Blowing the Whistle at the FDA, Jan 2001, exposing Dearborn and how OspA causes immunosuppression rather than, "was a vaccine."

01 Oct 2017


File List, RICO

1988 Steere says Lyme is like a B cell leukemia

Assoc Blogs-n-Webs:




Fungal Exosomes Inhibit Apoptosis

IDSA: "Vaccines serve the mfgs, not their victims"


BlumenthalAntiTrust Lawsuit

Exosomes, Blebs


CDC Admits Fraud, 2016
Dattwyler, 1988
Golightly, 1988
Dressler, 1994
BarbourFish, 1993
Dearborn, 1994

Pathogenic Fungi

Bush's warcrimes, Oct 2000





Confronting "government" "employees" over their scientific incompetence


The following is a letter sent to the CDC, NIH, Fauci regarding Paul Auwaerter, the current blowhard/mouthpiece of IDSA:
Dr. Paul Auwaerter Photo


To: KMDickson <>, "Strikas,Raymond A. (Ray) (CDC/OID/NCIRD)" <>,,,,

Subject: Paul Auwaerter Does Not Know What OspA Is Either

Date: Apr 20, 2012 4:35 AM


I called Auwaerter's office (Johns Hopkins)
and left a message asking if he knows what
OspA is. The answer was no, he did not.

I guess he is not an expert in Lyme Disease

Now, all of this is amusing given the fact
that Roland Martin quit the NIH's MS-Lyme
group once he found out OspA was causing
the immune suppression (and seronegative,
non-Dearborn Lyme) resulting in the MS
form of Lyme (known to be associated with
reactivation of Epstein-Barr):
"Borrelia burgdorferi Induces TLR1 and TLR2 in human microglia and peripheral blood monocytes but differentially regulates HLA-class II expression."

as does Adrianna Marques, who publishes these
articles with him:

"Borrelia burgdorferi lipoprotein-mediated TLR2 stimulation causes the down-regulation of TLR5 in human monocytes."

That^^ all means Chronic Lyme is seronegative
and the chronic exposure to OspA/Borreliosis
results in NO ANTIBODIES. We now know for
sure that Klempner's "study" can be thrown out
since the study design was based on the
fraudulent Dearborn diagnostic standard.

[Dearborn was fraudulent because there was
no consensus (I have the booklet) and because
Steere formulated the antibody panel via
classic research fraud, using high passage
strains and recombinant OspA and B without
the antibody-producing lipids attached, setting
up his own little racketeering enterprise with
Dave Persing (Corixa) and Yale's Robert
Schoen (L2 Diagnostics) for the intended
post-LYMErix monopoly on blood, patentable
goodies in the blood, and grants.]

Secondarily, Paul Auwaerter lists as his
expertise, Lyme and Epstein-Barr (known to
be activated by Lyme and probably LYMErix
as shown by Marques and Martin, above).

Thirdly, Bridgette Huber at Tufts, who
published with Allen Steere ("Lyme causes
only the HLA-linked hypersensitivity response")
but her area of expertise also includes
Lyme and Epstein-Barr (EBV activating a
human HERV, the favorite of Harold Varmus
former director of the NIH:

Fourthly, Adrianna Marques lists on *HER*
website, that her areas of expertise are
Lyme and Epstein-Barr (and some other herpes

So, it looks to me that "Post Lyme Syndrome" is,
in Marques' words, "Chronic Active Epstein-Barr."

Especially since Paul Duray (US Army and
the National Cancer Institute)
twice in IDSA's journals that the lymphocytes of
chronic Lyme victims "look like Epstein-Barr
transformed cells
" in 1989 and 1992.

How this happens, how these fungal antigens
seem to reactive Epstein-Barr, I do now know
exactly, other than the association to
exposure to OspA and subsequent production
of the immunosuppressive cytokine, IL-10.
(I won't produce the references for that
because they're well known and accessible.)

Now, the problem with Adrianna Marques is that
she is recruiting chronic Lyme victims to see
if she can get clean ticks to become infected
with Borrelia from biting post-treated Lyme
victims - from the blood.

But that's not really what's going on, is it?

It's going to be mycoplasma to which we have
become tolerant via exposure to OspA (blebbing
is the same as autovaccination), and some kind
of herpes, et al, correct?


Ticks are a great place for transkingdom
plasmid-sharing according to Barbour and

You're thinking of *that* ^ when Marques tries to
pharm Borrelia not known to be in the peripheral
blood of human victims due to the tissue penetrating,
plasminogen-hijacking OspA??


Kathleen M. Dickson



Subject: No one at the NIH, CDC, Yale, or IDSA knows what OspA is.

Date: May 1, 2012 4:54 AM


I see that there is a job opening for what looks like
Sally Rockey's job:

I am wondering if Ms. Rockey is leaving becuase no one
at the NIH knows what OspA is?

As most of you know, I have been asking around for over a month.

WHAT IS OSP-A (the structure)?

No one at Yale knows.

IDSociety doesn't know.

NIAID doesn't know.

Francis Collins can't/won't answer the question.

Paul Auwaerter's office tells me he does not know.

Lawrence Tabak hasn't answered me.

No one at the CDC knows (despite CDC officers
owning several patents for recombinant antigens
of Borrelia burgdorferi).

Nobody knows what OspA is.

I find this odd, since this vaccine designed the "disease." 
Allen Steere left OspA and B out of the diagnostic
for Lyme disease using illegal (according to the
NIH Rocky Mountains Lab) "high-passage" strains of Bb
and recombinant antigens of the protein ends of OspA and B
without the lipid attached to leave OspA and B out of the current
CDC diagnostic standard, only to find the Western Blots of OspA-vaccinated
people were unreadable anyway:
(presumably ^^ because OspA sticks to itself as CDC officer
Alan Barbour once claimed).

The first order of business is the clarification of falsified antibody testing for “Lyme Disease.”
In 1992 Allen Steere went to Germany, with, as he claimed,

“The group 1 strain of B. burgdorferi, G39/40, used in this study and in the previous study of US patients was isolated from an Ixodes damini tick in Guilford, Connecticut [21].  The group 2 strain, FRG [Federal Republic of Germany], was isolated from Ixodes ricinus near  Cologne [22].  The group 3 strain, IP3, was isolated from Ixodes persulcatus near Leningrad [23].  All three strains used in this study were high passage isolates, which were classified by Richard Marconi (Rocky Mountain Laboratory, Hamilton, MT) using 16S ribosomal RNA [per the DNA/RNA Shell Game - never used to detect spirochetes in humans after treatment]  sequence determination as described [11, 24].  The recombinant preparations of OspA and OspB used in this study were purified maltose- binding protein-Osp fusion proteins derived from group 1 strain B31 [25].  The fusion proteins contained the full-length OspA or OspB sequence without the lipid moiety or the signal sequence -"

-- illegal, plasmid-dropping, antigen-and-antibody dropping, “high passage strains,” and OspA and B with no lipids attached (not likely to produce antibodies), resulting in the new, 1994, Centers for Disease Control (CDC), “case definition” of “Lyme Disease (8, 9)."  

Without the lipids attached, the outer surface proteins are
less likely to produce antibodies.

Now, at one time (1991), Allen Steere wrote that
there were 2 outcomes to Lyme: RA and MS.

Currently, only the RA outcome is recognized
(see the CDC's Dearborn definition where only
the HLA-linked hypersensitivity response without
OspA-B is recognized) as a "case."

The basis of the 4.7 million dollar grant that
went to Mark Klempner was to study the 30 day
intravenous ceftriaxone treatment of people
among whom 2/3rds never had had intravenous
ceftriaxone before:
"A total of 42 (33 percent) of the patients had previously received intravenous antibiotic treatment for a mean (±SD) of 30±12 days. All other previous treatment consisted of oral antibiotics." 

Klempner ^^^ found that 30 days of IV ceftriaxone was
not enough ceftriaxone for people who had never
had ceftriaxone before.

Perhaps no one was looking too closely at
the grant they approved.  Klempner found
that the "standard of care" - arbitrarily defined
in the first place - was not enough intravenous
antibiotics for these people.

But that's a tough call, isn't it?  You don't
need INTRAVENOUS antibiotics for knee diseases -
the "case definition."

Maybe if we knew what OspA was.  After all, it
defined the disease.  Both in the classical
commercial sense (the Dearborn definition) and in
Allen Steere's HLA-linked hypersensitivity sense.

A man named Roland Martin left his job at
the NIH once he found out OspA actually caused
the disease he was studying - the MS outcome:
"Borrelia burgdorferi Induces TLR1 and TLR2 in human microglia and peripheral blood monocytes but differentially regulates HLA-class II expression."
"These results show that signaling through ***TLR1/2*** [note- remember the structure/function relationship-added by KMD] in response to B. burgdorferi can elicit opposite immunoregulatory effects in blood and in brain immune cells, which could play a role in the different susceptibility of these compartments to infection."

"Borrelia burgdorferi lipoprotein-mediated TLR2 stimulation causes the down-regulation of TLR5 in human monocytes."

"Toll-like receptors (TLRs) trigger innate immune responses via the recognition of conserved pathogen-associated molecular patterns. Lipoproteins from Borrelia burgdorferi, the agent of Lyme disease, activate inflammatory cells through TLR2 and TLR1. We show that stimulation of human monocytes with B. burgdorferi lysate, ***lipidated outer surface protein A, and triacylated lipopeptide Pam3CysSerLys4 results in the up-regulation of both TLR2 and TLR1 but the down-regulation of TLR5, the receptor for bacterial flagellin, and that this effect is mediated via TLR2.*** TLR4 stimulation had no effect on TLR2, TLR1, and TLR5 expression. Human monocytes stimulated with TLR5 ligands (including p37 or flaA, the minor protein from B. burgdorferi flagella) up-regulated TLR5. ***In addition, TLR2 stimulation rendered cells hyporesponsive to a TLR5 agonist.*** These results indicate that diverse stimuli can cause differential TLR expression, and we hypothesize that these changes may be useful for either the pathogen and/or the host."

I hypothesize that TLR2/1 agonists suppress
the immune response and that everything Mark
Klempner and IDSA, Yale, and the CDC say is

What do you say?


Anyone know what OspA is?

The CDC to this day maintains that it is
a vaccine.  NIH never retracted their statement
that it was.

My personal observation is that, like Roland
Martin is revealing, OspA caused the seronegative
neurological disease outcome sometimes called
Multiple Sclerosis - the job he was hired to study
at the NIH.

OspA does something to the immune system that
is not like what Allen Steere and the CDC claim
with their "case definition."

And MS is largely recognized to be caused
by Epstein-Barr/Similar herpes viruses.


It would be nice to know what OspA is.

We might be able to end the OspA-linked

Kathleen M. Dickson
former Pfizer Analytical
Methods Development and Validation


Subject: A 5-points rule on fungal vaccines and brain-damaged children (failed TB and Lyme fungal vaccines notwithstanding)
Date: May 1, 2012 6:00 AM


I would like to propose a rule on fungal antigens and vaccines
which is related to the OspA outcomes:

1) "Finally, there is the risk that the virus may not be fully or completely inactivated or attenuated and thus, the vaccine may actually cause disease."

2) Measles, Mumps, and Rubella -- Vaccine Use and Strategies for Elimination of Measles, Rubella, and Congenital Rubella Syndrome and Control of Mumps: Recommendations of the Advisory Committee on Immunization Practices (ACIP)

"Updated information on adverse events and contraindications, particularly for persons with severe HIV infection, persons with a history of egg allergy or gelatin allergy, persons with a history of thrombocytopenia, and persons receiving steroid therapy [are immunosuppressed- KMD]."


3) CDC: Human Exposure to Brucella abortus Strain RB51 -- Kansas, 1997

An ^^^ immunosuppressed pregnant cow was given a Brucella (LYMErix-like) "live attenuated" vaccine and the baby cow ended up with the disease, which then was transferred to the humans handling the cow and her dead baby.  This parallels what is happening to children who are vaccinated while immunosuppressed, or who receive mycoplasmally (LYMErix-like) contaminated vaccines.


4) in the case of pandemic MRSA, as we have seen, the vaccine didn't work because TLR2 agonists (lipoproteins) suppress the immune system.  We also learned from the MRSA vaccine patent, that:

"Several established vaccines consist of live attenuated organisms where ***the risk of reversion to the virulent wild-type strain exists. In particular in immunocompromised hosts this can be a live threatening scenario.*** Alternatively, vaccines are administered as a combination of pathogen-derived antigens together with compounds that induce or enhance immune responses against these antigens (these compounds are commonly termed adjuvant), since these subunit vaccines on their own are generally not effective.",771,728.PN.&OS=PN/7,771,728&RS=PN/7,771,728


5) The effect of exogenous corticosterone on West Nile virus infection in Northern Cardinals (Cardinalis cardinalis)

Do not ^^ cause immunosuppression/stress as
it causes increased mortality from existing
infections and especially do not cause stress to
humans by treating them to *ABUSE* instead
of medicine when they don't the special "controversial"
disease that fits the commercial (unknown) vaccine model:

Because ^^ that - the abuse - activates Epstein-Barr.


RULE:  Don't give immunosuppressed mammals live attenuated vaccines, and especially don't give them vaccines that contain fungal or mycoplasmal antigens like LYMErix or OspA.

What do you say?   Do you like that rule?
Especially if we added the OspA/MS/Roland Martin
"Immunosuppression>> MS Outcome of Lyme" data
to that data set?

What about if we added the 1918 Spanish
Flu data to the rule:  "People with strong
(Steere-HLA-like, Dearborn definition-like)
immune responses are likelier to choke to death
on the pneumonia (fungal) that follows the influenza,
whereas immunosuppressed people won't produce a
hyperinflammatory response and thereby survive the

We haven't had any new Rules in a while.

All we ever hear is that "Lyme causes nothing"
and "no disease outcomes happen from Lyme"
and "nothing causes anything" and "Lyme victims
are looney" and "Lyme victims have paranoid
delusional wannabee diseases" and the latest:
"Lyme victims are terrorists !!!":

"But get the mysterious unknown OspA vaccine."

- - - - - - -


What do you think?

How about that for a rule?

You might have to find out what OspA is, first.

No hurry.  It was ordered removed by the FDA
in Feb, 2002 for producing "chronic Lyme-like"
illness (clue).

And that was the last time Uncle Sam said
anything *TRUE* about "Lyme Disease."

10 years....

Kathleen M. Dickson
former Pfizer Analytical Methods Development
and VALIDATION chemist


To:  (Harvard Business School Mormon featured in WaPo)
Cc:,, "Allen,Marin (NIH/OD) [E]" <>,,, "Strikas,Raymond A. (Ray) (CDC/OID/NCIRD)" <>,,,,,,,,,,,,,

Subject: Fearless WaPo Leader Anthony Fauci's Patent for Immunosuppression Outcomes like LYMErix Disease


Date: May 17, 2012 6:16 AM

Dear Sir (Harvard Business School's
Clayton Christensen),

Since your Harvard and Mormon expertise is
in "asking the right questions," I ask you
again to please ask Anthony Fauci if he
could find out for the nation what OspA,
the Lyme vaccines (ImmuLyme and LYMErix)

Remember that Fauci said publically on CNN
on the Lou Dobbs show that OspA was "scientifically
a vaccine":

"We've had an effective vaccine, but it's the kind of vaccine that you have to essentially vaccinate people each year. And from the standpoint of it's use, it has not been used as efficiently as it could have been used. So scientifically, we had a vaccine and still do have a vaccine, but it's not really well used." -- Anthony Fauci

But no one at the NIH, CDC,, or
Yale has been able to answer my simple question
as to what exactly OspA is/was/does.

Now, a few years ago, Anthony Fauci was featured
in WaPo and the US News and World Report (now owned
by Mort Zuckerman who was a supporter of the criminal
Lyme cryme gang, the - the non-non-profit
who designed the disease, "Lyme" around the outcomes of
OspA as a vaccine), credited as a "Leader" like yourself:

Notice how ^^ Fauci laughs nervously while he explains
that he has no clue what his scientists are doing,
making it not too believable that he would be
recognized as a leader, but then again, this is
WaPo and the US News.

Specifically, I would like you to note that Anthony
Fauci owns a patent for a treatment of the immune
suppression outcomes of diseases like LYMErix Disease
(the disease caused by LYMErix or exposure to OspA):

- - - - -
"Illustrative of specific disease states in treatment of which the present invention can be applied are HIV infection and other diseases characterized by a decrease of T-cell immunity, for example, mycobacterial infections like tuberculosis and fungal infections such as cryptococcal disease." See More
United States Patent: 5696079
"A method for activating a mammalian immune system entails a series of IL-2 administrations that are effected intermittently over an extended period. Each administration of IL-2 is sufficient to allow spontaneous DNA synthesis in peripheral blood or lymph node cells of the patient to ..."

- - - - - -

Meanwhile, there is a new campaign on by the NIH to
hook up with BigPharma to resuscitate dead near-drugs, but
still no one mentions publically what either Lyme Disease or
LYMErix Disease actually is, ***so where would we even start to
treat these outcomes?***

I have been asking the NIH, CDC, NIAID, Fauci's office,, and Yale over and over again for the
past 2 months, if anyone could tell us what this
antigen is, OspA. 

No answer. 

Only, now, a cowardly threat by Francis Collins, the
fake Catholic.

*WE* know what OspA is, because its structure
was published in a non-PubMed-linked Korean
Chemistry journal (Mass-Spec) and more recently
by the Japanese (NMR).

But the NIH cannot tell us what it is.  They cannot
publically state what OspA is.  Either they don't
know or they are afraid to admit the truth.

We believe the latter to be a big possibility - they're
all cowards - because Francis Collins, the fake-Catholic
threatened me with "calling the police" if I called
his office again to report that none of the people who
work for him can tell me what OspA is.

That kind of behavior is just plain *trashy.*  The NIH
and CDC said OspA was a vaccine.  But they're going
to throw me in jail for asking what it is?

This is "Leadership?"

So, we would like you, Mr Harvard Business School
and Mormon who "asks the right questions" Mr. Christensen 
to pick up the phone and call Francis Collins and Anthony
Fauci and ask on our behalf for an answer to the OspA

Thank you.

Here is Francis Collins' phone number: 301-496-2433 

Kathleen M. Dickson
former Pfizer Analytical Methods
Development and Validation chemist



Once the NIH/CDC publically state what OspA is, that is the end of all "Lyme Disease" guidelines and diagnostic schemas.

The Dearborn "case definition" was used to qualify the outcomes of the OspA vaccines. 

Once we find out LYMErix or OspA could never have been a vaccine - because the NIH/CDC admit what it is - we will learn that the case definition used to qualify them must be false.

And if Dearborn is false, so too, is Klempner and the "guidelines."

Everything gets thrown out.  We treat based on the data and evidence available.


Is this "Leadership" crap one big circle jerk or what?  They chronically reward and award each other for doing nothing and not being able to think.  Their personalities are like in the new Sasha Baron Cohen movie, The Dictator.

Francis Collins:  "Ask again what OspA is, and I keel you."