Cryme Disease video playlist
Bad Ass Cryme Blog
141219_post-sepsis Rituximab (again)
Pandora: ME/CFS related to
Follow the Lyme/LYMErix
Scam in the order it occurred, below:
Older data on the incurability of Relapsing Fever
1986, McSweegan trashes Navy for $$$ for
1988, Dattwyler & about immune-suppressing, seronegative Lyme
1990, CDC: "Diagnose Lyme as if it was Relapsing
Allen Steere "NeuroLyme won't
test positive," 1990.
CDC officer Allen Steere falsifies testing in
1992, CDC patents with
SmithKline show 2 kinds of Lyme
Barbour and Fish slam Neurologic Lyme victims.
Compare the 2 kinds of Lyme in the RICO
1994, FDA LYMErix Meeting
CDC's Dearborn Booklet .pdf
CDC's invitation to participate in Dearborn
Dearborn, Who Said What?
Igenex, Harris, Dearborn .pdf
Evidence Lyme criminals knew
LYMErix produced the same "multisystem disease" as "Chronic Lyme"
LYMErix Damage Coverup (short)
1998, CIA Oilmen & Israelis plan to overthrow
Saddam for the oil.
ActionLyme/Kathleen Dickson predicts all of Bushie's outcomes in Oct 2000
predicts Bush will have us worshipping his bombs (Shock and Awe"), in Oct 2000
during the Gore Debates
With 20-30 million people
disabled from Fibro, GWI, Lyme, and CFIDS, not to mention the millions of
children brain damaged for life (does anyone know the statistics on cost of
lifetime of care for Autistic children?) and a workforce that is no more
than 50% fully employed (23% of all USA employed are Govt employees), one
wonders what the USDOJ thinks they're doing?
Don't they have anyone there at the USDOJ
concerned about the 25% or more disabled workforce?
Do they have anyone working at the USDOJ who knows anything about math?
Numbers and stuff?
Finance or a nations' reliance on economic - trading - activity?
There is something fundamentally wrong with
Americans' brains. And I am not even referring to those who are disabled
with chronic sepsis-like brain illnesses.
These people are straight up NAZIs. This
is the Gestapo that emerged in the 35 years since that moron Reagan, who
knew nothing about GNP or work or productivity or physics,... and the
Bushies were even dumber.
Chris Hedges nails it:
"The innate barbarity that war and violence breed is justified by a
saccharine sentimentality about the nation, the flag and a perverted
Christianity that blesses its armed crusaders. This sentimentality, as
Baldwin wrote, masks a terrifying numbness. It fosters an unchecked
Facts and historical truths, when they do not fit into the mythic vision
of the nation and the tribe, are discarded. Dissent becomes treason. All
opponents are godless and subhuman. “American Sniper” caters to
a deep sickness rippling through our society. It holds up the dangerous
belief that we can recover our equilibrium and our lost glory by
This is what the international patient and scientist
community needs to know about the
Turncoats, and the criminal and incompetent CDC. You can't believe the CDC
at all, and here are several good reasons why:
"There is no such thing as seronegative Lyme or Lyme Disease cases that
do not meet the Dearborn case definition" -- CDC officers Allen
Steere and Mark Klempner.
Borrelia do not have LPS and are only
bearers of lipoproteins which are fungal and TLR2/1-agonists or triacyl
lipopeptides which they deliberately shed (bleb off) as "EXOTOXINS." Why do
they insist Borrelia are not shedders of fungal, immunosuppressive exotoxins
Who is Steve Schutzer
and why/how is he a liar?
Schutzer and Pat
Coyle used to publish about antigen-antigen
complexes in the spinal fluid of Lyme victims to prove they were
seronegative Lyme. You can verify in PubMed.
Pat Coyle only specializes in MS now,
and goes borneo on ya if you mention Lyme at her MS talks. She acts
like she was threatened.
Schutzer was the editor of the Lyme
Disease, Molecular and Immunologic Approaches Cold Spring Harbor conference
in 1992 where in they talk about NeuroLyme seriously, and Paul Duray again
mentioned EBV possibly being activated in CNS Lyme:
Steve Schutzer was put on a round
table/panel in one of the LDF Lyme conferences in 2000 or 2001 and it was
asked if one or two bands would be good enough to detect Lyme. Donta said
like Band 23 and 41 should be fine. Igenex's Nick Harris agreed. I ferget
who the 4th person was. Schutzer said "NO, BECAUSE CFIDS PEOPLE OFTEN
HAVE A FEW LYME BANDS."
Uncle Sam Dot Mil talks about anti-personnel biological weapon Time Bombs or
Trojan Horse bioweapons:
"Methods of using antipersonnel agents undoubtedly wary so that no uniform
pattern of employment or operation is evident [make sure it does not produce
antibodies, is the short version- KMD]. It is likely that agents will be
used in combinations so that disease symptoms will confuse diagnosis and
interfere with proper treatment. It is also probable that biological agents
would be used in heavy concentrations to insure a high percentage of
infection [or just use the OspA vaccine- KMD] in the target area. The use of
such concentrations [or the multiple infections it causes, due to the
immunosuppression like HIV, Lyme, or LYMErix as acquried immune deficiencies
- KMD] could result in the breakdown of individual immunity because the
large number of micro-organisms entering the body could overwhelm the
natural body defenses [or just infect or inject people with an immune
suppressor like OspA from a tick or a syringe,
and the reverse will happen: people will acquire multiple infections because
their immunity is trashed by OspA- KMD].
And the genetics of anserina-come-burgdorferi:
How could all
of them be such
F*ck-Ups, Turncoats and LIARS?
They all jumped-ship and turned coats right around the time I blew
on the whole thing, from Dearborn being a fraud to OspA causing
Think about it. You're gonna believe
these CDC assholes about
Let's make sure we all know what we are talking about since the CDC and
IDSA do not. Even Yale says they do not know what
OspA is - yet they owned that patent for LYMErix. And you can't make
this shit up, because this is America.
We have to OCCUPY the USDOJ in Washington to force the "government"
"lawyers" tell the CDC and IDSA spirochetes are not
bacteria and that OspA was a fungal antigen and never could have been a
And that is because this is America where only stupid criminal shit
150123 Sweeg in Russia, ALDF
Bioweapons Data-Pharming, Tom Dispatch on Weapons $$$
Dickson on Friday,
January 23, 2015 at 4:00am
http://travelmag.co.uk/2003/05/sent-to-siberia/ << Sweeg
really was in Russia, there he is talking about it, LOL.
THE ARTICLE by TOM
DISPATCH on the WALKING DEAD in Washington (below) ALSO APPLIES
to the 30 million of us disabled from Lyme, GWI, ME/CFS and brain
damaged from vaccines (Autism),.. actually that could be a higher number
if 12 million is the official number of CFS (4 mil) and Fibro (8 mil)
alone. In addition to this "safety" and "intelligence" concern, the cat
is totally out of the bag on bioweapons and bad vaccines,
*** Recall that in May
2007 the Russian Duma banned export of human DNA to America, when Sweeg
himself was allegedly over there in Russia just a few years earlier
allegedly heppin-em destroy their biological weapons.*** (That was the
era when his buddy Durland Fish said Sweeg was "hung like a horse," - re
Sweeg in Akademgorodok, Russia, since
Durland is a pig and grotesque.)
I can't say for sure if it
is related but I had begun faxing the foreign embassies in the summer of
2006 and explained what they (Lyme Crooks) were doing and told them not
to let them sample any DNA because they were bioweapons data-pharming (HLAs).
In Sep, 2006, I had 14,000 visitors to ActionLyme that month, alone,
That is, ASSOCIATES of the ALDF.com at
New York Medical College were sampling HLA data from around the world
(that evidence is on ActionLyme), We know that a successful bioweapon
would be undetectable so it would be the ABSENCE of "disease" associated
HLAs that would make an effective bioweapon, because no antibodies would
be produced. Recall that in PNAC, the neocons were talking about using
race-specific bioweapons. A race specific bioweapon would be a stealth
disabler for that group or would produce few HLA-linked hypersensitivity
cases that identified the original infection.
Below this link I will
link the evidence for what I am talking about above:
"Russian ban on export of
In May 2007, Russian
newspaper Kommersant reported that the Russian government banned all
exports of human biosamples. The report claims that the reason for
the ban was a secret FSB report about on-going development of "genetic
bioweapons" targeting Russian population by Western institutions. The
report mentions the Harvard School of Public Health, American
International Health Alliance, United States Department of Justice
Environment and Natural Resources Division, Karolinska Institutet and
United States Agency for International Development."
HERE is DURLAND FISH using
a fake Lyme victim name to say Sweeg is "Hung Like a Horse" in the
conversation about Sweeg in Russia, Akademgorodok:
The PNAC document where the Israeli Neocons discuss using "race-specific
These are the Russian-name
-sounding associates of the Lyme crooks HLA-data-pharming all over the
world (the publish from and with people at NYMC or the Lyme crooks'
Some of the other Russian
Scientists either publishing in Durland Fish's 'Journal", or New York
Medical College- Look closely at all of their publications. They're
very interested in genetic backgrounds of peoples of foreign nations:
That all ^^^ means the
bioweapons to be developed against these people have to be stealth
against their HLAs. That is, the disease cannot be an HLA-linked
disease or else there will be antibodies against the stealth infection,
which would render the disease detectable. Read more about it here.
HERE IS ONE OF THE FAXES I SENT ALL OVER in the spring and
summer of 2006, re Pam3Cys and an 11 million dollar BIODEFENSE CONTRACT
given to the crooks and wherein, it is clear they know a TLR2/1 agonist
could never be a vaccine since Corixa, Dave Persing and Robert Schoen
knew "OspA was too toxic in the native form."
^^^^ Don't lose that data
that is scanned in in that report, above, it's worth billions to us ;)
I'd like to thank all the Govt's biodefense labs for
being such huge and persistent fans of ActionLyme. It came
up in conversation recently on the Occupy the USDOJ Facebook group (June
2015), what would be their interest here? Welp, they'd be somewhat
more accountable to new diagnostic/analytical machinery and techniques for
identifying infectious diseases, obviously. They can't allow
themselves to get away with being your average typical brainless
streetwalker bigpharma ho "MD," who takes no responsibility upon
him-/herself to find out what eff is going on with 40 years of
"controversy," Blumenthal lawsuits, FDA-retracted "vaccines," ... that
fairy-ass Allen Steere saying he was
"stalked," when no, I spoke with David Grann in person at the Albany rally
and told him everything I told -
and showed, with 19 pages out of the Dearborn booklet, provided
to all 15 FDA Vaccine Committee members- the FDA 4 months earlier when I
blew the whistle on Streere's Dearborn testing scam... So, how could
we be "Stalking Steere" in Boston, when we've been trying to have him BUSTED
for over 15 years...
It might be in the Govt bioweapons goons' interest to know what actual
science and methods are already out there - long now - on how to perform
valid and sensitive analyses on these stealth disablers.
So, the people need to know what the US Bioweapons Govt agencies are
interested in. ("Doctors," shit, you're better off talking to a
pharmacist. They at least are required to have a science background.)
Virological diagnosis of central nervous system infections by use of PCR
coupled with mass
spectrometry analysis of
cerebrospinal fluid samples
No one can stop this. Yale and the CDC are going to be busted
or the USA will not go forward, much less remain technologically viable in
the med sci field. That's it. Play or Crash, Uncle Sam spook
piggies. Prosecute or eat dust and put the USA and our allies in
danger from a lack of sophistication in biotech and bioweapons agents
And Now it is Time to Consider the Format of
It's amazing that we have had to put up with this bullshit
for so many years just because some Yale related scam artists
wanted royalties from their fake fungal "vaccine," LYMErix, and
to monopolize their future TBDs enterprise in vaccines and test
kits. You can read more of their same old hysterics in the
journals, "new" for 2015. It's the same old bullshit and
nonsense because they can't get any grants - the NIH cut them
off. Besides, how many times can you ask for a grant to
study a disease that does not exist.
It's quite obvious they're terrified of being prosecuted.
They can't let it go. If they weren't guilty and
terrified, they would cease and desist and move on. But it
should be obvious to any casual observer that these were the
same fellas who said we need a vaccine against Lyme since it was
such a terrible disease... and now they spend all their time and
energy saying it's not a disease. Suttins up.
It's about the testing fraud, which is a murder charge,
especially over the ALS link.
Like any other RICO fraudster prosecution, they will all lose
all their personal property as well as spend the rest of their
lives in jail, and Yale will lose all of Yale, which is
worth at least $20 billion. That's just the value of
their Endowment Fund.
Yale patented an FDA-validated test for Lyme in 1991-1993, but
they did not use this FDA-validated test to assess their other
patent, LYMErix, because they knew LYMErix was not a vaccine and
caused systemic disease. You can't inject people with
fungi. That's the reason for the Thimerosal in the first
New Yorker Needs to Find a Real Scientist to do their Science
Writing (Michael Spector, gimmeabreakii)
Dickson on Saturday,
January 17, 2015 at 2:31am
firstname.lastname@example.org, email@example.com, firstname.lastname@example.org,
AllenM1@mail.nih.gov, email@example.com, firstname.lastname@example.org,
email@example.com, firstname.lastname@example.org, email@example.com,
firstname.lastname@example.org, email@example.com, firstname.lastname@example.org,
email@example.com, Lawrence.Tabak@nih.gov, firstname.lastname@example.org,
Yorker needs to get an actual scientist to write about
science ("Lyme Disease")
16, 2015 6:28 AM
this commentary, but it's very obvious the writer (Michael
Spector) has no clue what the controversy is about.
"Controversy" is a weasel word used by the likes of
BigTobacco to offset the pursuit into the heart of the
criminal matter. In the case of the Lyme disease
crimes, it's simple research fraud mainly having to do with
an intended vaccines "enterprise" by the fake non-profit,
a complex issue, but not really. Not to someone with a basic
biology science background, and that is my point, here, as
regards your magazine. Where do we start? Probably with the "New
Great Imitator, Lyme Disease," according to Allen Steere and
IDSA, that causes RA and MS and Lupus and ALS (all
proven/revealed by the same people who now deny Lyme spirochetes
cause any kind of neurologic disease). So why is what was once a
Great Imitator (more correctly, “Detonator”), now only strictly
an autoimmune arthritis in a knee that needs no treatment?
an even better question, now that psychiatry has been officially
debunked and no one can even get a grant if they use
scientifically invalid terms like "somatoform," what new kind of
derogatory language will be used to demonize and discredit
people with neurologic New Great Imitator outcomes?Why are
neurologic outcomes from a neurologic disease no longer
not too much about lame-brained ILADS.org and
their “long term antibiotics.” They’re discredited
scientifically not only for this stance, but because they refuse
to even delve into the matter of the research fraud committed by
primarily CDC employees (Allen Steere, Barbara Johnson, Mark
Klempner, Alan Barbour), over the Dearborn case definition
(1994-5), but also how the OspA vaccines caused the same
systemic, “multi-system,” (Persing and Schoen), "protean" (Ben
Luft) - read, neurologic Great Imitator -, disease as what most
of the rest of us call Chronic Lyme.
How does OspA cause chronic neurologic disease if it is only a
Are there any parallels in the scientific literature about how
if you inject fungal antigens into people that could be a bad
It's a simple thing.
are no Tuberculosis vaccines for the same reason. They put
Thimerosal in vaccines for the same reason. Fungal antigens are
sometimes called superantigens or toxins. Maybe the magazine can
hire someone who knows this little about science.
"Anecdotal reports of inadequately documented
clinical or laboratory associations with B. burgdorferi infection ill serve the medical
community and the public. Convincing and compelling evidence (e.g., like not playing the RNA/DNA
PrimerShellGame with human specimens)
should be required to support
Now that it has been proven
3), re-proven (4,
and re-re-proven (6,
7) once again and in recent history, that indeed “Lyme
9)” is a chronic, un-eradicable infection primarily of the
central nervous system, formerly, formally serologically known as
Relapsing Fever (10,
12), and that long term intravenous
antibiotic treatment generally results in relief-but-then-relapse (3,
5), one wonders, “Where do we go from here?”
The first order of business is the clarification of falsified
testing for “Lyme Disease.”
Allen Steere went to Germany, with, as he claimed,
“The group 1 strain of B. burgdorferi, G39/40, used in this study and
in the previous study of US patients was isolated from an Ixodes
damini tick in Guilford, Connecticut . The group 2 strain, FRG
[Federal Republic of Germany], was isolated from Ixodes ricinus near
Cologne . The group 3 strain, IP3, was isolated from Ixodes
persulcatus near Leningrad . All three strains used in this study
were high passage isolates, which were classified by Richard Marconi
(Rocky Mountain Laboratory, Hamilton, MT) using 16S ribosomal RNA
sequence determination as described [11, 24]. The recombinant
preparations of OspA and OspB used in this study were purified maltose-
binding protein-Osp fusion proteins derived from group 1 strain B31 . The fusion proteins contained the full-length OspA or OspB
sequence without the lipid moiety or the signal sequence -"
plasmid-dropping, antigen-and-antibody dropping, “high passage
strains” and OspA and B with no lipids attached (not likely to produce
antibodies), resulting in the new, 1994, Centers for Disease Control
(CDC), “case definition” of “Lyme Disease
At the Dearborn, MI, Consensus Conference regarding the
“standardization” of Western Blotting for “Lyme Disease,” the invited
labs assessed Steere’s scientifically fraudulent proposal for a new
diagnostic standard for Lyme from 8% to 28% accurate (detected % of
As part of their Dearborn submission, New York Medical College (NYMC)
assessed the Steere proposal in 1993 as:
“Overall, 51 of 59 (86%)
convalescent-phase serum specimens were reactive by IB, 35 of which
were interpreted as positive: 26 based on IgM criteria, 8 based on
both IgG and IgM criteria, and 1 based on IgG criteria,”
or 9 out of
59 patients were positive for Steere’s Dearborn proposal in IgG. Or,
15% accurate (13).
With this Steere-Goes-To-Germany standard the two OspA vaccines (ImmuLyme and
LYMErix) were allegedly assessed for safety and efficacy (14,
which meant 85% of known cases would be thrown out of consideration,
which, hypothetically, was the game plan all along. Later we found
out that the Western Blots in OspA vaccinated people were not readable
due to multiple reactive species or generalized darkening of the
Western Blot strips, rendering any potential bands that would
demonstrate illness/Lyme breakthrough in vaccinated persons,
That leaves us with the years 1992 to the present, almost 2011
In terms of research dollar-years and lives we’re now back at Square
One, wondering what is to be done with the testing for “Lyme Disease,”
and even more importantly, what do we call a “case” of “Lyme Disease”
once the millions of people who were misdiagnosed in the Lyme-OspA
scam process have gone on to chronic illness?
We know from scientifically valid
biomarkers of illness
(scientifically valid in the sense that valid methods were used to
assess the markers or signs that a person suffered real illness or
health irregularities), the true and correct DNA and RNA methods to
determine chronic infections status, and especially of the prominent
question of “seronegativity” being associated with the greatest
apparent suffering. In 1994 at the meeting of the Food and Drug
Administration regarding potential Lyme vaccines, Raymond Dattwyler
(SUNY-Stony Brook) noted that “the ones that failed to mount a
vigorous immune response tended to do worse, clinically. So, there was
an inverse correlation between the degree of serologic response and
In 2005, Mark Klempner (Boston University) and Gary Wormser (NYMC)
reported that the arthritis cases and seroposivity tended to be
associated with Allen Steere’s alleged haplotypes over which
SmithKline was sued in a class action after the Yale-owned LYMErix
trial (18): “Patients generally feel well aside from their arthritis
In 1988, Raymond Dattwyler published that he wondered about
seronegative “Lyme” and the suppression of NK cells (19), and he
wondered enough to come up with a new assay to determine if exposure
to Lyme and a lowered immunological response (20). His new assay was
called “Seronegative Lyme disease. Dissociation of specific T- and B-lymphocyte responses to Borrelia burgdorferi.” Later, Allen Steere
used this same seronegative “Lyme” assay and determined that 4/9 of
his lab workers had been exposed to Borrelia (21).
The question was why.
We know from the previous history of Relapsing Fever (before the era
of Allen Steere, et al), that antigenic variation was the nature of
the relapse, and that spirochetes become host- and tissue- adapted.
This meant that persons who had missed the opportunity for early
diagnosis and treatment would not have the Dearborn “case definition”
of “late, OspA-hypersensitivity, specific HLA-linked, Lyme Arthritis”
in “early Lyme,” would be left to his/her peril in finding a competent
MD who had been exposed in pre-medicine undergraduate study to a
course in genetics. These organisms are taxonomically classified by
differences in flagellin. The best way to detect Lyme with antibody
testing is via all-borrelial-specific anti-flagellin antibody
detection, because flagellin is not a variable antigen. The group
who perfected and patented the anti-Borrelia- burgdorferi specific
flagellin method was Yale’s Erol Fikrig and Richard Flavell in 1991
Why this test was not used to assess Fikrig and Flavell’s
OspA vaccine patent, LYMErix, is anyone’s guess.
Regardless of this State of Nonsense (Connecticut), …
People wonder what to make of the serious illness they suffer as a
result of “Lyme” and LYMErix vaccination, which appeared to be similar
(24). In 1995, David Persing and Yale’s Robert Schoen patented a
method wherein they, the winners, would be in receipt of all the
government funding and also a national monopoly on the - as they hoped
and intended - post-OspA-vaccinated United States:
“Additional uncertainty may arise if the-vaccines are not completely
protective; vaccinated patients with multisystem complaints
characteristic of later presentations of Lyme disease may be difficult
to distinguish from patients with vaccine…. ” (25,
most of all, would have access to all the potentially patentable
goodies in the national blood with this monopoly on testing.
Further investigations into the outcomes of other lipoprotein/fungal
vaccines revealed a similar outcome to that observed in the un-reported LYMErix adverse events patients, resulting in the January
2001 FDA hearing on the matter and the class action lawsuits. Adverse
events were only reported to the FDA by the vaccines trials'
administrators if they were of the arthritis kind, since “arthritis
only” had become the new “case definition” at the 1994 Dearborn
”conference.” When Dennis Parenti of SmithKline reported that there
were only two neurological adverse events to LYMErix at the 2000 Lyme
Disease Foundation Conference in Hartford, CT, several physicians and
attendees immediately got up and walked out of the conference room,
while the rest groaned.
The functional results of OspA vaccination were, we hypothesize, not
dissimilar from the results of structurally similar vaccine antigens
that are managed by TLR2:
“These results are consistent with a model in which the presence of
the 19-kD protein [of Mycobacteria tuberculosis] has a detrimental
effect on the efficacy of vaccination with live mycobacteria.” (27)
“Synthetic analogs of lipopeptides from Treponema pallidum also
inhibited Ag [antigen] processing.”
“the immunosuppressive effect is dependent on glycosylated and
acylated 19-kDa lipoprotein [of Mycoplasma tuberculosis] present in
the phagosome containing the mycobacterium. These results suggest that
the diminished protection against challenge with M. tuberculosis seen
in mice vaccinated with M. smegmatis expressing the 19-kDa lipoprotein
is the result of reduced TNF-alpha and IL-12 production, possibly
leading to reduced induction of T-cell activation. (29)
“Thus, tolerance to LPS and mycobacterial components cannot be
attributed solely to a decrease in TLR/MD-2 expression levels,
suggesting inhibition of expression or function of other signaling
intermediates 2002, Induction of bacterial lipoprotein tolerance is
associated with suppression of toll-like receptor 2 expression.”(30)
"Surprisingly, in 27-kDa-vaccinated mice (protein or DNA vaccines)
challenged by M. tuberculosis H37Rv or BCG strains, there was a
significant increase in the numbers of CFU in the spleen compared to
that for control groups. Furthermore, the protection provided by BCG
or other mycobacterial antigens was completely abolished once the 27-kDa antigen was added to the vaccine preparations. This study
indicates that the 27-kDa antigen has an adverse effect on the
protection afforded by recognized vaccines." (31)
And, we know that:
[((2003) Borrelia burgdorferi-induced tolerance as a model of
persistence via immunosuppression.]
“If left untreated, infection with Borrelia burgdorferi sensu lato may
lead to chronic Lyme borreliosis. It is still unknown how this
pathogen manages to persist in the host in the presence of competent
immune cells. It was recently reported that Borrelia suppresses the
host's immune response, thus perhaps preventing the elimination of the
pathogen (I. Diterich, L. Härter, D. Hassler, A. Wendel, and T.
Hartung, Infect. Immun. 69:687-694, 2001). Here, we further
characterize Borrelia-induced immunomodulation in order to develop a
model of this anergy. We observed that the different Borrelia
preparations that we tested, i.e., live, heat-inactivated, and
sonicated Borrelia, could desensitize human blood monocytes, as shown
by attenuated cytokine release upon restimulation with any of the
different preparations. Next, we investigated whether these Borrelia-specific stimuli render monocytes tolerant, i.e. hyporesponsive,
towards another Toll-like receptor 2 (TLR2) agonist, such as
lipoteichoic acid from gram-positive bacteria, or towards the TLR4
agonist lipopolysaccharide. Cross-tolerance towards all tested stimuli
was induced. Furthermore, using primary bone marrow cells from TLR2-
deficient mice and from mice with a nonfunctional TLR4 (strain C3H/
HeJ), we demonstrated that the TLR2 was required for tolerance
induction by Borrelia, and using neutralizing antibodies, we
identified interleukin-10 as the key mediator involved. Although
peripheral blood mononuclear cells tolerized by Borrelia exhibited
reduced TLR2 and TLR4 mRNA levels, the expression of the respective
proteins on monocytes was not decreased, ruling out the possibility
that tolerance to Borrelia is attributed to a reduced TLR2 expression.
In summary, we characterized tolerance induced by B. burgdorferi,
describing a model of desensitization which might mirror the
immunosuppression recently attributed to the persistence of Borrelia
in immunocompetent hosts”. (32)
AGAIN: “Next, we investigated
whether these Borrelia-specific stimuli render
monocytes tolerant, i.e. hyporesponsive, towards another Toll-like
receptor 2 (TLR2) agonist, such as lipoteichoic acid from gram-positive bacteria, or towards the TLR4 agonist lipopolysaccharide.
Cross-tolerance towards all tested stimuli was induced.”
Epstein-Barr’s activity regarding TLR2:
(2007) Epstein-Barr virus induces MCP-1 secretion by human monocytes
“Epstein-Barr virus (EBV) is a gammaherpesvirus infecting the
of the human adult population in the world. TLR2, a member of the Toll-like receptor (TLR) family, has been implicated in the immune
responses to different viruses including members of the herpesvirus
family, such as human cytomegalovirus, herpes simplex virus type 1,
and varicella-zoster virus. In this report, we demonstrate that
infectious and UV-inactivated EBV virions lead to the activation of NF-kappaB through TLR2 using HEK293 cells cotransfected with TLR2-expressing vector along with NF-kappaB-Luc reporter plasmid. NF-kappaB
activation in HEK293-TLR2 cells (HEK293 cells transfected with TLR2)
by EBV was not enhanced by the presence of CD14. The effect of EBV was
abrogated by pretreating HEK293-TLR2 cells with blocking anti-TLR2
antibodies or by preincubating viral particles with neutralizing anti-EBV antibodies 72A1. In addition, EBV infection of primary human
monocytes induced the release of MCP-1 (monocyte chemotactic protein
1), and the use of small interfering RNA targeting TLR2 significantly
reduced such a chemokine response to EBV. Taken together, these
results indicate that TLR2 may be an important pattern recognition
receptor in the immune response directed against EBV infection.” (33)
And we know that:
“Yale researcher Stephen Barthold, a veterinarian and professor of
comparative medicine who developed the first mouse model of Lyme
disease, studies the expression of B. burgdorferi surface proteins
throughout various stages of the spirochete’s life cycle. He finds
that during the early stages of infection, B. burgdorferi avoids
immune detection by decreasing its expression of surface proteins or
cloaking its expressed surface proteins under a layer of slime. "It's
using some sort of stealth-bomber-type mechanism," he says. Or, using
another diversionary tactic called blebbing, the spirochete can pinch
off bits of its membrane in order to release its surface proteins
Explains Barbour: "It’s like a bacterial Star Wars defense program,"
in which released surface proteins might intercept incoming host
antibodies keeping the spirochete safe from immunological
which is what we call the auto-vaccination with OspA - the immune-suppressing fungal antigen
amd TLR2-agomist known also, structurally, as Pam3Cys
In short, “Lyme Disease” is chronic and seronegative because it is
chronic, or chronically shedding variable (Relapsing Fever-esque)
outer surface lipoproteins like OspA, or, as Alan Barbour states in
his US Patent #6,719,983:
“2.1 Methods of Treatment
”An important aspect of the invention is the recognition that
VMP-like sequences recombine at the vls site, with the result that
antigenic variation is virtually limitless. Multiclonal populations
therefore can exist in an infected patient so that immunological
defenses are severely tested if not totally overwhelmed. Thus there is
now the opportunity to develop more effective combinations of
immunogens for protection against Borrelia infections or as preventive
inoculations such as in the form of cocktails of multiple antigenic
variants based on a base series of combinatorial VMP-like antigens.”
“antigenic variation is virtually limitless. Multiclonal populations
therefore can exist in an infected patient so that immunological
defenses are severely tested if not totally overwhelmed.”
What are the other functional outcomes of the chronic agonism of TLR2
– the one that manages triacyl lipopeptides like OspA or Pam3Cys that
render the immune system overwhelmed, not to mention the persons who late in
the disease who do not serologically (antibodies) react to antigen
from spirochetes fresh out of a tick due to antigen variation,… not to
mention the diminution of antibody production due to chronic TLR2
Says Paul Duray of the National Cancer Institute and Ft. Detrick
"On occasion, these atypical-appearing large lymphocytes have been
misinterpreted in biopsy by several laboratories as cells of a
malignant lymphoma or leukemia. Bb antigens, then, may stimulate
growth of immature lymphocytic suibsets in some target organs, as well
as in the cerebrospinal fluid (Szyfelbein and Ross 1988). Usual
bacterial infections do not produce such lymphocytic infiltrates in
tissue. These immunoblastoid cells in Bb infections at times resemble
those found in Epstein-Barr virus infections. Does Bb reactivate
latent virus infections in tissues? Do some tick inocula harbor
simultaneous infectious agents (ixodid ticks can harbor Rickettsiae,
Babesia microti, and Ehrlichia bacteria, in addition to Bb), producing
multi-agent infections in some hosts? Further studies can clarify
these issues by mans of tissue-based molecular probe analysis." (36)
The lymphocytes of these chronic “Lyme” victims look like
In 2003, Hulinska et al report in
“Interaction of Borrelia
sensu lato with Epstein-Barr virus in lymphoblastoid cells:
“Since the possibility of interruption of latent EBV infection has
been suggested by the induction of the lytic virus cycle with chemical
substances, other viruses, and by immunosuppression, we hypothesized
that the same effect might happen in B. burgdorferi sensu lato
infection as happens in Lyme disease patients with positive serology or both agents. We have observed EBV replication in lymphoblastoid
cells after superinfection with B. garinii and B. afzelii strains
after 1 and 4 h of their interaction. We found that viral and
borrelial antigens persisted in the lymphoblasts for 3 and 4 days.
Morphological and functional transformation of both agents facilitate
their transfer to daughter cells. Association with lymphoblasts and
internalization of B. garinii by tube phagocytosis increased
replication of viruses more successfully than B. afzelii and chemical
inductors. Demonstration of such findings must be interpreted
cautiously, but may prove a mixed borrelial and viral cause of severe
neurological disease.” (37)
One of the characteristics of chronic neurologic “Lyme” is that it is
called an “aseptic meningitis” (38,
This is, of course, while the German Multiple Sclerosis expert Roland
Martin had been recruited by the National Institute of Health to
research the association between “Lyme” and Multiple Sclerosis.(41,
42). Martin went home once he found out that OspA-induced
immunosuppression was the likelier reason “Lyme” was mistaken for
Multiple Sclerosis or vice versa.
DISCLAIMER: We Lyme victims did not dream up this nonsense. We only
Could the co-TLR2 agonists (inducing tolerance and a lack of
antibodies) Epstein-Barr and Borreliosis
be simply responsible for: “These immunoblastoid cells in Bb infections
at times resemble those found in Epstein-Barr virus infections. Does Bb
reactivate latent virus infections in tissues? Do some tick inocula
harbor simultaneous infectious agents (ixodid ticks can harbor
Rickettsiae, Babesia microti, and Ehrlichia bacteria, in addition to
Bb), producing multi-agent infections in some hosts?”(36) ?
It is common knowledge that 95% of the adult population in Western
cultures has been exposed to Epstein-Barr and its common similar
herpes viruses. Hypothetically, there is a mouse herpesvirus
(gamma2herpes) that to our knowledge has not been investigated
regarding transmission to other hosts via ticks. We’re not privileged
to know much about what happens on Plum Island. We only know there is
a “Plum Island” strain of mycoplasma, and that Yale’s Durland Fish
experimented with trying to get African Swine Fever virus to “take” to
local hard-bodied ticks at that Not-Bioweapons/Just-Helpin laboratory
However, it's something that might have been discovered 10 or 15 years
sooner had not certain persons associated with Yale and New York
Medical College made an agreement with Kaiser-Permanente (45) to sell
a vaccine against a disease they suddenly decided doesn’t actually
cause any illness (and the world is flat, the moon made of green
cheese, and all hospitals are going to become brothels because it’s
cheaper and therefore cost-effective to deploy self-alleged MDs who
never perform any scientifically valid rule-outs before making
theoretical diagnostic assumptions based on “projection,” rather than
bog down the likes of the actual laboratories with the fancy machinery
that serves no purpose other than to take up space and look shiny,
just like the misguided folks at Silly NASA…).
“Epstein-Barr virus (EBV) efficiently drives proliferation of human
primary B cells in vitro, a process relevant for human diseases such
as infectious mononucleosis and posttransplant lymphoproliferative
disease. Human B-cell proliferation is also driven by ligands of Toll-like receptors (TLRs), notably viral or bacterial DNA containing
unmethylated CpG dinucleotides, which triggers TLR9. Here we
quantitatively investigated how TLR stimuli influence EBV-driven B-cell proliferation and expression of effector molecules. CpG DNA
synergistically increased EBV-driven proliferation and transformation,
T-cell costimulatory molecules, and early production of interleukin-6.
CpG DNA alone activated only memory B cells, but CpG DNA enhanced EBV-mediated transformation of both memory and naive B cells. Ligands for
TLR2 or TLR7/8 or whole bacteria had a weaker but still superadditive
effect on B-cell transformation. Additionally, CpG DNA facilitated the
release of transforming virus by established EBV-infected
lymphoblastoid cell lines. These results suggest that the
proliferation of EBV-infected B cells and their capability to interact
with immune effector cells may be directly influenced by components of
bacteria or other microbes present at the site of infection.--
Toll-like receptor agonists synergistically increase proliferation and
activation of B cells by epstein-barr virus. (46)
We know that “Lyme” is associated with the production of Amyotrophic
Lateral Sclerosis (in 47% of the ALS cases in Lyme-endemic areas) (47)
and Multiple Sclerosis. And we know that ALS is associated with
mycoplasmal infection (tolerance to TLR2 agonists), and, MS, and
apparently “Lyme” is associated with Epstein-Barr virus or something
like it (36,
We know that 90% of Chronic Fatigue patients found out they had “Lyme
Disease” when finally assessed by a reputable laboratory – one not
like Quest Diagnostics, which uses strain B31, the non-neurotropic,
non-OspC- bearing, non-band-23-producing strain (49). We hypothesize
that since it is known that mycoplasmal infections in the blood
disrupt the osmotic potential in erythrocytes (disrupt the transfer of
oxygen), and metabolism (these things need to be fed, after all) (50,
51), and since there has been reported a high association to
mycoplasmal infections in Chronic Fatigue and Gulf War Illness victims
53), that the tolerance to TLR2 agonists Lyme/LYMErix results in
tolerance to mycoplasma in the blood (Chronic Fatigue) with no
relative antibody production (28).
“Lyme” also produces a Lupus-like syndrome (54), and recently (and
formerly, formally associated with Allen Steere) was linked by the
Allen-Steere-Lupus-Lyme group (now known as the biotech spin-off, “L2-Diagnostics”) with Epstein-Barr (55).
We hypothesize that due to the induction of tolerance to TLR2
like fungal or mycoplasmal antigens (Pam3Cys or OspA blebbing or
vaccination) interrupting Epstein-Barr latency,
"Chronic infection or colonization by mycoplasma(s) could gradually
significantly alter many biologic properties of mammalian host cells
in culture, including induction of malignant transformation. We
examined effects of Mycoplasma fermentans infection on the continuing
survival and immortality of human peripheral blood mononuclear cells
(PBMCs) from healthy blood donors. Without specific supplemental
growth factors, human PBMCs normally die rapidly, with few cells other
than macrophages/monocytes surviving after 2 weeks in cultures. Only
occasional Epstein-Barr virus (EBV)-positive B lymphocytes would
continue to proliferate and undergo spontaneous immortalization. Our
present study revealed that infection of human PBMCs in culture with
the incognitus and PG18 strains of M fermentans, but surprisingly not
with some other strains tested in parallel, markedly enhanced the rate
of EBV-positive B lymphocytes to undergo immortalization (74% vs 17%).
Compared with spontaneously immortalized PBMCs, the PBMCs immortalized
in cultures infected with the mycoplasmas often had prominent
karyotype changes with chromosomal loss, gain, or translocations.
Furthermore, many of these immortalized B lymphocytes were found to be
monoclonal in nature. The in vitro findings would be of relevance to
lymphoproliferative disorders that occurred in patients with immune
suppression. The mycoplasma-mediated promotional effect in cell
immortalization and its potential clinical implications warrant
further study. --2004; Blood; Mycoplasma fermentans infection promotes
immortalization of human peripheral blood mononuclear cells in
the induction of chronic Lyme results in the un-latency of Epstein-Barr. Says one researcher,
Regulation and dysregulation of Epstein-Barr virus latency:
implications for the development of autoimmune diseases.
"Epstein-Barr virus (EBV) is a human herpesvirus hiding in a latent
form in memory B cells in the majority of the world population.
Although, primary EBV infection is asymptomatic or causes a self-limiting disease, infectious mononucleosis, the virus is associated
with a wide variety of neoplasms developing in immunosuppressed or
immunodeficient individuals, but also in patients with an apparently
intact immune system. In memory B cells, tumor cells, and
lymphoblastoid cell lines (LCLs, transformed by EBV in vitro) the
expression of the viral genes is highly restricted. There is no virus
production (lytic viral replication associated with the expression of
all viral genes) in tight latency. The expression of latent viral
oncogenes and RNAs is under a strict epigenetic control via DNA
methylation and histone modifications that results either in a
complete silencing of the EBV genome in memory B cells, or in a cell-type dependent usage of latent promoters in tumor cells, germinal
center B cells, and LCLs. Both the latent and lytic EBV proteins are
potent immunogens and elicit vigorous B- and T-cell responses. In
immunosuppressed and immunodeficient patients, or in individuals with
a functional defect of EBV-specific T cells, lytic EBV replication is
regularly activated and an increased viral load can be detected in the
blood. Enhanced lytic replication results in new infection events and
EBV-associated transformation events, and seems to be a risk factor
both for malignant transformation and the development of autoimmune
diseases. One may speculate that an increased load or altered
presentation of a limited set of lytic or latent EBV proteins that
cross-react with cellular antigens triggers and perpetuates the
pathogenic processes that result in multiple sclerosis, systemic lupus
erythematosus (SLE), and rheumatoid arthritis. In addition, in SLE
patients EBV may cause defects of B-cell tolerance checkpoints because
latent membrane protein 1, an EBV-encoded viral oncoprotein can induce
BAFF, a B-cell activating factor that rescues self-reactive B cells
and induces a lupus-like autoimmune disease in transgenic mice."(57)
Down-regulation of MHC class II expression
through inhibition of CIITA transcription by lytic transactivator
Zta during Epstein-Barr virus reactivation.
"The presentation of peptides to T
cells by MHC class II molecules is of critical importance in
specific recognition to a pathogen by the immune system. The level
of MHC class II directly influences T lymphocyte activation. The aim
of this study was to identify the possible mechanisms of the
down-regulation of MHC class II expression by Zta during EBV lytic
cycle. The data in the present study demonstrated that ectopic
expression of Zta can strongly inhibit the constitutive expression
of MHC class II and CIITA in Raji cells. The negative effect of Zta
on the CIITA promoter activity was also observed. Scrutiny of the
DNA sequence of CIITA promoter III revealed the presence of two Zta-response
element (ZRE) motifs that have complete homology to ZREs in the DR
and left-hand side duplicated sequence promoters of EBV. By
chromatin immunoprecipitation assays, the binding of Zta to the
ZRE(221) in the CIITA promoter was verified. Site-directed
mutagenesis of three conserved nucleotides of the ZRE(221)
substantially disrupted Zta-mediated inhibition of the CIITA
promoter activity. Oligonucleotide pull-down assay showed that
mutation of the ZRE(221) dramatically abolished Zta binding.
Analysis of the Zta mutant lacking DNA binding domain revealed that
the DNA-binding activity of Zta is required for the trans repression
of CIITA. The expression of HLA-DRalpha and CIITA was restored by
Zta gene silencing. The data indicate that Zta may act as an
inhibitor of the MHC class II pathway, suppressing CIITA
transcription and thus interfering with the expression of MHC class
II molecules." (58)
It could be that it’s actually OspA-induced Epstein-Barr that is the
“New Great Imitator.”
And that the Yale-Plum-Permanentes are the Greatest Great-Imitators.
Of either physicians or scientists.
Still, where do we go from here?
“We don’t know.” First, we have to deal with the sugarplum fairy-dancing cocoanut-heads, masquerading as “MDs” and “scientists,” doing
perpetual, eternal lapses and re-lapses between the green cheese moon
and the flat earth on the dot guv dime because they haven’t the plain
old regular nuts to admit that they knew all along - and from the
beginning, 1992, when Allen Steere went to Europe with his “high
passage” plasmid-dropping spirochete strains and “recombinant OspA-B
with the no-lipid attached” to come up with a “diagnostic standard”
for “Lyme” that ended up with none of the two “primary immunodominant
antigens” represented - that OspA was not a vaccine.
-- -- -- -- --
“To be most effective, advances in regulatory
science must be fully integrated into the entire product development
process,” says the new
FDA chief, Margaret Hamburg.
The Biomarkers if Illness, developed
by the ALDF.com or IDSociety.org
And the End of the Almighty Checklist - a sign
of the auto-detonation of the adherents to Psychiatry:
What do the Biomarkers, published by the
same gang who now says "Lyme is a consequence of inadequate sexual
release," say "Chronic Lyme/LYMErix Disease" is?.
Using Dark Field to study the
modified erythrocytes, modified via the tolerance induced from
chronic stimulation of TLR2 by spirochetal blebbing of the likes of TLR2
agonist OspA, and subsequent colonization by mycoplasma [recall that
many sufferers of “Chronic Fatigue Syndrome” have been found to own a
colony of Candida (a fungus)].
Brazil makes fun of US "Lyme" "scientists"
mycoplasma-like and spiroplasma-like organisms in the blood of Lyme-like
See Erythrocytes of RBC in Biomarkers page. Crooks don’t want anyone
using Dark Field to look at the blood. Sweeg went after Lisa Masterson
for making this proposal.
The "We Don't Know What
You Have" Negative Data Rule re the DNA/RNA testing for all the mycoplasma
and activated viral infections that won't be producing antibodies (because some are also TLR2 agonists
like Epstein-Barr, and chronic TLR2 agonism results in no-antibodies (Justin
Radolf: ,” Inhibition of
MHC-II Ag processing by either MTB bacilli or purified MTB
19-kDa lipoprotein was dependent on Toll-like receptor (TLR)
2 and independent of TLR 4. Synthetic analogs of lipopeptides
from Treponema pallidum also inhibited Ag processing. Despite
the ability of MTB 19-kDa lipoprotein to activate
microbicidal and innate immune functions early in infection,
TLR 2-dependent inhibition of MHC-II expression and Ag
processing by MTB 19-kDa lipoprotein during later phases of
macrophage infection may prevent presentation of MTB Ags and
decrease recognition by T cells.”
This may be the
main reason the crooks don’t want us treated with antibiotics- we will
create antibiotic resistant Tuberculosis (because we will likely have
seronegative TB oand other primarily TKR2 agonising infections like
Staph aureus), not to mention, changing the RNA of the likes of
mycoplasma as the Chinese did:
J Clin Microbiol. 2010 Sep 22. [Epub ahead of print]
Nested PCR-Linked Capillary Electrophoresis and Single-Strand
Conformation Polymorphisms for Detection of Macrolide-Resistant
Mycoplasma pneumoniae in Beijing.
QEEG or Fibro-Lenny Sigal’s
(who we’ll now refer to as Lenny Squiggy) findings that persons with
chronic Lyme have changes to electroencephalograms.
http://www.ncbi.nlm.nih.gov/pubmed/7554300 “Abnormal QEEG and/or
EPs were found in 75% of the active Lyme disease patients and in 54% of
the post CNS Lyme disease patients.”
modified cytokine profile
suggested by Lenny Sqiggy in the Cold Spring Harbor Conference
book (1992, Schutzer:) mentioned above, to which Paul Duray attended and
re-iterated his “EBV-modified lymphocytes” findings:
Says Sigal: “Since cytokines can alter endothelial cell function and
increase the entry of inflammatory cells to the organ whose vasculature
has been modified, the presence of cytokines in the Lyme synovium may
have an important indirect effect on local inflammation [Yednock et al.
1992]. The presences of circulating levels of these cytokines may be
the cause of the certain clinical features of Lyme disease; eg., sleep
disorder due to elevated levels of IL-1 (Opp and Krueger 1991) may be
part f the cause of fibromyalgia seen during and after active Lyme
disease (Sigal 1990)
1991, Steve Schutzer.
5) MMP-130 and
GFAp in the CSF of
Neuroborreliosis (which is not "Lyme Disease,"
BTW, because Lyme Disease became "only the HLA-linked knee-presentation"
Nitric Oxide in the CSF of Lyme victims (we now know that the
exposure to these reactive oxygen species/byproducts are downregulators
J Infect Dis. 1994
Borrelia burgdorferi and Escherichia coli lipopolysaccharides induce
nitric oxide and interleukin-6 production in cultured rat brain cells.
Division of Endocrinology, New England Medical Center Hospitals, Boston,
CORRECT Borrelia (all species) DNA/RNA
primers and not the OspA gene,
because that changes according to
Alan Barbour; we can throw out all "studies" in which the
OspA gene was the determinant for infection with the "Lyme"
spirochete [~10 citations]
and the degradants of monoamines in the CSF of borreliosis victims (JJ
brain SPECT and PET imaging
(we now know that the metabolism of erythrocytes are hijacked by the
mycoplasma in the blood - infections we can't fight off because they
have OspA or TLR2 agonizing lipoproteins in them),
NOTE: psychiatry likes to suggest that changes
to the brain perfusion seen in chronic Lyme/Fatigue victims is a result
of Bipolar, when not ever once was anyone who was diagnosed with Bipolar
have shown hyper- or hypo- perfusion as a result of any of the “poles”
without other true, real, scientific valid biomarkers of real illness
first performed as a rule out. Secondarily, when studies have been
performed in which there are brain structural changes to the likes of 17
year olds who are determined to be psychopathic or sociopathic, have
these conditions studied a contribution by trauma. As a third
outstanding parameter, why, we wonder, are lawyers undable to view
anything from a scientific standpoint? Why do they score so low on the
visual-spatial scale of cognitive abilities? Why are lawyers so
left-brain dominant and why do they have no understanding of vocabulary
as a simple means to draw a picture in the mind of another.
Most of all, why is this seen in both lawyers and psychiatrists? Why
are the best scientists and engineers known to be visual-spatial or
right-brain dominant? Is this nature or nurture? The victims of their
chronic fraud crime are not entitled to know why self-alleged scholars
as these are clearly unable to think. We are not entitled to see their
own cognitive differentials and potentials.
anti-phospholipid antibodies (now known to be caused by activated
Epstein-Barr, as reported by Allen Steere's own Lyme/Lupus
reporting team member, Joe Craft at Yale),
[specific to borrelia or not; scientists seem to think an antibody
against flagellin cross reacts with nerve and other tissues, such as
skin and intestine (H. pylori and Campylobacter, producing autoimmune
hives, thyroid disease, GERD...)], [Refs,
Barbour patent, Lenny Sigal]
Biochim Biophys Acta. 1993 Mar
Molecular mimicry in Lyme disease: monoclonal antibody H9724 to B.
burgdorferi flagellin specifically detects chaperonin-HSP60.
Center for Advanced Biotechnology and Medicine, Piscataway, NJ.
anti-heat shock proteins
(a sign of MS), (others besides Sigal) Bands 60, 74, etc.
1989; Total, anti-viral,
and anti-myelin IgG subclass
reactivity in inflammatory
diseases of the central nervous
subclass levels, anti-viral,
anti-myelin basic protein (anti-MBP),
and anti-ganglioside 1
(anti-GM1) IgG subclass levels
were measured in 6 patients with
herpes simplex virus
encephalitis (HSVE), 16 with
borreliosis, 8 with other
bacterial infections, 12 with
multiple sclerosis (MS), 13 with
panencephalitis (SSPE), 5 with
glioblastoma and 12 controls.
Total IgG1 levels were elevated
in cerebrospinal fluid (CSF)
from all patient groups (but not
in the controls), IgG2 in
bacterial infections, IgG3 in
HSVE and borreliosis and IgG4 in
some SSPE patients. The
varicella zoster virus and
rubella) IgG antibodies in MS
were restricted to IgG1,
anti-measles IgG to IgG1 and
sometimes IgG4 in SSPE,
anti-borrelia IgG to IgG1, IgG2
and IgG3. In contrast to
anti-viral antibodies, anti-MBP
and GM1 antibodies belonged to
IgG1, IgG3 or IgG4 in MS. The
nature of the immunological
activation appears to be
reflected in the subclass
patterns elicited in the central
nervous system. Different IgG
subclass patterns in infectious
diseases and MS suggest a
non-specific B-cell activation.
GFAp or glial fribrillary acidic protein in the CSF - signs of
gliosis mentioned by Yale's Robert Schoen when he mentioned
reasons why we need a Lyme vaccine,
http://www.annals.org/content/132/8/661.full.pdf+html (gliosis would
be a sign of multiple CNS degenerative diseases),
peripheral neuropathies and Lyme
meningitis are also seen at this
stage. In late-stage disease,
the central nervous system may
be involved. A new diagnostic
test measuring glial fibrillary
acidic protein in cerebrospinal
fluid may prove to be a useful
tool for measuring such
-- Yale's Robert Schoen talking
about how serious Lyme is, and
that we need a vaccine for this
disease that goes away and has
no illness signs and is
psychiatric and the result of
- performed in monkeys with Lyme but never Lyme-MS Victims which show
Lyme is an active meningitis, and now performed by the Japanese when
deciphering the cause of meningitis in a patient who was mistakenly
diagnosed with MS when they had Large B-cell Lymphoma of the CNS:
Primary central nervous system large B-cell lymphoma with prolific,
mixed T-cell and macrophage infiltrates, mimicking multiple sclerosis.
I think we should fund an
EMG study where we try to find out
whether or not Lyme victims suffer “hysteria” or the
electrification of our hysters. I think this most of all would be a
fun study: Could we find out via EMG that not-enough-sex really causes
Lyme-ALS, or Lyme-MS or Lyme-Lupus, or Lyme-stroke, or bad knees, or
Guillain-Barre? Who wants to volunteer for that study? And what about
men with Low-T? Can that study be performed? Can we women watch?
We’ll rate their manliness while this EMG study of their genitals to
determine if not-enough sex is “The Reason for Low T or the Inability of
Psychiatrists and Lawyers to Think like Scientists or Engineers” is
performed with our own Checklist.
17) T cell proliferation:
Steere references Dattwyler’s report
None of these biomarkers were applied by Mark Klempner from 1997 to 2001
when he determined that “there is no such thing as Chronic Lyme,”
despite being the author of two of the main signs that it is. The first
was Klempner’s MMP-130 only found in the spinal fluid of chronic Lyme
victims and his other two studies from 1992 where he determined and
published the reasons he believed ceftriaxone failed to eradicate all
Now, back to Reason:
What we do know about
such emotionalisms over science is that aggression comes from cowardice
or the fear of being less-than another person. We call it jealousy, we
call is envy and we call it pride, hubris and arrogance. Or we fear not
being believed that we suffer a terrible illness – one not detectable by
antibodies, such as those fungal like stealth infections as Relapsing
Fever or Relapsing Fever plus the synthetic antigen Pam3Cys.
Common sense tells us that you can’t see Multiple
Sclerosis or Lupus, but sometimes medical schools attract and even
accept people who have studied only the likes of English literature or
psychology or philosophy in undergraduate study. These are the toughest
nuts to crack. You can’t teach an old dog new tricks.
hypocrites, rightly did Isaiah prophesy of you:
PEOPLE HONORS ME WITH THEIR LIPS,
BUT THEIR HEART IS FAR AWAY FROM ME.
9'BUT IN VAIN DO THEY WORSHIP ME,
TEACHING AS (G)DOCTRINES
THE PRECEPTS OF MEN.'"
Or, they purport self-flattering, unscientific
concepts as medical doctrine because it is too frightening to see and
accept the reality that they’re truly addicted (repetitious cognitive
loops or mental frameworks) in a medical sense to some philosophical
(psychiatric) Kool Aid, dreamed up by some pervert and drug addict
The biggest of the guns in our arsenal – besides
all of the published applications of the science that says Pam3Cys or
OspA or LYMErix or the HIV antigens gp120 and 41 – are TLR2 agonists and
result in the downregulation of the immune response and the inhibition B
cell apoptosis… is the RNA/DNA Shell Game, we know to be criminal in
1) J Infect Dis. 1992 Aug;166(2):440-4.
Fibroblasts protect the Lyme disease spirochete, Borrelia burgdorferi,
from ceftriaxone in vitro.
Georgilis K, Peacocke M, Klempner MS.
Department of Medicine, New England Medical Center, Boston,
2) Ann Intern Med. 1994 Oct 15;121(8):560-7.
The long-term clinical outcomes of Lyme disease. A population-based
retrospective cohort study.
Shadick NA, Phillips CB, Logigian EL, Steere AC, Kaplan RF, Berardi
VP, Duray PH, Larson MG, Wright EA, Ginsburg KS, Katz JN, Liang MH.
Department of Rheumatology-Immunology, Brigham & Women's Hospital,
Boston, MA 02115.
3) Neurology. 2003 Jun 24;60(12):1923-30.
Study and treatment of post Lyme disease (STOP-LD): a randomized
double masked clinical trial.
Krupp LB, Hyman LG, Grimson R, Coyle PK, Melville P, Ahnn S, Dattwyler
R, Chandler B.
Department of Neurology, Stony Brook University Medical Center, Stony
Brook, NY 11794-8121, USA.
4) Microbes Infect. 2006 Nov-Dec;8(14-15):2832-40. Epub 2006 Sep 22.
Invasion of human neuronal and glial cells by an infectious strain of
Livengood JA, Gilmore RD Jr.
Centers for Disease Control and Prevention, Division of Vector-borne
Infectious Diseases, 3150 Rampart Road, CSU Foothills Campus, Fort
Collins, CO 80522, USA.
5) Neurology. 2008 Mar 25;70(13):992-1003. Epub 2007 Oct 10.
A randomized, placebo-controlled trial of repeated IV antibiotic
therapy for Lyme encephalopathy.
Fallon BA, Keilp JG, Corbera KM, Petkova E, Britton CB, Dwyer E,
Slavov I, Cheng J, Dobkin J, Nelson DR, Sackeim HA.
Columbia University, 1051 Riverside Drive, Unit 69, New York, NY
10032, USA. b...@columbia.edu
Microbiology. 2000 Jan;146 ( Pt 1):119-27.
6) Antimicrob Agents Chemother. 2010 Feb;54(2):643-51. Epub 2009 Dec
Ineffectiveness of tigecycline against persistent Borrelia
Barthold SW, Hodzic E, Imai DM, Feng S, Yang X, Luft BJ.
Center for Comparative Medicine, School of Medicine, University of
California at Davis, One Shields Avenue, Davis, CA 95616, USA.
7) Serum-starvation-induced changes in protein synthesis and
morphology of Borrelia burgdorferi.
Alban PS, Johnson PW, Nelson DR.
Department of Biochemistry, Microbiology, and Molecular Genetics,
University of Rhode Island, Kingston 02881, USA.
8) J Infect Dis. 1994 Feb;169(2):313-8.
Antibody responses to the three genomic groups of Borrelia burgdorferi
in European Lyme borreliosis.
Dressler F, Ackermann R, Steere AC.
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Oh, this is rich, LMAO...
In an era of "personalized medicine" and with Obama's "Brain Initiative"
to replace the officially DEBUNKED psychiatry, it's past time
for this kind of bullshit to be considered TABOO. Actually, it should
be PROSECUTED for medical FRAUD:
"Doctors" don't know dick about medical science.
You know this is true since Thomas Insel had to declare psychiatry "INVALID"
(none of them did, LOL, duh),
We Lyme activists got LYMErix off the market by starting a campaign to
get all the systemic adverse events reported to the FDA (no doctors did),
No "doctor" testified at the FDA 14 years ago explaining and demonstrating
that none of the participates of the Dearborn conference agreed with
Steere's proposal. We got stuck with this RESEARCH FRAUD anyway and it
hasn't been prosecuted yet,
No "doctor" revealed to the FDA that LYMErix caused immunosuppression
rather than "was a vaccine."
No "doctor" sued IDSA over their research fraud "guidelines,"
A SENATOR DID.
No "doctor" confronts the FDA over not assuring the Lyme testing meets
their own criteria for a VALIDATION -
No "doctor" even gives a shit what OspA is. They don't even ASK,
They still don't ask. Yet, there are no other fungal
vaccines. Duh. They have a pretty high opinion of themselves for
knowing nothing other than "Rx --> PAY ME, NEXT!!!"
They don't care what a disease is. All they care about is this
simple, linear "A-->B" formula. Someone walks in, they get an Rx, they
pay, they leave. That's all "doctors" think about. Pharmacists
have better scientific training than "MDs." "Doctors" as dumb as any
other non-Lyme patient. ONLY Lyme victims know what is
going on in the sci-med world. We've been left to our own devices.
We figured it all out. No "doctors" were involved in any
"Doctors" live in a like a Wonderland or Oz or a Medical Matrix. They
can't be trusted because they have no WILL to know, and that
is no more than a character flaw. I'd like to write a new DSM based on
these kinds of character flaws. Maybe that'll be the real "Lyme
Whistleblower" book everyone needs to read.
The reason the CDC is lying about Lyme, ME/CFS and Autism,
is because these devastating outcomes are all caused by the same
mechanisms. Search for a description of "Post-Sepsis Syndrome"
online and you will find it is the same as CFIDS/Chronic Lyme. So,
imagine that kind of brain inflammation seen in sepsis, but in babies
undergoing neurogenesis and neurodevelopment... You can't really
expect a good result in babies, when the
encephalitis damage is
so severe in Chronic Fatigue Syndrome/Post-Sepsis.
Now, importantly, POST-SEPSIS or post septic cytokine storm seen in
Lyme, CFIDS, and Autism babies... comes with humoral immunosuppression and
not classic "HLA-linked autoimmune inflammation." So that's the
information you need to parse out CDC's bullshit. These waste-basket
diseases are NOT classical inflammation or "autoimmunity" with the
self-reacting T cells and the auto-reacting antibodies.
FROM MY FIRST WEBSITE (Nov 2000) - WHERE THE CROOKS SAY...
SHAPIRO: Somehow, for that form of the disease, antibiotics are
effective. They do fine. But then there's some other form of the
disease which is, you can't put your hand around it. *** They don't have
objective findings of inflammation, which is the way bacteria cause
So, it's clear that if you don't have the HLA-linked hypersensitivity
response, you are
not allowed to have a disease. That was the Dearborn scam...
Letter to Pandora,
explaining how they have been hoodwinked by the CDC:
"CFIDS/FM is/are not “neuro-endocrine-immune diseases.” They/it are really actually chronic active herpesviruses, immunosuppression with opportunistics, much like AIDS (no T cell killer virus, just T cell anergy).
"First, have a look (in the references, here) at the recent description of “post-sepsis syndrome” (PSS) and the statements/reports by Washington University St. Louis, MO. (wustl.edu) and the NIH confirming that model on what this disease is, how it comes about, (1-8), the older immunosuppression data on fungi like mycoplasma and mycobacteria- formerly called epERYTHROzoons - clue (9, 1953), past fungal vaccines failures and what those failure-outcomes looked like (10-14),
and of course the Autism pandemic, which parallels all these mechanistically (15-21, 51).
"I would like you to note that in this report, I put a lot of the explanations in the references section. Since CFIDS people have cognitive issues (53), particularly right brain issues or visual-spatial processing deficits, I have to make the overall picture as brief as possible in the body of this report."
This is the year the Lyme cryminals are going to get BUSTED or
we are ALL going to know why (usda.gov, Plum Island, & BigVaccines).
We're going to Occupy the USDOJ (Washington, DC) in May-July, 2015 and all
abused groups are welcome. The primary reason for the abuses of Lyme
and similar (Gulf War Illness, Chronic Fatigue, Fibromyalgia) victims is
that the CDC does not want anyone to know how the childhood vaccinations
are causing Autism,... because those mechanisms are the same in Lyme,
GWI, Fibromyalgia, ME/CFS. The children are getting the viruses
instead of the protection due to immunosuppression. And the
childrens' immune systems are being ruined by all the hypervaccination,
explaining the epidemics of allergies, asthma, ADD, and leukemia in
American children, too.
So, everyone is welcome to show up at the Occupy and stay as long as
you like. Come and in Solidarity, demand Justice. Remember,
"America" used to be short for "WE'RE NOT PUTTING UP WITH THIS
Understanding the Govt's Hired Psychopaths:
I just had another thought on this matter of how assholes germinate
and grow into psychopaths (psychiatrists, the CDC, and similar vicious
tards). That is, as long as we're talking about the replacement for
scientifically invalid psychiatry:
Does everyone remember that Susan O'Connell beeotch who used to work for
Porton Down and the UK NHS? How she was the gatekeeper for Lyme/TBDs
in the UK,... and worked with the USA Lyme crooks,
And how she leaked to Lady Mar (who basically goofed her out of it), that
"Porton Down was no longer working on Borrelia as a bioweapon"
and that they had "given it over to industry?"
It wasn't really given over to industry. Edward McSweegan, the
arrogant moron (showing the dynamic between arrogance and stupidity),
cheated the US Navy out of their TBD's funding with his fake Senator
Goldwater "Whistleblower" Letter in 1986. Those are the morons who thunk up
a vaccine for relapsing fever, the very thing that defies the concept of
So, what is wrong with Sweeg's brain? Why is he a crazy, vicious
moron, like his buddy Durland? How did Sweeg become a psychopath?
He reveals it here:
"As a graduate student twenty years ago, I had a departmental
recruiting poster tacked up on the wall next to my desk. It read, in
part, "If you are curious, patient, and *** awfully damned intelligent,"
consider a Ph.D. in microbiology." In 1984 a degree in microbiology
seemed like a good idea."
He had convinced himself he was UNIQUE!!! SPECIAL!!! BRILLIANT!!! It's
always the same thing. Self-idolatry - believing there is something UNIQUE!!
or SPECIAL!! about yourself. It is the same reason Lucifer fell and the
source of all the trouble in the world. Same vanity. Same Influence of
Spirit to believe you're above the rest.
But evil has no creative abilities. That is
probably why it took the HEAD OF THE NIMH to finally declare psychiatry to
be scientifically invalid. Notice this did not come from psych.org. None of
them thunk up whether or not psychiatry was valid. They, all
of psychiatry, did not have THE INSIGHT (keyword).
And speaking of the CDC/Porton Down and beeotches named Susan, let's keep
in mind that
Vernon (CDC officer) who threw out the cells to which mycoplasma
adhere or live inside of, when trying to show that mycoplasma are not
involved in Chronic Fatigue Syndrome:
22) CDC’s Lies: “Absence of Mycoplasma species DNA in chronic fatigue syndrome”, throwing out the blood cells in order to find no mycoplasma:
“Blood was collected in sodium citrate Vacutainer tubes (Beckton Dickinson) and shipped by overnight courier to the Centers for Disease Control (CDC), where plasma was collected by separation on lymphocyte separation medium (LSM; ICN Biomedicals). Plasma (1 ml) was concentrated to approximately 250 μl in a Centricon centrifugal filter unit YM-100 (Millipore).
Cell-free plasma DNA was extracted by using a QIAamp DNA Mini kit (Qiagen) according to the manufacturer's instructions and quantified by using a DyNA Quant 200 fluorometer (Amersham Biosciences).”
They're all like that. Where do they get off being psychopaths?
There's your answer. They have some false idea that they're superior
creatures, just like the NAZIs. But it's transparent and imbecilic.
Psychiatry claims Somatoform diseases are where the alleged victims
have scientifically valid signs of illness or detectably pathophysiologies
in the absence of real disease - which is the very definition of
magic or paranormal, -
yet they otherwise ignore
all the evidence of paranormal (or paraphysical, as I call it, which refers
to intelligences without physical bodies) events.
You have to think long, hard and deep about that. What does it mean
when you have selective cognition, yet claim to be, and charge to be, and
claim to be an expert in legal cases, and in the magnitude of lives they
ruin who are genuinely sick with real disease like, as you have seen, CFS,
ME, Lyme, Fibro, etc., worldwide.
One more recommendation: Read Hostage to the Devil by
Malachi Martin and watch the subtle way Martin trashes these idiots,... and
what the demons reveal about psychiatrists ;)
Rings true, because it is.
BAD-ASS Lyme Cryme Website:
... "When it comes to the Lyme criminals – those
associated with Yale, LYMErix, and the Dearborn stunt (involving CDC
officers), our silence is what keeps them alive in this war between the sick
and the unsick. Their strategies are obvious, and involve the three rewards
most valued by psychopaths – money, power and fame – in addition to their
desire to stay out of prison. Case in point: the University of Kansas doctor
credited with “discovering” the Bourbon virus. His patient died, yet he is
rewarded with the fame of discovering a new, deadly virus that possibly
isn’t deadly at all, but an innocent bystander in the fungal-viral-bacterial
septic soup that the CDC can’t acknowledge for fear of losing all
credibility, once and for all. (Plus that prison thing.) He’ll likely patent
the organism, then get research funds (our tax dollars at work) to invent a
vaccine, which will generate more fame and fortune (in the form of
royalties) not only for him, but his employer, the State of Kansas....
Lyme Crymes Video Playlist (like a
Powerpoint presentation on YouTube, stop at each frame and go at your own
VID1 FLAGELLIN ONLY IS VALID for diagnosis of Lyme or any borreliosis
There has been an FDA-Validated method to detect Borreliosis and even
Lyme borreliosis since 1991. Yale patented it under US 5,618,533. It
detects early, late, neurologic, and all cases of Lyme. Because these
crooks pulled the Dearborn stunt later, their vicious antics you currently
see, and abuses against Lyme victims is intended to prevent homicide charges
over the Dearborn case definition.
VID2 BRAIN PERMANENT
This is just the beginning of the data on persistence
and viability of the spheroplast form. I will add more on this later. See
VID3 RELAPSINGFEVER VS OSPA DISEASE
This video, 3, shows the Lyme Cryminals knew LYMErix
caused the same "multisystem disease" as we know of as Chronic Lyme. They
changed the definition of the disease at Dearborn because they knew LYMErix
vaccination caused the same disease. They were prepared for that outcome
since they published a book in 1998 anticipating those outcomes and
especially as regards children who potentially became ill from this vaccine.
They intended to accuse the parents of Munchausen's by Proxy.
VID4 OSPA TOXICSHOCK SEPSIS
This video shows you how OspA causes the
immunosuppression state of Chronic Sepsis or chronic active herpesviruses as
result of being exposed to fungal antigens. These are a series of references
by Dattwyler, Harding, Medvedev, etc and re tolerance to other antigens such
as viral (TR7/9) and bacterial lipopolysaccharide (TLR4-agonist). Your
disease is polymicrobial, ongoing sepsis, much like AIDS.
VID5 Dearborn Falsified Lyme Case Definition
Now that you have seen the first 4 videos and especially the 4th one on
how Lyme causes systemic disease, like a chronic sepsis, with the
ongoing schmorgasbord of reactivated herpesviruses, tolerance to other
infections (bacterial), and that this is an AIDS-like disease... you
want to know which criminals are behind this scam and how they pulled it
VID6 HLA NEGATIVE DISEASES AND IDSA GUIDELINES
No one knows what Dearborn means, Steere does not
know what he is talking about, SmithKline makes fun of him, Klempner's
"re-treatment" "study" was anything but, yet is the basis of the IDSA
"Guidelines on the Diagnosis and Treatment of Lyme Disease," all of
IDSA's aggression - including the "Guidelines" - towards VERY sick
people is about avoiding a prosecution over the Dearborn case definition
Be sure to review the
PrimerShellGame to dispel the rumor that "biofilms" are the reason
spirochetes are antibiotic resistant. Spirochetes are individual
operators. They do not cluster in vivo in mammals.
VID7 STEERE LYME FUNGAL CAUSES EVERYTHING
It's hard to think of what to say about this. In
1991, Steere admitted Lyme caused everything, from RA to MS to
Fibromyalgia. Later of course he claimed Lyme only caused arthritis.
IDSA still claims Lyme only causes a hypersensitivity response in a knee
with no other symptoms. Nevertheless, the topic seems to be how the CDC
is so good at hiring and creating psychopaths.
VID8 DNA RNA
The DNA/RNA gene shell game. This is a short
version of this aspect of the crime. The Lyme crooks use the wrong DNA
to not find Lyme, say OspA could never be a vaccine due to antigenic
variation, and use this nonsense gene to NOT FIND LYME when convenient.
But they use the RIGHT DNA when they want to patent something. This
information will be very useful to the prosecution since it is so
obvious and blatant, and the Klempner non-retreatment report is
fraudulently restricting diagnosis and care,... as you will see.
Importantly, we have no MDs in America with any
basic science training or you would not be hearing/seeing this from me.
(It's a bit much for the patients to have to do all the research, but
that's America, Land of the Boneheads.)
RICO Patents, etc.
FUNGI ACTIVATING INACTIVE VIRUSES,...
AND STRESS (CORTISOL) ACTIVATING EBV/SIMILARS in ASTRONAUTS AND MEDICAL
1) Lyme and LYMErix (fungal, and not bacteria but their
own phylum) result in Lupus and MS (both known to be caused by
THOSE ^^^ 2 (MS and Lupus) are "AUTO-IMMUNE" with
the lots and lots of antibodies against, in large part, the SECONDARY
infections, EBV/similars. But not everyone has the HLAs for "autoimmune"
2) The new SEPSIS reports by wustl and the NIH (fungal
antigens shut off the immune response ENTIRELY as shown in the numerous
reports I have written this last 2 months). *** It is FUNGI or TL2/1
agonists that do this, mainly *** as shown in the videos
3) TWO REASONS ME/CFS VICTIMS ARE TREATED LIKE CRAP BY
THE MEDICAL COMMUNITY:
^^^ THEY HAD IT TOO, BUT NOT WITH FUNGI ^^^^
4) MOLD AND EBV:
And I have found more such reports in PubMed, you can
5) -- THE ONE BIG LIE the CDC TELLS, re ME/CFS:
CDC’s Lies: “Absence of Mycoplasma species DNA in
chronic fatigue syndrome”, throwing out the blood cells in order to
find no mycoplasma:
“Blood was collected in sodium citrate Vacutainer tubes
(Beckton Dickinson) and shipped by overnight courier to the Centers for
Disease Control (CDC), where plasma was collected by separation on
lymphocyte separation medium (LSM; ICN Biomedicals). Plasma (1 ml) was
concentrated to approximately 250 μl in a Centricon centrifugal filter unit
YM-100 (Millipore). Cell-free plasma DNA was extracted by using a QIAamp DNA
Mini kit (Qiagen) according to the manufacturer's instructions and
quantified by using a DyNA Quant 200 fluorometer (Amersham Biosciences).”
Bitch centrifuged out all the cells to which mycoplasma
adhere or live inside of, then looked for mycoplasma in a test tube full of
nothing. Cute. That's the CDC and Susan (Suzanne) Vernon for ya.
6) CDC: Measles, Mumps, and Rubella -- Vaccine
Use and Strategies for Elimination of Measles, Rubella, and Congenital
Rubella Syndrome and Control of Mumps: Recommendations of the Advisory
Committee on Immunization Practices (ACIP)
"Updated information on adverse events and
contraindications, particularly for persons with severe HIV infection,
persons with a egg allergy or gelatin allergy, persons with a history of
thrombocytopenia, and persons receiving steroid therapy [are
immunosuppressed - KMD]."
7) CDC: Human Exposure to Brucella abortus Strain
RB51 -- Kansas, 1997
In the above, an immunosuppressed pregnant cow was
given a Brucella (LYMErix-like) "live, attenuated" vaccine and the baby cow
ended up with the disease, which then was transferred to the humans handling
the cow and her dead baby. This parallels what is happening to children who
are vaccinated while immunosuppressed, or who receive mycoplasmally (LYMErix-like)
8) Fungi Activate Viruses, Rockefeller, 1953 [Go back
to the 1950s to see Rockefeller Bioweapons Inc experimenting with mycoplasma
antigens like OspA - Enhancing Mouse Leukemia Virus, just like fungally-contaminated
vaccines do, which was the reason they put Thimerosal in vaccines, to
prevent fungi from doing this (Clue: HIV and "Lyme" bearing the same
immunosuppressing triacyl lipopeptide fungal antigen, OspA]:
ACUTE HEPATITIS ASSOCIATED WITH MOUSE LEUKEMIA
IV. THE RELATIONSHIP OF EPERYTHROZOON COCCOIDES
TO THE HEPATITIS VIRUS OF PRINCETON MICE
"In Swiss mice, animals with high natural resistance to
hepatitis virus, the pathogenicity of this agent was markedly enhanced by
combined infection with eperythrozoa. Eperythrozoa were maintained
throughout 18 successive passages in normal Princeton and Swiss weanlings
with intact spleens. The combined infection of Princeton mice with
eperythrozoa and the virus component of Gledhill, Dick, and Andrewes, which
is nearly inactive when injected alone, resulted in acute hepatitis with
9) The effect of exogenous corticosterone on
West Nile virus infection in Northern Cardinals (Cardinalis cardinalis)
“Corticosterone was administered at levels that
individuals enduring chronic stressors (i.e ., long-term inclement weather,
food shortage, anthropogenic pollution) might experience in the wild.
Corticosterone greatly impacted mortality: half of the corticosterone-implanted
cardinals died between five - 11 days post-inoculation whereas only one of
nine sham-implanted (control) birds died.
”No differences were found in viral titer between
corticosterone- and sham-implanted birds. However, cardinals that survived
infections had significantly higher average body temperatures during peak
infection than individuals that died.
”In sum, this study indicates that elevated
corticosterone could affect the survival of WNV-infected wild birds,
suggesting that populations may be disproportionately at-risk to disease in
When this happens in Medical school students and
astronauts - the activation of EBV by CORTISOL and not some mysterious brain
energy weapon called somatoformia - they call it something real: EBV.
10) NYTimes reveals Thimerosal is put in vaccines to
prevent FUNGI since it is well known that FUNGI activate viruses.
NYTimes; Doctors admit Thimerosal is put in vaccines
to prevent fungi:
Vaccine Rule Is Said to Hurt Health Efforts (Dec, 2012)
"But a proposal that the ban include thimerosal, which
has been used since the 1930s to prevent bacterial and fungal contamination
in multidose vials of vaccines, has drawn strong criticism from
"They say that the ethyl-mercury compound is critical
for vaccine use in the developing world, where multidose vials are a
"'Banning it would require switching to single-dose
vials for vaccines, which would cost far more and require new networks of
cold storage facilities and additional capacity for waste disposal, the
authors of the articles said.'"
^^ They are revealing why so few doctors' own kids get
vaccines-acquired brain damage. Their kids probably get the first shot out
of the vial.
11) OspA acts like BCL2, inhibiting apoptosis
Targeting proteins computationally
"Apoptosis is regulated by both pro- and anti-apoptotic
members of the B cell lymphoma-2 (Bcl-2) family of proteins. Epstein Barr
virus (EBV), the causative agent of Burkitt’s lymphoma, encodes a
pro-survival mimic of these Bcl-2 proteins called BHRF1 that inhibits
cellular apoptosis during infection and is thought to contribute to
BTW ^^ they're fans of ActionLyme, Fred Hutch Cancer :)
You would think that in the 20th and 21st centuries we
in the English speaking world would have some sort of a
collective idea on how to prevent all but a couple' percent of our
populations from falling through the cracks into poverty, near-starvation,
homelessness, chronic illness, drug addiction, getting nothing but
assholes-and-morons for politicians, our childish "foreign policy," having
to suffer the cognitive-deficits-in-journalism, etc. But when you
consider who are the characters who have self-assigned themselves the
Authority, or are the planners, or are the people who originally thunk up
the idea that post-WWII, America was going to rule the Earth (Rockefellers,
Israelis, Banksters, the Petro-fellas), you would think they would have come
up with some sort of a moral theme or motif or humanness concept that ties
together the plight of Earth itself, with harmonious cooperation between
needs and resources. But that never enters the Capitalists' minds.
No. They were convinced by some diabolical influence that humans are
mere animals, competing as animals to be the fittest.
Now consider the word "fit." When you think of "fit," does some bankster or
lawyer or salesman come to mind? When someone like Mr. Kahn of Kahn Academy
comes along and blows the entire Education industry out of the water - from
out of nowhere - what does that say about the Establishment? What are
we doing wrong? Who comes up with the idea that a formal education on
education makes you a teacher? They consumed this education on
education but by what criteria are the planning and the protocols weighted?
Why do we have the entire CDC, NIH, AMA, FDA, DHHS, and all of medical
mainstream America not aware of the fact that there is no Tuberculosis
vaccine for the same reason there is no Lyme vaccine, ... and is the same
reason we put Thimerosal in vaccines (fungi = bad)? How dare the NIH
and CDC (not to mention Yale) tell me they don't know what the Lyme vaccine
Where does anyone get off saying they should be paid for being an expert,
when they admit they do not know the basic things about human illness and
disease ("doctor," or HMO CEO)?
The world now dominated by the USA has no cohesive plan or idea. No
reputable intent. The United States Government are a bunch of animals
who think their job is to control the other animals. Fear is their essential
theme and motif.
The world is ruled by a bunch of cowards.
And that's funny to me because COWARD was the thing I was
always taught was the LAST thing you'd ever want to be.