120420; "Lyme Disease" Update from the CDC, NIH, NIAID, Yale and IDSociety.org, none of whom, in a month's time, has been able to tell me what OspA (the Lyme vaccines) is - they all say they do not know:
 

The following is a letter sent to them regarding Paul Auwaerter, the current blowhard/mouthpiece of IDSA:
 

(UPDATE ON PAUL AUWAERTER - HE DOES NOT KNOW What OspA IS):

To: KMDickson <janmusinski@earthlink.net>, "Strikas,Raymond A. (Ray) (CDC/OID/NCIRD)" <ras8@cdc.gov>, olib@od.nih.gov, afauci@niaid.nih.gov, joseph.breen@nih.gov, michael.greenwood@yale.edu

Subject: Paul Auwaerter Does Not Know What OspA Is Either

Date: Apr 20, 2012 4:35 AM

Greetings,

I called Auwaerter's office (Johns Hopkins)
and left a message asking if he knows what
OspA is. The answer was no, he did not.

I guess he is not an expert in Lyme Disease
either.

Now, all of this is amusing given the fact
that Roland Martin quit the NIH's MS-Lyme
group once he found out OspA was causing
the immune suppression (and seronegative,
non-Dearborn Lyme) resulting in the MS
form of Lyme (known to be associated with
reactivation of Epstein-Barr):
"Borrelia burgdorferi Induces TLR1 and TLR2 in human microglia and peripheral blood monocytes but differentially regulates HLA-class II expression."
http://www.ncbi.nlm.nih.gov/pubmed/16783164

as does Adrianna Marques, who publishes these
articles with him:

"Borrelia burgdorferi lipoprotein-mediated TLR2 stimulation causes the down-regulation of TLR5 in human monocytes."
http://www.ncbi.nlm.nih.gov/pubmed/16479520

That^^ all means Chronic Lyme is seronegative
and the chronic exposure to OspA/Borreliosis
results in NO ANTIBODIES. We now know for
sure that Klempner's "study" can be thrown out
since the study design was based on the
fraudulent Dearborn diagnostic standard.

[Dearborn was fraudulent because there was
no consensus (I have the booklet) and because
Steere formulated the antibody panel via
classic research fraud, using high passage
strains and recombinant OspA and B without
the antibody-producing lipids attached, setting
up his own little racketeering enterprise with
Dave Persing (Corixa) and Yale's Robert
Schoen (L2 Diagnostics) for the intended
post-LYMErix monopoly on blood, patentable
goodies in the blood, and grants.]

Secondarily, Paul Auwaerter lists as his
expertise, Lyme and Epstein-Barr (known to
be activated by Lyme and probably LYMErix
as shown by Marques and Martin, above).
http://www.hopkinsmedicine.org/awomansjourney/baltimore/2009_speaker_profiles/2009%20_paul_auwaerter_profile.html

Thirdly, Bridgette Huber at Tufts, who
published with Allen Steere ("Lyme causes
only the HLA-linked hypersensitivity response")
but her area of expertise also includes
Lyme and Epstein-Barr (EBV activating a
human HERV, the favorite of Harold Varmus
former director of the NIH:
http://www.ncbi.nlm.nih.gov/pubmed?term=huber+bt

Fourthly, Adrianna Marques lists on *HER*
website, that her areas of expertise are
Lyme and Epstein-Barr (and some other herpes
viruses).
http://www.niaid.nih.gov/labsandresources/labs/aboutlabs/lcid/clinicalstudiesunit/Pages/marques.aspx

So, it looks to me that "Post Lyme Syndrome" is,
in Marques' words, "Chronic Active Epstein-Barr."

Especially since Paul Duray (US Army and
the National Cancer Institute) reported
twice in IDSA's journals that the lymphocytes of
chronic Lyme victims "look like Epstein-Barr
transformed cells" in 1989 and 1992.

How this happens, how these fungal antigens
seem to reactive Epstein-Barr, I do now know
exactly, other than the association to
exposure to OspA and subsequent production
of the immunosuppressive cytokine, IL-10.
(I won't produce the references for that
because they're well known and accessible.)

Now, the problem with Adrianna Marques is that
she is recruiting chronic Lyme victims to see
if she can get clean ticks to become infected
with Borrelia from biting post-treated Lyme
victims - from the blood.

But that's not really what's going on, is it?

It's going to be mycoplasma to which we have
become tolerant via exposure to OspA (blebbing
is the same as autovaccination), and some kind
of herpes, et al, correct?

Anyone?

Ticks are a great place for transkingdom
plasmid-sharing according to Barbour and
Burgdorfer.

You're thinking of *that* ^ when Marques tries to
pharm Borrelia not known to be in the peripheral
blood of human victims due to the tissue penetrating,
plasminogen-hijacking OspA??

Anyone?

Kathleen M. Dickson