Blowing the Whistle at the FDA, Jan 2001, exposing Dearborn and how OspA causes immunosuppression rather than, "was a vaccine."
 

Go ahead and save all these cryme files to your hard drives: File List, RICO. 

Most people are aware by now, thankfully, that the US Govt and especially the HHS.gov are 200% brainless and incompetent.  OspA never could have been a vaccine; click this link and see for yourselves.  

We have a 9th Criminal Charge Sheet, about how rOspA was never proven to prevent spirochetes, those were all "bogus articles" and research fraud.  You'll be able to see that all the HATRED towards Lyme victims really had to do with the fact that these rOspA vaccines were entirely faked "qualifications" and used ridiculous experiments to show they prevented Lyme.  But no vaccine ever prevented spirochetes, not even the dog vaccines.

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As many of you Lyme and CFIDS campaigners know, we lobbied in Washington, DC, June 2017 for a hearing in the Senate regarding the Lyme disease crimes that we want referred to the Justice Department for prosecution.  On this page, please find all the information handed out and the presentation we gave the Congressmen's assistants and legal staff/lawyers.

The presentation primarily had to do with all the previous attempts to have the Lyme crymes prosecuted such as the 2006 Blumenthal lawsuit, the RICO complaint filed in 2003, the fake vaccine going down (2002), and the more recent attempt in 2014 by Senators Durbin, Blumenthal, et al, to force the FDA to finally do their jobs and look at the falsified case definition for Lyme we call "Dearborn."

To be perfectly clear:  You can't have a vaccine against a disease known to shed surface antigens that are fungal and known to cause immunosuppression.  Notice there are no vaccines against Tuberculosis or other fungal diseases.  To get around this scientific dilemma, CDC staff who own patents for vector borne diseases (ALDF.com) simply declared that only the hypersensitivity or HLA-linked allergy outcome would be allowed to be a "case" of "Lyme disease" at a fake consensus conference in Dearborn, Michigan, in 1994. 

Contrary to the foolishness of the non-profits of Lyme and ILADS.org, the CDC is not "endorsing IDSA's guidelines."  IDSA is endorsing CDC staff crime by endorsing CDC's falsified case definition, from which the IDSA "guidelines" devolved.  It is the CDC, Yale, UConn, NYMC and New Jersey Medical and Dental, mainly, behind this scam.  The State of Connecticut participated in Yale's/CDC's crimes, particularly as regards the falsified pediatric LYMErix vaccine trial.  The States of Connecticut, New York and New Jersey will be on the hook for damages, along with Yale.  Possibly even Massachusetts.  Consider:  "The New England Journal of Medicine" cant even get this straight.


No one should trust those 3 states (NY, CT, NJ), primarily, with any more money for "Lyme research."  All such statements and requests are merely stalling tactics.  We already know OspA alone caused the post-sepsis outcome, which is mostly viral (reactivated EBV, CMV, etc., tolerance to all sorts of other TLR agonist types besides TLR2/1).  When they claim they need more money, what they want to do is stall the federal Justice Department prosecution and subsequent class action lawsuits against them by the other 46-47 states, just like Enron and Madoff.

Everyone should be thinking in terms of damages, every state government, that is.  This is going to be way bigger than Enron.   Connecticut will have to have a Going Out of Business sale, where all the State's parks and attractions - along with all of Yale's properties like the Beinicke Rare Book museum and the Peabody - will have to be sold off to  private entities.  Taxes will go through the roof.  ... CT in particular because of the fake pediatric LYMErix trial.  This is technically "assault (see the charge sheets on truthcures.org)."

 

Stuff you cant make up.  See Allen Steere describe how you can diagnose Lyme clinically in 1988 (and also says Lyme is like a B cell leukemia):



https://www.linkedin.com/pulse/things-lyme-cryminals-said-before-1990-when-founded-aldfcom-dickson
 

This sets up a category of people to sue in a class action.  Importantly, all the slander and libel by the ALDF.com, the CDC, Yale staff and others, assure this is a RICO charge, which means people can go after these Lyme criminals for their personal property.  So, not only will they get jail time, but if they ever get out of jail, they will not have a cent to their names.


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Free 200+ page scientific book on the various fraud crimes committed by CDC and Yale employees.  Includes information on how the pediatric vaccines fail by giving the children the very brain damage these viruses these vaccines were intended to prevent (particularly the MMR), according to IDSA, NYU's Stanley Plotkin, and Johns Hopkins' Paul Auwaerter.  

Also included are details about how the University of Connecticut "assaulted" Czech children with a non-vaccine (LYMErix) that they knew would do those children no good.  There was NO chance that vaccine would have prevented Lyme, not only because OspA is pam3cys, a triacyl-, fungal lipoprotein which causes immunosuppression, but because there is none of the USA Borrelia strain B31 OspA in Europe.  

We show that Allen Steere published in 1988 that Lyme caused a B cell mutation (lymphoma-like) that rendered them incompetent and that that's actually the disease IDSA, the CDC, Yale and UConn deny exists and wrote out of the "case definition" because that immunosuppression disease was also caused by LYMErix and they knew this in 1993 - before the Dearborn stunt (where the CDC falsified the testing for Lyme, 1994). 

We show NIAID's "Director" Anthony Fauci even has a patented treatment for fungal immunosuppression diseases, yet he claimed on national TV (Lou Dobbs) that "We have a scientifically validated Lyme vaccine, but it's not well used" - 1.5 years after it was ordered off the market by the FDA via ultimatum, in February, 2002. 

We show that Simon Wessely, et al, while finding that Gulf War Illness was associated with the vaccines, later claimed the Veterans were just cowardly airy-fairies and are using "brain magic" (like telekinesis) to fake their scientifically valid illness signs.  (Yes.  That is what "somatoform" technically means.) 

The Criminal Charges Sheets - this is actually a book; you can also find it here:      truthcures.org/charge-sheets


1. ALDF-CDC "Enterprise" (read "RICO") Conspires to Defraud USA in Dearborn-Vaccine Scam; see how next, in the subsequent charge sheet on patents, the very people who falsified the testing are the ones who own the patents for the bogus vaccines and test kit products:

2. Lyme Disease Patents owned by the Dearborn scammers, CDC officers, Yale in association with Corixa, Mayo Clinic and Imugen. Leaving OspA and B out of the Dearborn standard was intended to facilitate a monopoly on post-LYMErix approval on blood testing for all vectior-borne disease:

3. Lyme Disease Biomarkers, as compared to scientifically invalid psychiatric check lists. These biomarkers were identified by the very people who later said Lyme was not even a disease, and who are the same people who own the vaccine patents and falsified the testing at Dearborn:

4. The Primers (DNA, RNA) Shell Game; the very people who own all the patents and falsified the testing for Lyme in order to falsify the outcomes of those bogus products, use the wrong DNA to not-find Lyme or other spirochetes in humans, while using the correct DNA to patent borrelia-specific DNA; no biofilms.

5. Occam's Razor -- Or, If it quacks like a duck, it must be Epstein-Barr/post-sepsis syndrome (as the REAL "Great Imitator"):

6. The Common Mechanisms of Fungal-Viral Damage in CFIDS, Vaccines-Autism, and "Chronic Lyme"/ New Great Imitator, per the CDC, NIH and IDSA; This paper reveals the CDC's own data on what Lyme and CFIDS are, and how immunosuppression-via-fungal contamination also explains the failed childhood vaccines, giving children the very viruses the vaccines are intended to prevent (with resultant encephalitis):

7. Simon Wessely and the abuse of Gulf War veterans, Justina Pelletier and 21st century witch trials; with scientifically valid evidence for real illness, a vast majority of post-sepsis and vaccine injured are slandered and libeled with invalid psychiatric terminology:

8. The State of Connecticut and Yale Assaulted Czech Children with a known fake vaccine (OspA or LYMErix) just to see how serious would be the adverse events.

9.  New-  About how the OspA vaccines were never shown to prevent spirochetes, this was all research fraud.

All in one pdf, HERE
 

161219 - Lyme Crooks Wormser and Steere - and even the "CDC officer" criminal Paul Mead - finally admit Late Lyme and LYMErix diseases are immunosuppression outcomes.  
'Say the "TLR2/1 agonism" (immunosuppression) is probably the "more important" driver of the disease outcome.

Abstract (published Dec 15, 2016):  https://www.ncbi.nlm.nih.gov/pubmed/27976670

Full Text:  http://www.actionlyme.org/Steere_Wormser_Admit_Immunosuppression.2016_Dec_15.pdf
 

Mario Philipp has long been the guy who says Borrelia and OspA causes immunosuppression, so look at all of his reports:
https://www.ncbi.nlm.nih.gov/pubmed/?term=Philipp+and+borrelia

Ref 79, above, (Mario Philipp): Interleukin-10 anti-inflammatory response to Borrelia burgdorferi, the agent of Lyme disease: a possible role for suppressors of cytokine signaling 1 and 3.
https://www.ncbi.nlm.nih.gov/pubmed/16988256
 

You will recall that when I blew this whistle at the FDA on Lyme (Dearborn) and LYMErix on Jan 31, 2001, I mentioned Dattwyler's "suppression of Natural Killer cell activity" report in response to Lyme and OspA, and I also mentioned that Mario Philipp had published that OspA caused immunosuppression via IL-10:
http://www.fda.gov/ohrms/dockets/ac/01/slides/3680s2_11.pdf


The "Govt" has never thanked us for getting rid of that TLR2/1-agonist fungal toxin of a non-vaccine.

 

The crooks report above, saying the more serious disease outcome may be due to its fungal-ish agonism of TLR2/1, is a little bit of a screwed up way of saying it, but they, the CDC and the Lyme criminals, finally admitted their whole Lyme story was a lie.   Notice that ILADS has NEVER said what Late Chronic Lyme was.  They lost their opportunity to be the ones to bring the truth forward as I had predicted, also.
 

Steere writes about deformed B cells, later shown to be an outcome of LYMErix, too:
Remember, OspA is Pam3Cys...

Ann N Y Acad Sci. 1988;539:65-79.
Clinical pathologic correlations of Lyme disease by stage.
Duray PH1, Steere AC.

”....Soon after the onset of ECM, the organism disseminates hematogenously, with what appears to be random dispersal throughout the body. The immune response involves virtually all of the organs and structures of the reticuloendothelial system including the bone marrow, and clinical pain and discomfort seems to correlate with hyperplasia of lymph nodes and spleen and bone marrow. Diffuse visceral involvement in this acute stage mimics infectious mononucleosis or disseminated viral syndromes. These include conjuctivitis, pharyngitis, pneumonitis with dry cough and mild pleuritic pain, hepato-splenic tenderness, lymph node swelling of the neck and groin, and orchitis. There is lymphoid hyperplasia of the lymph nodes and spleen consisting of prominent germinal centers and numerous perifollicular lymphocytes, with proliferation of plasma cell precursors and mature plasma cells. The plasma cell precursors are large, appear tumor-like, and can resemble Reed-Sternberg cells. Others look like typical immunoblasts (FIG. 1). In one example, cervical lymph nodes show cell degeneration with karyorrhexis and nuclear debris of lymphoid elements. This patient had repeated high fevers and marked discomfort of neck nodes. Large atypical immunoblasts can also be seen in the spleen and bone marrow. The red pulp of the spleen is congested, not unlike that seen in infectious mononucleosis. Spirochetes can be demonstrated in the lymph nodes, spleen and bone marrow and liver. There is a transient hepatitis reflected by elevated liver cell enzymes such as SGOT, SGPT, and GGT. The liver can vary from a mild lymphocytic portal triaditis all the way to liver cell derangement that simulates acute viral hepatitis. The cells at this stage appear swollen with clear cytoplasm and microvesicles of fat (FIG. 2). Numerous leukocytes are seen in the sinusoids, and there is Kupffer cell hyperplasia..."
https://www.ncbi.nlm.nih.gov/pubmed/2847622
http://www.actionlyme.org/clinical-pathologic-correlations-of-lyme-disease-by-stage-Steere-Duray.pdf 

J Immunol. 2005 Jun 1;174(11):6639-47.
Role of TLR in B cell development: signaling through TLR4 promotes B cell maturation and is inhibited by TLR2.
Hayashi EA1, Akira S, Nobrega A. 
Author information
AbstractThe role of TLR4 in mature B cell activation is well characterized. However, little is known about TLR4 role in B cell development. Here, we analyzed the effects of TLR4 and TLR2 agonists on B cell development using an in vitro model of B cell maturation. Highly purified B220(+)IgM(-) B cell precursors from normal C57BL/6 mouse were cultured for 72 h, and B cell maturation in the presence of the TLR agonists was evaluated by expression of IgM, IgD, CD23, and AA4. The addition of LPS or lipid A resulted in a marked increase in the percentage of CD23(+) B cells, while Pam3Cys had no effect alone, but inhibited the increase of CD23(+) B cell population induced by lipid A or LPS. The TLR4-induced expression of CD23 is not accompanied by full activation of the lymphocyte, as suggested by the absence of activation Ag CD69. Experiments with TLR2-knockout mice confirmed that the inhibitory effects of Pam3Cys depend on the expression of TLR2. We studied the effects of TLR-agonists on early steps of B cell differentiation by analyzing IL-7 responsiveness and phenotype of early B cell precursors: we found that both lipid A and Pam3Cys impaired IL-7-dependent proliferation; however, while lipid A up-regulates B220 surface marker, consistent with a more mature phenotype of the IgM(-) precursors, Pam3Cys keeps the precursors on a more immature stage. Taken together, our results suggest that TLR4 signaling favors B lymphocyte maturation, whereas TLR2 arrests/retards that process, ascribing new roles for TLRs in B cell physiology.
https://www.ncbi.nlm.nih.gov/pubmed/15905502