu shot, everyone else in the family comes down with that very same vaccine virus. This works out well for like Walgreen’s, who then sell everyone who got Vaccine-Grandma-Flu all the over the counter flu remedie$.
Even DARPA wants nothing to do with the CDC regarding disease detection and have stated that they will only be working with the NIH and FDA on that from now on (27). Note – this used to be CDC’s job, being on the lookout for new infectious diseases. Now the US Military doesn’t want the CDC involved. We’ll follow their lead. Leave CDC out. Ignore. They’re criminals and have no talent (redundant).
Major Clue that associated for us, CFIDS and Lyme: OspA – THE BOMB!!
Call OspA or the fake LYMErix vaccine, THE BOMB!! or the Great Detonator that solved the 500 year old medical
mystery of Syphilis as the Great Imitator. You will recall that the CDC Crooks Inc say chronic Lyme is really Fibromyalgia (DSM) or CFIDS (DSM). But the biggest clue as to what these waste-basket illnesses are was that the LYMErix (OspA or Pam3Cys or a TLR2/1-agonist fungal antigen) non-vaccine [ordered off the market by the FDA under an ultimatum in 2002 (28), caused the same chronic, “protean,” “multi-system” disease as “Chronic Neurologic Lyme (CNL)]” (29-33). This chronic-fatiguing-illness-just-like-Chronic-Lyme outcome in LYMErix injured people, we learned about independently, in the spring of 1999 when LYMErix first came on the market. Those sick people called Lyme support group leaders, that is, complaining that they had “Lyme again,” after getting that non-vaccine, LYMErix. They had SYSTEMIC LYME, not bad knee…
??? ‘Didn’t make any sense since this was a recombinant antigen, not spirochetes, this “vaccine.”
Later we learned the CDC’s false-front non-profit, the ALDF dot com/Yale (Mayo Clinic included) crooks already knew all about the fact that LYMErix caused systemic disease as shown in those references, 29-33. In 1993, CDC officer Alan Barbour and Yale’s Durland Fish trashed CNL patients prior to the Dearborn event basically calling them nuts, while admitting these OspA vaccine trials were already underway (34). By 1994, CDC officer Allen Steere had gone to Europe to falsify the testing (35, 36) for the future CDC’s Dearborn, MI, farce of a consensus conference (37).
That a recombinant antigen could cause Chronic Systemic Lyme, was the mystery that, solved, solved these other diseases like ME, CFIDS, Fibro, Gulf War Illness – those with “The Triad” of Chronic Fatigue, Neurologic and Cognitive, and Musculoskeletal signs. One does not have to have Lyme or LYMErix to have this outcome, it could have been caused by a bad vaccine or from living in a moldy home or both or all of them. It does not really matter, the result is the same – something like Post Sepsis Syndrome, which is ongoing active infections and nothing really “post” about it at all.
LET’S BACK UP TO BLEBBING AND THE AUTO-VACCINATION WITH OSP-A-like molecules:
We should back up here and explain about spirochetal blebbing or the auto-vaccination with OspA and shed similar fungal antigens like it. Many people with Lyme – and especially not our self-alleged “treaters,” ILADS.org - do not understand this basic biology of Relapsing Fever. Let’s quote the expert, the owner of the most patents for vector borne disease vaccines and test kit antigens than anyone in the world, CDC officer and former head of the NIH’s Rocky Mountain Bioweapons Lab, Alan Barbour, the same guy who trashed us with Durland Fish in 1993 to sell bad vaccines (34):
In a 1986 publication called the “Biology of Borrelia species,”(39) where Barbour says to treat Syphilis with attenuated (high passage) borrelia to raise a fever (since antibiotics don’t work), he shows Borrelial blebs and cysts or spheroplast or “gemma” are regeneration forms.
In that same report he advised treating syphilis with attenuated relapsing fever borrelia (Lyme) in order to raise a fever (OspA-like antigens are endotoxins or pyrogens or can cause something close to sepsis, clue) which means he knows antibiotics don’t work.
In 1996, in an article in “The Scientist,” Barbour and Barthold talked about the shedding or blebbing off of OspA-like-antigen-covered antigens and antigenic variation (40) in this way:
"It's using some sort of stealth-bomber-type mechanism," he says. Or, using another diversionary tactic called blebbing, the spirochete can pinch off bits of its membrane in order to release its surface proteins. Explains Barbour: "It's like a bacterial Star Wars defense program," in which released surface proteins might intercept incoming host antibodies, keeping the spirochete safe from immunological attack.”
They also say in the same article (40) that the spirochetes hide in the brain which is an immune privileged site. Of course Barbour would know, since he reported that rodent brains used to be the storage media, spirochetes so reliably infect the brain (39). If the spirochetes are on the brain, we can’t use a culture method from blood. Therefore, the only diagnostic method that could be validated would have to be Borreliae-specific recombinant flagellin-based to capture these specific antibodies, the only ones that are not variable.
FUNGAL-VIRAL SYNERGY? POST-SEPSIS SYNDROME?
Now we know these waste-basket-, DSM-Witchmajick- diseases are really about fungal-viral synergy as it was recently well described by wustl, the NIH, and the other references you’ve seen. It’s about chronic active herpes viruses; it’s a pre-leukemia-like disease with “Epstein-Barr-like-transformed cells in CNS” (2,3); with cross-tolerance from fungal tolerance (TLR2/1) expanded over to TLR4, and TLR7/9 agonist tolerance (41, 42) such that you can’t really deal with any infections.
This disease - let’s call it Post Sepsis Syndrome because the NIH does - is not really about chronic “auto-immune”-like inflammation in the majority of us However, in the HLA-linked people, some of the subsequent-to-sepsis or secondary-to-fungal infections can cause this, such as mycoplasma or EBV, CMV, or HHV-6. For example, you might not be HLA-predisposed to HLA-linked Rheumatoid Arthritis from fungi, mycoplasma or OspA, but you might have the MS HLAs or the classic hypersensitivity response and allegedly auto-reactive T cells as a secondary response to the HERPESVIRUSES that have been activated. That is, the secondary infections activated by a toxic event like living in a moldy home or a exposure to a bad (fungal contaminated) vaccine could cause you to have an allergy response to say Epstein-Barr or mycoplasma or whatever, but in those minority of HLA-linked cases, the victims are nowhere near as severely ill as the people without those disease-specific HLA. You probably have noticed that MS victims do not complain of such severe fatigue as ME/CFS/Lyme victims do. The reason is, we were told at the 1998 FDA Meeting on LYMErix by a one Vijay Sikand, is that these HLA-linked people “make enough antibodies to fight off the disease.” But the vast majority of us do not have these disease-associated HLAs that will produce hypersensitivity responses with the lots of antibodies. Instead we have no antibodies. And that’s all meant to be a huge secret since it reveals the source of the Autism pandemic, as previously mentioned.
As another example of the association between HLA-linked disease and non-HLA-linked disease, in 2005, a one CDC officer Mark Klempner reported, finally, his HLA results from his 1997, 4.7 million dollar research fraud grant. Klempner used the Dearborn “Lyme Disease” criteria - which was research fraud -, gave 2/3 of his victims the minimum, Standard of Care, 30 days of IV ceftriaxone and called that “re-treatment” (43). Two-thirds had never had this standard of care 30 days of IV ceftriazone so it could not have been “re-treatment” in 2001. In 2005, with data from the same “study,” Klempner mentioned that the people with the HLA-linked arthritis outcome (the current, CDC- falsified 1994 “Dearborn,” “Lyme Disease” case definition is ONLY this HLA-linked arthritis, alone), only have “bad knees” – arthritis - and no other symptoms (44). No fatigue, No brain signs, etc. It took 11 years and an extra 5 million dollars for the CDC/Klempner to re-report that “Lyme Disease,” their Dearborn falsified case definition where ONLY people with ONLY HLA-linked late Lyme arthritis cases are allowed to be a “case” of “Lyme Disease,” meant “only HLA-linked arthritis and no other symptoms.” The rest? The 85% without the disease specific HLAs and are the sickest? They have a non-disease, and are “psychiatric.” You knew that.
NO NIIID, JUST SILLY DISEASES,… AND THE AUTISM PANDEMIC
There is no National Institute of Immunosuppression and Infectious Diseases (NIIID), there is only ALLERGY – the opposite – and Infectious Disease.
Because these mechanisms explain the mechanisms behind the Autism pandemic. You know this for sure because while everyone talks about “personalized medicine,” including Francis Collins, Director of the NIH, all the pediatric vaccines are one- size-fits-all. One in 50 kids is brain damaged for life, and there have got to be 30 million Americans with Lyme, ME/CFS/FM, Gulf War Illness and a hodge podge of other Silly Diseases like Ehud Dammit or whatever you callit, Mitochondrial-how-bout-Epstein-Barr, and Mother effer gene methylation baloneyitis. .. all because the CDC wants to sell vaccines.
All this abuse and disability seems to only be a bargain for Walgreens and BigPharma, but the Social Security Administration and Medicare pick up the tab on us, - we, the CEO’s and the investors’ Collateral Damagees.
Recall that if you’re not very sick with these same infections, you can get an MS diagnosis, clue, and nobody kicks your ass literally to the curb, resulting in homelessness and all kinds of other distresses before you’re approve for Disability. Most of us have to be tortured by the FINALLY, OFFICIALLY more totally illegitimate “MDs,” the psycho-projectionists / psycho-pervos, for months to years, first. NAZI Collaborators were also hung after Nurenberg, so we comfort ourselves with that imagery. (A day after I wrote this report we got the news about the sicko CIA torturers.)
How do the reactivated latent herpesviruses, mycoplasma, Candida (remember, that’s fungal, and you can’t ever fight them off, since TLR2/1 agonist tolerance is not reversible with any mab drug at present), cause chronic fatiguing disease, with dementia, neurologic signs and musculoskeletal complaints if it’s not via that more classical “auto-immune” route? There is one important article you should start off reading, as it was authored by 2 of the CDC’s bioweaponeers, Tully and Baseman, so it would be hard for the CDC to argue against:
“Mycoplasmas: sophisticated, reemerging, and burdened by their notoriety.”
“This host-adapted survival is achieved through surface parasitism of target cells, acquisition of essential biosynthetic precursors, and in some cases, subsequent entry and survival intracellularly.”
There are several mechanisms. We mentioned how the mycoplasma (bearers of TLR2/1 agonists and/or Pam3Cys) to which we have now become tolerized cause fatigue via O2-blocking (46, 47) and modifying cell metabolism. Epstein-Barr and similar herpes viruses also hijack cell metabolism. EBV is well known to cause chronic fatigue, MS and Lupus. EBV and mycoplasma cause mitochondrial dysfunction (48, 49). The reactive oxygen species are also well known to cause cell damage (50), not just the infected cell, but cells around it. In the brain, in particular, these immunosuppression diseases caused by Lyme or other fungal antigens do cause chronic inflammation with systemic immunosuppression and no antibodies (4-5). Lyme causes central nervous system (CNS) glial cell destruction. A spinal fluid marker for that (GFAp) was mentioned by none other than Yale’s Robert Schoen when trying to sell the LYMErix vaccine, mentioning how terrible Lyme is,… which is “only a knee disease and causes no other symptoms ” (45). You do not have to have tons of antibodies and a classic HLA-linked disease in order to be sick.
The NIH reported in 2006 that exposure to LYMErix caused inflammation in the brain but immunosuppression in the body or humorally (4). We just saw that result in the news recently about how Autism brains are chronically inflamed (51). That is so similar to the acquired brain damage seen in CFIDS people as recently reported even in the New York Times (52), although no one fact checked: speech is processed in the left hemisphere, while visual-spatial abilities are a right-brain function. These are not coincidences. We know what these diseases are. The proofs run in parallel. Another line of proof is in how all the Tuberculosis or other TLR2/1-agonists vaccines all failed. They all made their victims worse and more susceptible to diseases (11-14).
Lyme, CFIDS, FM, etc are in the cancer class – cancer being the opposite of classic autoimmune or HLA-linked allergy diseases.
30 million of us disabled with something like this Post-Sepsis with the chronic active infections,… and God only knows how many kids brain damaged for life, and there is no NIIIDS. These diseases are of NO-IMMUNITY. They are not “neuro-immune” and certainly not “neuro-endocrine.” It’s triple-fatigue from multiple infections and like a pre-cancer.
If you have any questions, please join us in the OCCCUPY the USDOJ Facebook group:
The OCCUPY is planned for next May-July 4th, in Washington, DC. Obviously the CDC is in cahoots with vaccine manufacturers as well as the insurance companies. This will be prosecuted or the foreign press can have something else to gnaw on besides the CIA torturers.
Kathleen M. Dickson,
former Pfizer Analytical Chemist and the Lyme
2) 1989 (this is in IDSA's own journal):
Clinical pathologic correlations of Lyme disease.
NCI and US Army Ft Detrick Pathologist Paul Duray on the CSF cells looking like "Epstein-Barr-like transformed cells" in IDSA's 1989 Reviews Supplement on Spirochetal Diseases: Rev Infect Dis. 1989 Sep-Oct;11 Suppl 6:S1487-93. "Immature B cells can also be seen in the spinal fluid. These cells can appear quite atypical- not unlike those of transformed or neoplastic lymphocytes." --
3) 1992: Duray again in 1992, in Steve Schutzer's review of the 1992 Cold Spring Harbor Conference on Lyme: "On occasion, these atypical-appearing large lymphocytes have been misinterpreted in biopsy by several laboratories as cells of a malignant lymphoma or leukemia. Bb antigens, then, may stimulate growth of immature lymphocytic suibsets in some target organs, as well as in the cerebrospinal fluid (Szyfelbein and Ross 1988). Usual bacterial infections do not produce such lymphocytic infiltrates in tissue. ****These immunoblastoid cells in Bb infections at times resemble those found in Epstein-Barr virus infections.**** Does Bb reactivate latent virus infections in tissues? Do some tick inocula harbor simultaneous infectious agents (ixodid ticks can harbor Rickettsiae, Babesia microti, and Ehrlichia bacteria, in addition to Bb), producing multi-agent infections in some hosts? Further studies can clarify these issues by mans of tissue-based molecular probe analysis." -
Paul Duray, NCI, NIH, Ft. Detrick, at the 1992 Cold Spring Harbor Crooks' Conference, published in Steve Schutzer's Lyme Disease: Molecular and Immunologic Approaches. - book.
4) 2006: The NIH (NINDS’s MS-Lyme Group) group that discovered that *** OspA *** was the cause of the MS/New Great Imitator outcome of Lyme reporting in the New York Times in the summer of 2013 (Martin and Marques, 2006);
this article says these OspA like antigens constantly shed by Borreliae cause immunosuppression in the humoral immune system, but apparently a chronic inflammatory state in the central nervous system:
"Borrelia burgdorferi Induces TLR1 and TLR2 in human microglia and peripheral blood monocytes but differentially regulates HLA-class II expression."
5) this report by the same group as the one above (4) means you might not even have anti-flagellar antibodies (flagellin is a TLR5-agonist) after being exposed to shed fungal OspA like antigens (TLR2/1-agonists):
"Borrelia burgdorferi lipoprotein-mediated TLR2 stimulation causes the down-regulation of TLR5 in human monocytes.
6) 2013 - Same NIH MS-Lyme Group as above, 4) and 5), Martin and Marques:
"When Lyme Disease Lasts and Lasts" – NYTimes, Jane Brody
"Complicating the picture is the fact that some people with PTLDS symptoms apparently never had Lyme disease in the first place, Dr. Marques said in an interview. There are other infectious organisms — Epstein-Barr virus, for example — that can produce similar symptoms and may be the real culprits."
7) 2014: Washington University, St. Louis, MO, discovers that sepsis is like Lyme, in that the survivors of it are likely to have survived via the immunosuppression (TLR2-agonist tolerance/Endotoxin tolerance), but the result is the reactivation of latent viruses:
"Dormant viruses re-emerge in patients with lingering sepsis, signaling immune suppression"
FULL JOURNAL REPORT
Reactivation of Multiple Viruses in Patients with Sepsis
8), NIH, 2014, "Surviving Sepsis: Detection and Treatment Advances" By Carolyn Beans for the National Institutes of Health | August 18, 2014 08:43am ET
9) IV. THE RELATIONSHIP OF EPERYTHROZOON COCCOIDES TO THE HEPATITIS VIRUS OF PRINCETON MICE "In Swiss mice, animals with high natural resistance to hepatitis virus, the pathogenicity of this agent was markedly enhanced by combined infection with eperythrozoa. Eperythrozoa were maintained throughout 18 successive passages in normal Princeton and Swiss weanlings with intact spleens. The combined infection of Princeton mice with eperythrozoa and the virus component of Gledhill, Dick, and Andrewes, which is nearly inactive when injected alone, resulted in acute hepatitis with fatal outcome."
10) past fungal vaccines failures and what those failures looked like ( long list)
11) Mycobacterium tuberculosis LprG (Rv1411c): A Novel TLR-2 Ligand That Inhibits Human Macrophage Class II MHC Antigen Processing1
The 19-kD antigen and protective immunity in a murine model of tuberculosis.
“These results are consistent with a model in which the presence of the 19-kD protein has a detrimental effect on the efficacy of vaccination with live mycobacteria.”
Mycobacterium tuberculosis 19-kilodalton lipoprotein inhibits Mycobacterium smegmatis-induced cytokine production by human macrophages in vitro.
14) The Mycobacterium tuberculosis recombinant 27-kilodalton lipoprotein induces a strong Th1-type immune response deleterious to protection.
15) CDC SAYS…on what causes Autism (You will see how it mirrors the other data on Lyme, LYMErix, and why the CDC/Vernon would claim mycoplasma do not cause disease):
CDC’s Patent, US # 7,632,510,
Methods of inducing flavivirus immune responses through the administration of recombinant flaviviruses comprising an engineered japanese encephalitis virus signal sequence
"Finally, there is the risk that the virus may not be fully or completely inactivated or attenuated and thus, the vaccine may actually cause disease."
16) CDC SAYS,…Measles, Mumps, and Rubella -- Vaccine Use and Strategies for Elimination of Measles, Rubella, and Congenital Rubella Syndrome and Control of Mumps: Recommendations of the Advisory Committee on Immunization Practices (ACIP)
"Updated information on adverse events and contraindications, particularly for persons with severe HIV infection, persons with a egg allergy or gelatin allergy, persons with a history of thrombocytopenia, and persons receiving steroid therapy [are immunosuppressed - KMD]."
17) CDC SAYS: Human Exposure to Brucella abortus Strain RB51 -- Kansas, 1997
[In the above, an immunosuppressed pregnant cow was given a Brucella (LYMErix-like) "live attenuated" vaccine and the baby cow ended up with the disease, which then was transferred to the humans handling the cow and her dead baby. This parallels what is happening to children who are vaccinated while immunosuppressed, or who receive mycoplasmally (LYMErix-like) contaminated vaccines -KMD.]
18) In the case of pandemic MRSA, as we have seen, the vaccine didn't work because TLR2 agonists (lipoproteins) suppress the immune system. We also learned from this MRSA vaccine patent, that (US patent 7,771,728, Intercell AG) that THERE IS A RISK OF REVERSION TO VIRULENCE OF LIVE ATTENUATED VIRUSES INJECTED INTO IMMUNOSUPPRESSED PERSONS:
Method for identification, isolation and production of antigens to a specific pathogen
"Several established vaccines consist of live attenuated organisms where the risk of reversion to the virulent wild-type strain exists. In particular in immunocompromised hosts this can be a live threatening scenario. Alternatively, vaccines are administered as a combination of pathogen-derived antigens together with compounds that induce or enhance immune responses against these antigens (these compounds are commonly termed adjuvant), since these subunit vaccines on their own are generally not effective."
19) Fauci just announced (Sep, 2014) huge new funding for adjuvants for vaccines for immunosuppressed children, duh, why would he do that if this was not a problem.
"NIH awards seven new vaccine adjuvant discovery contracts"
"The goal of this research is to identify novel adjuvant candidates that safely and selectively boost vaccine-induced immune responses,” said NIAID Director Anthony S. Fauci, M.D. “Such adjuvants could be used to improve current vaccines, extend the vaccine supply or enhance vaccine efficacy in people with immature or weakened immune systems, such as infants and the elderly.”
20) CDC SAYS that stress hormones activate viruses, which means it’s unlikely that people are causing illness with their magical brains, the current technical definition of “Somatoform”…
The effect of exogenous corticosterone on West Nile virus infection in Northern Cardinals (Cardinalis cardinalis)
“Corticosterone was administered at levels that individuals enduring chronic stressors (i.e ., long-term inclement weather, food shortage, anthropogenic pollution) might experience in the wild. Corticosterone greatly impacted mortality: half of the corticosterone-implanted cardinals died between five - 11 days post-inoculation whereas only one of nine sham-implanted (control) birds died. … No differences were found in viral titer between corticosterone- and sham-implanted birds. However, cardinals that survived infections had significantly higher average body temperatures during peak infection than individuals that died… In sum, this study indicates that elevated corticosterone could affect the survival of WNV-infected wild birds, suggesting that populations may be disproportionately at-risk to disease in stressful environments.”
[The same is true for humans and cortisol and the activation of latent herpesviruses, therefore it’s not “psychiatric” or “psychosomatic.” It’s reactivated EBV/similars due to cortisol, meaning real, and not due to having the powers of a witch.]
21) NYTimes; Doctors admit Thimerosal is put in vaccines to prevent fungi:
Vaccine Rule Is Said to Hurt Health Efforts (Dec, 2012)
"But a proposal that the ban include thimerosal, which has been used since the 1930s to prevent bacterial and fungal contamination in multidose vials of vaccines, has drawn strong criticism from pediatricians…. They say that the ethyl-mercury compound is critical for vaccine use in the developing world, where multidose vials are a mainstay…Banning it would require switching to single-dose vials for vaccines, which would cost far more and require new networks of cold storage facilities and additional capacity for waste disposal, the authors of the articles said.'"
[^^ They are revealing why so few doctors' own kids get vaccines-acquired brain damage. Their kids probably get the first shot out of the vial.]
22) CDC’s Lies: “Absence of Mycoplasma species DNA in chronic fatigue syndrome”, throwing out the blood cells in order to find no mycoplasma:
“Blood was collected in sodium citrate Vacutainer tubes (Beckton Dickinson) and shipped by overnight courier to the Centers for Disease Control (CDC), where plasma was collected by separation on lymphocyte separation medium (LSM; ICN Biomedicals). Plasma (1 ml) was concentrated to approximately 250 μl in a Centricon centrifugal filter unit YM-100 (Millipore). Cell-free plasma DNA was extracted by using a QIAamp DNA Mini kit (Qiagen) according to the manufacturer's instructions and quantified by using a DyNA Quant 200 fluorometer (Amersham Biosciences).”
23) mycoplasma inhibit apoptosis of infected cells, just like OspA, which is the first step in nearly all diseases:
The inhibitory effect of Mycoplasma fermentans on tumour necrosis factor (TNF)-alpha-induced apoptosis resides in the membrane lipoproteins.
“Mycoplasma have been shown to be involved in the alteration of several eukaryotic cell functions, such as cytokine production, gene expression and more. We have previously reported that infection of human myelomonocytic U937 cell line with live Mycoplasma fermentans (M. fermentans) inhibited tumour necrosis factor (TNF-alpha)-induced apoptosis.”
24) 2004 – PS: this was our first clue as to how LYMErix caused the same disease as Chronic Lyme:
Mycoplasma fermentans inhibits tumor necrosis factor alpha-induced apoptosis in the human myelomonocytic U937 cell line.
“In conclusion, M. fermentans significantly inhibits TNFalpha-induced apoptosis in U937 cells, and its effect is upstream of the mitochondria and upstream of caspase-8.”
25) Mitochondrial activities in human cultured skin fibroblasts contaminated by Mycoplasma hyorhinis
26) 1964, CDC, RECOVERY BORRELIA AND TREPONEMA AFTER LYOPHILIZATION
27) DARPA wants thing to do with the CDC:
Military and medical science meet to put human body on a chip
“Today, the US military and the National Institutes of Health (NIH) proposed developing just such a chip in a US$140 million effort over the next five years. The Pentagon’s storied Defense Advanced Research Projects Agency (DARPA) is teaming with NIH — a first — to execute the project, with each agency committing $70 million. The NIH contribution would come from its director’s discretionary ‘Common Fund’, but be administered through a proposed new translational medicine centre at the biomedical agency. The Food and Drug Administration will also play a part, advising the agencies on how to meet its requirements for safety and effectiveness.”
28) Karen Forschner of The Lyme Disease Foundation discovers “LYMErix is Toxin” in Bridgitte Huber’s patent, US 6,689,364, resulting in the FDA giving SmithKline and Yale, the patent owner, an ultimatum to either remove LYMErix voluntarily or the FDA would order it off the market themselves:
“The present invention provides novel polypeptides which are substantially free of a B. burgdorferi spirochete or fragments thereof and which are thus useful in compositions and methods for the treatment and prevention of B. burgdorferi infection and Lyme disease. In one preferred embodiment, this invention provides modified OspA polypeptides and pharmaceutically effective compositions and methods comprising those polypeptides. Preferred modified OspA polypeptides are characterized by modifications which diminish and/or ablate their ability to bind the human MHC allele HLA-DRB1*0401.”
29) Mayo’s RICO Patent for the post-LYMErix-approval world and their intended monopoly on blood and vector-borne diseases DNA; US Patent 6,045,804:
"Additional uncertainty may arise if the vaccines are not completely protective; vaccinated patients with multisystem complaints characteristic of later presentations of Lyme disease may be difficult to distinguish from patients with vaccine failure….
"The present invention provides a method useful to detect a B. burgdorferi infection in a subject. The method provided by the invention is particularly useful to discriminate B. burgdorferi infection from OspA vaccination, although it is sufficiently sensitive and specific to use in any general Lyme disease screening or diagnostic application. Thus, the method of the invention is particularly appropriate for large scale screening or diagnostic applications where only part of the subject population has been vaccinated or where the vaccination status of the population is unknown. " [a monopoly- KMD).
30) The people involved with that RICO patent are (Fikrig, Persing and Schoen, and the Mayo Clinic): Therefore all of them knew at that time – 1995 – that LYMErix vaccination caused a systemic disease like Chronic Lyme , but never advised the FDA of this when they claimed they had a vaccine in LYMErix, in 1998:
Borrelia burgdorferi enzyme-linked immunosorbent assay for discrimination of OspA vaccination from spirochete infection.
32) Dattwyler at the 1994 FDA meeting stating that the seronegative Lyme victims were the sickest:
DATTWYLER: “So, individuals with a poor immune response tend to have worse disease.”
33) Ben Luft at the 1998 FDA meeting saying LYMErix victims have “protean” disease manifestations just like Chronic Neurologic Lyme victims:
LUFT: "The point that I wanted to make in regard to the study is that there is very heavy dependence on serologic confirmation. And when we start thinking about the adverse events, *** it was stated originally when we got the overview of the disease that the disease is really quite protean [means not limited to "bad knees- KMD]. And actually the adverse events are very similar to what the disease manifestations are.**** And if you start to, as I think Dr. Hall was eluding to -- if you start to kind of say well how often do you actually become sero positive, you can start to have a different take on when someone has an adverse event of whether it is disease specific or infection specific versus vaccine specific. And I think that that is an important issue that we have to deal with…."
34) 1993, Barbour and Fish, trash us because a fake vaccine that caused the same disease they are about to discount at Dearborn.. is on the way
The Biological and Social Phenomenon of Lyme Disease
35) Steere in Europe (not in the Dearborn booklet) falsifying the testing with bogus high passage stains and recombinant OspA and B without the lipids attached, resulting in no immunogenicity and assuring OspA and B would be left out of the diagnostic standard, such that later, with Steere, Mayo, Imugen, and Yale’s L2 Diagnostics labs could have a monopoly on North American testing for vector borne diseases as shown in the RICO patent, Reference 29, above:
Antibody responses to the three genomic groups of Borrelia burgdorferi in European Lyme borreliosis.
36) Steere in Europe, this document ended up in the Dearborn booklet, but it was the previous one, reference 35 that had all the dirt on how Steere falsified the testing:
Western blotting in the serodiagnosis of Lyme disease.
37) DEARBORN, THE NEW, FALSIFIED CASE DEFINITION; Notice there is no band 31 (OspA) or 34 (OspB)
38) Free full text of that event so you can verify independently that this was not even a consensus conference. Everyone said Steere’s proposal sucked when tested in the field:
39) Biology of Borrelia species, Barbour and Hayes, 1986
41) Medvedev and Cross-Tolerance TLR4 – to LPS, or plain old regular bacteria
Induction of in vitro reprogramming by Toll-like receptor (TLR)2 and TLR4 agonists in murine macrophages: effects of TLR "homotolerance" versus "heterotolerance" on NF-kappa B signaling pathway components.
42) Harding and Cross Tolerance TLR7/9 agonists such as viruses
TLR2 signaling depletes IRAK1 and inhibits induction of type I IFN by TLR7/9.
“Because IRAK1 is required for TLR7/9-induced IFN-I production, we propose that TLR2 signaling induces rapid depletion of IRAK1, which impairs IFN-I induction by TLR7/9. This novel mechanism, whereby TLR2 inhibits IFN-I induction by TLR7/9, may shape immune responses to microbes that express ligands for both TLR2 and TLR7/TLR9, or responses to bacteria/virus coinfection.”
43) Klempner 4.7 million dollar fraud-on-the-government
Two controlled trials of antibiotic treatment in patients with persistent symptoms and a history of Lyme disease.
44) Klempner and Wormser rediscover that Lyme is a bad knee, 11 years after the Dearborn stunt, where Lyme was relabeled “only a bad knee”:
A case-control study to examine HLA haplotype associations in patients with posttreatment chronic Lyme disease.
“Patients feel well aside from their arthritis symptoms.” [Duh, we already knew that from the Dearborn-LYMErix stunt- KMD]
45) Schoen selling bad glial cells for a knee disease vaccine
The Lyme Disease Vaccine: Conception, Development, and Implementation
"Other peripheral neuropathies and Lyme meningitis are also seen at this stage. In late-stage disease, the central nervous system may be involved. A new diagnostic test measuring glial fibrillary acidic protein in cerebrospinal fluid may prove to be a useful tool for measuring such involvement (20)."
46) [The effect of Eperythrozoon suis infection on the osmotic fragility of erythrocytes]
“Osmotic fragility of erythrocytes was tested in weaned pigs experimentally infected with Eperythrozoon (E.) suis. Acute eperythrozoonosis of splenectomized pigs led to an increase of osmotic fragility. It is supposed that E. suis infection causes a structural change in erythrocyte membrane. Possible mechanisms of this cell membrane injury are discussed.”
47) Adhesion of mycoplasmas to eukaryotic cells.
“The binding of this fraction to red cells was relatively low but appeared to be specific, as it was inhibited by glycophorin but not by its hydrophobic moiety. The possibility is discussed that the exposure of the binding sites on the mycoplasma cell surface is influenced by the electrochemical ion gradient across the membrane.”
48) Epstein-Barr virus immediate-early proteins BZLF1 and BRLF1 alter mitochondrial morphology during lytic replication.
Mycoplasma fermentans inhibits tumor necrosis factor alpha-induced apoptosis in the human myelomonocytic U937 cell line.
“. To investigate the mechanism of M. fermentans antiapoptotic effect, the reduction of mitochondrial transmembrane potential (DeltaPsim) and the protease activity of caspase-8 were measured. In the infected cells, the reduction of DeltaPsim was inhibited (approximately 75%), and an approximately 60% reduction of caspase-8 activity was measured. In conclusion, M. fermentans significantly inhibits TNFalpha-induced apoptosis in U937 cells, and its effect is upstream of the mitochondria and upstream of caspase-8.”
50) Induction of Epstein-Barr virus (EBV) lytic cycle in vitro causes lipid peroxidation, protein oxidation and DNA damage in lymphoblastoid B cell lines.
51) Brain study shows inflammation is a marker of autism
“Inflammation response genes perpetually switched on in autistic brains
“In order to perform a more in-depth investigation, the researchers analyzed gene expression data from 104 brain samples from 72 individuals - some with autism and some healthy controls - which they say is the largest data set so far used in a gene expression and autism study”
You can see how ^^^ that is identical to what the NIH says about exposure to fungal antigens like LYMErix: [4) 2006: The NIH (NINDS’s MS-Lyme Group) group that discovered that *** OspA *** was the cause of the MS/New Great Imitator outcome of Lyme reporting in the New York Times in the summer of 2013 (Martin and Marques, 2006); this article says these OspA like antigens constantly shed by Borreliae cause immunosuppression in the humoral immune system, but apparently a chronic inflammatory state in the central nervous system:
"Borrelia burgdorferi Induces TLR1 and TLR2 in human microglia and peripheral blood monocytes but differentially regulates HLA-class II expression."
52) Brains of People with Chronic Fatigue Syndrome Offer Clues About Disorder
“The most striking finding was that in people with the disorder, one neural tract in the white matter of the right hemisphere appeared to be abnormally shaped, as if the cablelike nerve structures had crisscrossed or changed in some other way. Furthermore, the most seriously ill patients exhibited the greatest levels of this abnormality.
“The researchers also found in M.E./C.F.S. patients a thickening of the gray matter at the two points of the right hemisphere connected by this particular neural tract. And the overall volume of white matter in the brains of patients was reduced, compared with the brains of people without the disorder.”